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Anomalous Amyloid Microclots Found in 100% of the COVID-19 Vaccinated
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Welcome to the NoteBookLM.news series, where we delve deep into critical findings across science, medicine, and global affairs, prioritizing data-driven insights from the most consequential studies. Today, we are discussing two separate, but related, fields of research and analysis regarding the global health events of the past few years.
Focus 1: Anomalous Amyloid Microclots Found in 100% of the COVID-19 Vaccinated
Focus 1: Anomalous Amyloid Microclots Found in 100% of the COVID-19 Vaccinated
We begin with a profound biological finding detailed in a recent study by Nicolas Hulscher, Dr. Charles Hoffe, and Professor Michel Chossudovsky. This outstanding article focuses on anomalous amyloid microclots that are circulating in the blood vessels of people who received the COVID-19 vaccine. Critically, a new peer-reviewed study quietly revealed that every single vaccinated participant had fibrinolysis-resistant, ThT-positive amyloid microclots circulating in their blood. In fact, the data showed that 100% of the vaccinated individuals in the study had these amyloid microclots,,. This included every participant categorized as a "healthy control".
Dr. Charles Hoffe, whose conclusions were similar to those in this new study back in early 2021, previously referred to these phenomena as "Microscopic Blood Clots" or Hidden Covid-19 Vaccine Injuries. It is important to understand that many people who have been vaccinated may not be immediately aware of the injuries incurred, as adverse events are often not discernible or recorded. While major blood clots—the "big blood clots" that cause strokes and appear on CT scans or MRIs—are sometimes reported, Dr. Hoffe’s key insight was focusing on the microscopic blood clots generated by the mRNA vaccine,. These clots are so tiny they are undetectable by standard scanning technology and can only be detected using a D-dimer test.
The highly concerning element of these findings is that these people have no knowledge they are experiencing microscopic clotting. Furthermore, when tissue is damaged by blood clots in parts of the body that cannot regenerate, such as the brain, spinal cord, heart, and lungs, the resulting damage is permanent,.
The study itself reframes our understanding of a widely discussed condition. Although the authors of the original research framed their findings as relating to "Long COVID," the underlying data indicates they were observing Long VACCINE pathology,. This conclusion is supported by the fact that 94% of all study participants were vaccinated, and no participant had laboratory-confirmed SARS-CoV-2 infection through antibody testing, PCR, sequencing, or neutralizing antibody assays,. The assignment of "Long COVID" status was based solely on symptoms and clinician impression, meaning there is no evidence within the study that any participant was biologically positive for prior infection. Therefore, the clotting abnormalities documented cannot be attributed to infection, but rather to vaccination.
The defining criteria for identifying these structures was Thioflavin-T (ThT) positivity, an amyloid-binding fluorogenic dye, confirming that every microclot counted was, by definition, amyloidogenic. While small amyloid microclots were present in everyone vaccinated, the pathological burden differed sharply in those experiencing symptoms. Patients categorized as “Long COVID” (or Long VACCINE) exhibited extreme elevations in large, pathological amyloid microclots, with a total microclot burden approximately 20-fold higher than others. These larger, pathogenic clots were densely packed with materials including misfolded amyloid fibrin, neutrophil extracellular traps (NETs), myeloperoxidase, neutrophil elastase, and extracellular DNA,.
Further compounding these findings, a critical mechanistic experiment confirmed that spike protein alone produced identical amyloid microclots. When purified spike protein was added to fibrinogen in vitro, it generated insoluble, ThT-positive amyloid microclots, misfolded fibrin structures identical to those found in patient samples, and fibrinolysis-resistant aggregates consistent with vessel obstruction. This confirms that the spike protein directly induces amyloid microclot formation,.
These core findings—the 100% prevalence of amyloid microclots in vaccinated individuals and the direct induction of amyloid fibrin formation by spike protein—provide a clear mechanism for the large, rubbery white fibrous clots increasingly documented in deceased individuals since 2021. Forensic analysis of these postmortem clots, including surveys conducted among embalmers, demonstrates they are amyloidogenic fibrin aggregates, not standard thrombi. They are characterized by being protease-resistant and rubbery, exhibiting beta-sheet structures (ThT-positive), and having a dense fibrillar ultrastructure, all characteristics that exactly match the pathological microclots described in the new study, but in an end-stage, aggregated form,.
The biological progression outlined by these findings is straightforward: Spike exposure, whether through infection or mRNA vaccination, leads to the formation of amyloid microclots, which were present in 100% of the vaccinated subjects. These then accumulate into large, NET-rich, fibrinolysis-resistant clots—which were found at a 20-fold higher rate in Long vaccine patients—and ultimately merge into the massive, rubbery, white fibrous intravascular clots observed postmortem. Given the 94% vaccination uptake in the study cohort, the biological signal is overwhelmingly linked to spike exposure within the vaccinated population. These findings carry serious public-health implications, raising alarms about cumulative vascular injury globally since every vaccinated individual in the study showed these early-stage microclots.
Focus 2: The Worldwide Corona Crisis, Global Coup d’Etat Against Humanity
Focus 2: The Worldwide Corona Crisis, Global Coup d’Etat Against Humanity
Next, we turn to the second body of work presented in the sources, the comprehensive analysis by Michel Chossudovsky in his book, The Worldwide Corona Crisis, Global Coup d’Etat Against Humanity.
Chossudovsky’s work details how this insidious project, which he discusses, is actively destroying people's lives. His objective is to inform the global public and specifically refute the official narrative that has been used to justify the destabilization of the economic and social fabric of entire countries and the subsequent imposition of the "deadly" COVID-19 "vaccine". The book provides a detailed analysis across multiple dimensions, including the medical aspects, the economic and social repercussions, the political underpinnings, and the resulting mental and psychological impacts of the crisis.
Chossudovsky emphasizes that this crisis impacts humanity in its entirety, affecting nearly 8 billion people worldwide. He explicitly exposes the truth that there is no causal relationship between the virus and economic variables. Instead, the argument is made that the shutdown of the global economy was caused by the deliberate implementation of illogical and scientifically baseless lockdowns.
The broader context of the book presents this situation as a global coup d’état, also referred to as “The Great Reset”. The analysis suggests that if this ongoing movement is not resisted and defeated by freedom-loving people across the world, it will result in a dystopian future that has not yet been imagined.
thank you for listening to another session of the NoteBookLM.news series. You can find the full archive of all my podcasts at NotebookLM.news
Circulating Microclots Are Structurally Associated With Neutrophil Extracellular Traps and Their Amounts Are Elevated in Long COVID Patients
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Welcome to the NoteBookLM.news series, where we dissect complex scientific research and contextualize global events. Today, we delve into a new study that sheds light on the pathophysiology of Long COVID, while also revisiting crucial findings regarding vaccine-induced microclotting and the broader socio-political analysis of the recent global crisis.
Source 1: Circulating Microclots Are Structurally Associated With Neutrophil Extracellular Traps and Their Amounts Are Elevated in Long COVID Patients
Source 1: Circulating Microclots Are Structurally Associated With Neutrophil Extracellular Traps and Their Amounts Are Elevated in Long COVID Patients
We begin with a detailed research article in the Journal of Medical Virology, which provides essential insights into the persistent and challenging condition known as Long COVID (LC). This study focuses on the critical link between microclots and Neutrophil Extracellular Traps (NETs), arguing that this association not only suggests novel diagnostic pathways but also deepens our understanding of the thromboinflammatory processes underlying LC.
The foundational premise is that the persistence of vasculo-thrombotic complications is a possible contributing factor in the LC syndrome. The study successfully confirmed the association of LC with elevated levels of heterogeneous fibrin(ogen) amyloidogenic particles, referred to as microclots, and with markers of NETs, both of which are strongly linked to thromboinflammation.
The results demonstrated a powerful quantitative and structural association between NET markers—specifically Myeloperoxidase (MPO), Neutrophil Elastase (NE), and circulating DNA (cirDNA)—and both the size and number of microclots in patients diagnosed with LC. These markers, individually and combined, showed a strong diagnostic performance. Critically, the researchers suggest that NETs may physically be a component of circulating microclots. Furthermore, they hypothesize that increased NETs formation might promote the stabilization of microclots in the circulation, potentially leading to deleterious effects that causally contribute to the LC syndrome.
A central finding of this research is the quantitative elevation of these markers in symptomatic patients. The total number of microclots, as determined by imaging flow cytometry, was statistically and significantly higher in patients with LC compared to healthy individuals, showing a remarkable 19.7-fold median difference. This elevation was consistent across all arbitrarily selected microclot size ranges. Similarly, the plasma concentrations of NETs markers were substantially higher in LC patients, specifically cirDNA (5.7-fold higher), MPO (3.5-fold higher), and NE (14.9-fold higher).
The structural nature of these clots also distinguishes them from normal physiological clotting. The researchers define "microclots" as small, circulating fibrin amyloidogenic complexes that may form independently of thrombin and are resistant to fibrinolysis. Unlike healthy fibrin meshes composed of long fibers typically induced by thrombin, microclots are characterized by an amyloid-like nature, revealing themselves through staining with fluorogenic dyes like Thioflavin T (ThT). Previous research has established that these microclots not only contain fibrinogen molecules but also entrap various inflammatory molecules and antibodies.
Direct experimental evidence supports the mechanism of microclot formation: prior work showed that the simple presence of the spike protein S1 from SARS-CoV-2 is sufficient to induce fibrinolytic-resistant microclots. The current study validated this by demonstrating that adding spike protein or lipopolysaccharide (LPS) to purified fibrinogen in vitro induced the formation of insoluble microclots, confirming the presence of fibrin(ogen) and amyloidogenic characteristics within these structures.
The structural association between microclots and NETs was confirmed through fluorescence microscopy, which showed that MPO, ThT, and DNA clearly colocalized within microclots. This strong correlation suggests that the interplay between the innate immune response and blood coagulation, known as immunothrombosis, leads to the formation of microthromboses. The authors speculate that exacerbated and deleterious NETs production may be maintained by a positive NETs/inflammation feedback loop, causing a persistent stimulation of neutrophils. This mechanism, coupled with the binding and entrapment of microclots by NET parts, could inhibit their degradation, leading to their elevated number and prolonged presence in the circulation.
The clinical implications are substantial, suggesting that abnormal blood clotting and microclot formation are important factors that may explain the development and persistence of symptoms in LC. Furthermore, machine learning models, specifically the Random Forest classifier, were used to evaluate the diagnostic potential of these markers. The results showed that microclots, particularly in the 100–400 $\mu$m² size range, and MPO were the most critical features for differentiating LC patients from healthy subjects, achieving an impressive classification accuracy of 0.91 and an AUC of 0.95. The researchers emphasize that the combination of these markers offers a potential avenue for addressing the underdiagnosis of LC, improving diagnostic accuracy, and paving the way for targeted management strategies.
Source 2: Anomalous Amyloid Microclots Found in 100% of the COVID-19 Vaccinated
Source 2: Anomalous Amyloid Microclots Found in 100% of the COVID-19 Vaccinated
Drawing on previous discussions, we turn to the source detailing findings on anomalous amyloid microclots observed in the vaccinated population [Conversation History (CH)]. This study highlighted a key biological signal that every single vaccinated participant had fibrinolysis-resistant, ThT-positive amyloid microclots circulating in their blood [CH]. This crucial finding indicated that 100% of the vaccinated individuals in the study had these amyloid microclots, including those who were considered "healthy controls" [CH].
This research supported earlier observations made by Dr. Charles Hoffe, who previously identified these phenomena as microscopic blood clots [CH]. A significant concern raised is that many vaccinated individuals may be unaware of these microscopic injuries, as adverse events are often not immediately discernible or recorded [CH]. These microscopic blood clots, undetectable by standard imaging such as CT or MRI, are generated by the mRNA vaccine and can only be detected using specific tests like the D-dimer assay [CH].
The implications of these microscopic findings relate directly to long-term health consequences: when blood clots cause damage in tissues that cannot regenerate, such as the brain, spinal cord, heart, and lungs, the resulting tissue damage is permanent [CH].
A critical takeaway point from this research was the identification of the causative mechanism: the spike protein alone produced identical amyloid microclots [CH]. Specifically, adding purified spike protein to fibrinogen in vitro resulted in the generation of insoluble, ThT-positive amyloid microclots and misfolded fibrin structures that were identical to those found in patient samples [CH]. This direct link confirms that the spike protein induces the formation of amyloid microclots [CH].
The authors connected these microscopic findings to larger, post-mortem phenomena, suggesting a progression of injury: initial spike exposure leads to widespread amyloid microclot formation (100% prevalence in the vaccinated cohort), which then accumulate into larger, NET-rich, fibrinolysis-resistant clots (observed at a 20-fold higher rate in symptomatic patients), ultimately merging into the massive, rubbery, white fibrous intravascular clots found during forensic examination [CH]. The overwhelming majority of the study cohort was vaccinated (94% uptake), which strengthens the conclusion that the observed biological signal of vascular abnormalities is strongly linked to spike exposure within the vaccinated population [CH].
Source 3: The Worldwide Corona Crisis, Global Coup d’Etat Against Humanity
Source 3: The Worldwide Corona Crisis, Global Coup d’Etat Against Humanity
Finally, we turn to the broader socio-political and economic analysis offered by Michel Chossudovsky in his book, The Worldwide Corona Crisis, Global Coup d’Etat Against Humanity, which provides a distinct macro-level interpretation of the recent crisis [CH].
The essential goal of this work is to inform the global public and provide a critical refutation of the official narrative used to justify the economic and social destabilization of entire nations and the subsequent imposition of the "deadly" COVID-19 "vaccine" [CH]. Chossudovsky emphasizes that this project is actively destroying people's lives [CH].
The analysis within the book is comprehensive, detailing the medical, economic, social, political, and resulting mental and psychological impacts of the crisis [CH]. He asserts that the crisis is global in scope, impacting nearly 8 billion people worldwide [CH].
A central and critical takeaway is the argument that the situation constitutes a global coup d’état, frequently referred to as “The Great Reset” [CH]. Chossudovsky explicitly argues that there is no causal relationship between the virus itself and the resulting economic devastation [CH]. Instead, the shutdown of the global economy was caused by the deliberate implementation of illogical and scientifically baseless lockdowns [CH]. This line of reasoning supports the conclusion that the crisis was intentionally orchestrated, rather than being a natural response to a biological threat [CH].
The final and most crucial takeaway is a stark warning: the author suggests that if this ongoing movement is not resisted and defeated by freedom-loving people across the world, it will inevitably lead to a dystopian future that humanity has yet to imagine [CH].
thank you for listening to another session of the NoteBookLM.news series. You can find the full archive of all my podcasts at NotebookLM.news
The statement "Anomalous Amyloid Microclots Found in 100% of the COVID-19 Vaccinated" is the title of a detailed article by Nicolas Hulscher, Dr. Charles Hoffe, and Professor Michel Chossudovsky, and it characterizes what they identify as one of the most consequential biological findings of the pandemic era1,2,3,4,5.
The characterization of this statement, based on the sources, includes its origin, core biological basis, proposed mechanism, and critical implications:
Origin and Core Finding
The statement is derived from a detailed review of a new peer-reviewed study, which, according to the sources, quietly revealed this critical finding2,5. The authors of the review emphasize that while the original study did not explicitly acknowledge the demographic pattern, supplementary data showed that out of all participants, 94% were vaccinated2,5,6. The fundamental conclusion reached is that every single vaccinated participant in the underlying study had these microclots2,5,7. This result means that 100% of the vaccinated individuals in the study had amyloid microclots, including every participant categorized as a "healthy control"8,5,6,9.
Biological Nature of the Microclots
The statement refers to specific structures with defined characteristics:
1. Amyloidogenic Nature: The microclots were identified using Thioflavin-T (ThT), an amyloid-binding fluorogenic dye10,7. The defining criterion for counting a structure as a microclot was ThT positivity, meaning every structure counted is, by definition, amyloidogenic10,6. These are misfolded amyloid fibrin structures11,12.
2. Resistance to Breakdown: The clots are characterized as fibrinolysis-resistant2,5,13.
3. Scale: Dr. Charles Hoffe, who came to similar conclusions early in 2021, referred to these as "microscopic blood clots"14,15. He noted that these clots are too small to be found on standard scans like CT or MRI, and can only be detected using the D-dimer test16,17.
Causality and Mechanism
The statement strongly implies a causal link to the vaccination:
1. Reframing Pathology: Although the underlying study framed its results as being related to "Long COVID," the Hulscher review argues that the study is actually observing Long VACCINE pathology8,7. This assertion is based on the fact that none of the participants in the original study were confirmed to have had prior SARS-CoV-2 infection through laboratory methods (no antibody testing, PCR, sequencing, or neutralizing antibody assays were performed)11,18. Therefore, the observed clotting abnormalities cannot be specifically attributed to infection but rather to vaccination19,18.
2. Spike Protein Induction: The mechanistic link is confirmed by experiments showing that purified spike protein alone produced identical amyloid microclots in vitro8,7. This intervention produced insoluble, ThT-positive amyloid microclots, misfolded fibrin structures identical to those in patient samples, and fibrinolysis-resistant aggregates compatible with vessel obstruction, confirming that the spike protein directly induces amyloid microclot formation19,13,20.
3. Pathological Burden: While small microclots were universally present in the vaccinated, patients categorized as symptomatic ("Long VACCINE") exhibited an extreme elevation in large, pathological amyloid microclots21,6. The total microclot burden in these symptomatic individuals was approximately 20-fold higher than in others21,12. These larger clots were found to be densely packed with elements of inflammation, including Neutrophil extracellular traps (NETs), Myeloperoxidase, Neutrophil elastase, Extracellular DNA, and misfolded amyloid fibrin21,11,12.
Implications and Progression of Injury
The statement characterizes a profound public health issue with a proposed progression of injury:
1. Progression Model: The core findings—100% microclots in the vaccinated and direct spike-induced amyloid fibrin formation—provide a clear biological mechanism for the large, rubbery white fibrous clots increasingly reported in deceased individuals since 202122,13. The proposed progression begins with spike exposure (from infection or mRNA vaccination), leading to amyloid microclot formation (present in 100% of vaccinated subjects), which then accumulate into large, NET-rich, fibrinolysis-resistant clots (20x higher in symptomatic patients), ultimately merging into massive, rubbery, white fibrous intravascular clots23,24.
2. Vascular Injury and Permanence: The presence of these microclots in every vaccinated individual in the study raises alarms about cumulative vascular injury across the entire globe25,20. Damage caused by these clots in non-regenerative parts of the body, such as the brain, spinal cord, heart, and lungs, results in permanent damage to those tissues16,26,17.
3. Need for Action: The findings carry serious public-health implications, prompting the conclusion that public health agencies must launch an immediate, transparent investigation into the white fibrous clot situation, and that any platform delivering spike protein into human circulation must be immediately banned for human use25,27,28.
Confidential Report: Analysis of Systemic Vascular Pathology and Geopolitical Implications
Confidential Report: Analysis of Systemic Vascular Pathology and Geopolitical Implications
The Clotting Crisis: Universal Amyloid Microclots, Causal Mechanism, and Global Response Imperatives
The Clotting Crisis: Universal Amyloid Microclots, Causal Mechanism, and Global Response Imperatives
Prepared for: Executive Leadership Date: December 2025
Page 1: Executive Summary
Page 1: Executive Summary
This report synthesizes critical findings from recent biomedical studies and geopolitical analyses concerning the health and systemic consequences of the global COVID-19 vaccination campaign and subsequent policy responses. The core findings identify a novel and widespread vascular abnormality—fibrinolysis-resistant amyloid microclots—with profound clinical and public health implications, challenging the prevailing official narrative.
Key Findings:
Universal Microclot Prevalence in the Vaccinated: A new peer-reviewed study, upon detailed demographic analysis, revealed that 100% of vaccinated participants had circulating fibrinolysis-resistant, ThT-positive amyloid microclots in their blood. This finding included every individual classified as a "healthy control".
Spike Protein as the Direct Cause: Mechanistic experiments confirmed that the purified spike protein alone directly induces the formation of these identical insoluble, amyloid microclots in vitro.
Severe Pathological Burden in Symptomatic Patients: Patients experiencing persistent symptoms (termed "Long VACCINE pathology" in the analysis) exhibited a total microclot burden that was approximately 20-fold higher than controls. These clots were densely packed with inflammatory markers, including Neutrophil Extracellular Traps (NETs).
Progression to Forensic Findings: This widespread microscopic pathology provides a clear biological mechanism for the large, rubbery, white fibrous clots increasingly reported postmortem since 2021. Forensic analysis confirms these structures are amyloidogenic, protease-resistant fibrin aggregates that precisely match the end-stage form of the microclots observed in the living.
Geopolitical Context: Concurrently, detailed geopolitical analysis characterizes the entire crisis response—including the imposition of lockdowns and the "deadly" vaccine—as an "insidious project" and a "global coup d’état" aimed at destabilizing countries and implementing a dystopian "Great Reset".
Strategic Imperatives:
Immediate Moratorium: Any platform delivering spike protein into human circulation must be immediately banned for human use.
Mandatory Investigation: Federal public health agencies must launch an immediate, transparent investigation into these microclot findings and the white fibrous clot phenomenon; failure to do so constitutes a dereliction of duty.
Page 2: Introduction & Scope
Page 2: Introduction & Scope
1.0 Problem Definition: Undetected Vascular Injury
1.0 Problem Definition: Undetected Vascular Injury
The initial phase of the global health crisis saw reports of "big blood clots" resulting from the vaccine, which caused strokes and were visible on standard scans (CT, MRI). However, an important analysis by Dr. Charles Hoffe suggested that the mRNA vaccine primarily generates "microscopic blood clots". These microscopic blood clots are too small to be found on conventional scanning technology and are only detectable using a D-dimer test. Consequently, many vaccinated individuals remain unaware of the injuries incurred, as adverse events are often not immediately discernible or recorded.
1.1 Research Objectives and Methodology
1.1 Research Objectives and Methodology
This report draws on two distinct but complementary lines of investigation:
Biological Validation (Thierry et al. & Hulscher Analysis): Analyzing the findings of a peer-reviewed study that utilized advanced techniques like imaging flow cytometry and Thioflavin-T (ThT) staining to quantify and characterize circulating fibrin(ogen) amyloidogenic microclots. The primary objective was to establish the prevalence, structure, and mechanism of microclot formation.
Macro-Systemic Context (Chossudovsky Analysis): Reviewing a comprehensive analysis that contextualizes the medical, economic, social, political, and psychological impacts of the crisis, positing a systematic destabilization strategy.
1.2 Methodology Overview: Detecting Anomalous Clotting
1.2 Methodology Overview: Detecting Anomalous Clotting
Microclots are defined as small, circulating fibrin amyloidogenic complexes that may form independently of thrombin and are resistant to fibrinolysis. Unlike healthy fibrin meshes composed of long fibers, these anomalous clots are characterized by an amyloid-like nature.
Staining Criterion: Researchers identified these microclots using Thioflavin-T (ThT), an amyloid-binding fluorogenic dye. ThT positivity was the defining criterion; thus, every microclot counted is, by definition, amyloidogenic.
Quantitative Measurement: Imaging flow cytometry was developed to quantify these microclots in platelet-poor plasma, measuring total counts and size ranges (e.g., 100–400 $\mu$m²).
Page 3: Core Biological Finding: Universal Clotting in the Vaccinated
Page 3: Core Biological Finding: Universal Clotting in the Vaccinated
2.0 Undeniable Prevalence: 100% Microclots in Vaccinated Individuals
2.0 Undeniable Prevalence: 100% Microclots in Vaccinated Individuals
The most consequential biological finding revealed in the underlying research is the universal presence of amyloid microclots among the vaccinated population in the study cohort.
Study Demographics: Out of the participants in the analyzed study, 83 of 88 (94%) were vaccinated.
Inclusion of Controls: Every single vaccinated participant had fibrinolysis-resistant, ThT-positive amyloid microclots circulating in their blood. This alarming finding included every individual designated as a "healthy control".
Microclot Size Distribution: According to Table S11 (referenced in the source), every vaccinated person in the study, regardless of symptomatic status, had amyloid microclots present in multiple size ranges.
2.1 The Severity of Hidden Damage
2.1 The Severity of Hidden Damage
Dr. Charles Hoffe highlighted the seriousness of these "Hidden Covid-19 Vaccine Injuries". The most alarming aspect of the microscopic clotting is the potential for permanent organ damage in non-regenerative tissues.
Permanent Organ Damage: When blood clots damage tissues like the brain, spinal cord, heart, and lungs, the resulting injury is permanent because these parts of the body cannot re-generate.
Cumulative Vascular Injury: The sources conclude that since every vaccinated individual in the study showed these early-stage amyloid microclots, there are serious public-health implications concerning cumulative vascular injury across the entire globe.
Page 4: Pathophysiology Analysis I: Microclots & Spike Protein
Page 4: Pathophysiology Analysis I: Microclots & Spike Protein
3.0 Direct Spike Protein Causality
3.0 Direct Spike Protein Causality
Mechanistic experiments performed by the authors of the underlying study provide a direct link between the SARS-CoV-2 spike protein and the formation of these pathological microclots.
In Vitro Confirmation: When researchers added purified spike protein to fibrinogen in vitro, this single intervention produced three critical pathological outcomes:
Insoluble, ThT-positive amyloid microclots.
Misfolded fibrin structures identical to those found in patient samples.
Fibrinolysis-resistant aggregates compatible with vessel obstruction.
Mechanism Verified: This confirms that Spike protein directly induces amyloid microclot formation. Previous research also showed that the simple presence of the spike protein S1 from SARS-CoV-2 is sufficient to induce fibrinolytic-resistant microclots.
3.1 Characterization of Amyloidogenic Microclots
3.1 Characterization of Amyloidogenic Microclots
The identified microclots differ fundamentally from normal physiological clots, primarily due to their unique structural and chemical properties:
Amyloid Nature: The structures are stained using Thioflavin-T (ThT), indicating a conformational change into an amyloidogenic structure.
Protease Resistance: The clots are fibrinolysis-resistant, meaning they are difficult to break down by the body's natural processes. Previous research showed they are resistant even to powerful proteolytic enzymes such as trypsin.
Entrapment of Inflammatory Molecules: Microclots are known to contain not only fibrinogen molecules but also entrap various inflammatory molecules and antibodies, including proteins that prevent clot breakdown (e.g., alpha-2-antiplasmin).
Page 5: Pathophysiology Analysis II: Long VACCINE vs. Long COVID
Page 5: Pathophysiology Analysis II: Long VACCINE vs. Long COVID
4.0 Reframing the Diagnosis: Long VACCINE Pathology
4.0 Reframing the Diagnosis: Long VACCINE Pathology
Although the original study framed its results as a hallmark of "Long COVID," the Hulscher analysis argues that the underlying data reveals a different reality: the observed pathology is more accurately defined as Long VACCINE pathology.
Lack of Infection Verification: Despite the "Long COVID" label, none of the study participants were confirmed to have had SARS-CoV-2 infection. The study performed no antibody testing, no PCR, no sequencing, and no neutralizing antibody assays.
Symptom-Based Labeling: "Long COVID" status was assigned purely via symptoms and clinician impression. Therefore, the clotting abnormalities, in a cohort with 94% vaccination uptake, cannot be attributed specifically to infection, but rather to vaccination. The biological signal is overwhelmingly linked to spike exposure in a vaccinated population.
4.1 Extreme Pathological Burden in Symptomatic Cohort
4.1 Extreme Pathological Burden in Symptomatic Cohort
While small amyloid microclots were universally present across the vaccinated cohort (including "healthy" controls), the pathological burden differentiated sharply in symptomatic patients.
20x Higher Burden: Patients identified as having Long VACCINE pathology demonstrated an extreme elevation in large, pathological amyloid microclots. The total microclot burden was approximately 20-fold higher in these symptomatic patients.
Prevalence of Large Clots: In the Long VACCINE group, 98% of patients had large microclots in the 900–1600 $\mu$m² range, and 60% had very large microclots exceeding 1600 $\mu$m². This contrasts with healthy individuals, where only 68.4% and 13.1% had microclots in these size ranges, respectively.
Page 6: Coagulation Dynamics: The Role of NETs (Thierry et al.)
Page 6: Coagulation Dynamics: The Role of NETs (Thierry et al.)
5.0 Association of Microclots with Neutrophil Extracellular Traps (NETs)
5.0 Association of Microclots with Neutrophil Extracellular Traps (NETs)
The detailed peer-reviewed research by Thierry et al. in the Journal of Medical Virology explored the critical association between microclots and markers of Neutrophil Extracellular Traps (NETs), linking them to thromboinflammation. NETs formation (NETosis) is a part of the innate immune response where activated neutrophils release chromatin and granular proteins (like Myeloperoxidase and Neutrophil Elastase) to trap microbes.
5.1 Quantitative Correlation of Markers
5.1 Quantitative Correlation of Markers
The study confirmed a strong quantitative association between microclots and NETs markers in symptomatic patients:
Elevated NETs Markers in LC: Plasma concentrations of NETs markers were statistically much higher in Long COVID (LC) patients than in healthy individuals (HI).
Circulating DNA (cirDNA) was 5.7-fold higher in LC patients.
Myeloperoxidase (MPO) was 3.5-fold higher in LC patients.
Neutrophil Elastase (NE) was 14.9-fold higher in LC patients.
Strong Correlation in LC: In LC patients, the concentrations of NE, MPO, and cirDNA were all clearly associated with the total number of microclots. This correlation, which was absent in healthy individuals, indicates that the interplay between the innate immune response and blood coagulation (immunothrombosis) leads to the formation of microthromboses.
5.2 Structural Integration and Stabilization
5.2 Structural Integration and Stabilization
Structural analysis suggests that NETs are not merely correlated with microclots, but may actually be a physical component.
Colocalization: Fluorescence microscopy confirmed that MPO, ThT (marking amyloid), and DNA (marking NETs) clearly colocalized within microclots.
Impaired Degradation: The researchers speculate that higher NETs formation promotes the stabilization of microclots in the circulation. This structural association, where NETs parts bind to and entrap microclots, may inhibit clot lysis, contributing to their high numbers and persistence in circulation. This resistance to normal breakdown is a key factor in the development and persistence of symptoms in LC.
Page 7: Clinical and Forensic Implications: The Progression Model
Page 7: Clinical and Forensic Implications: The Progression Model
6.0 Microscopic Damage Leading to End-Stage Clotting
6.0 Microscopic Damage Leading to End-Stage Clotting
The core finding of 100% amyloid microclots in vaccinated individuals and direct spike-induced amyloid fibrin formation offers a clear biological mechanism that explains the large, rubbery white fibrous clots increasingly reported in the deceased since 2021.
6.1 The Progression Model (From Living to Deceased)
6.1 The Progression Model (From Living to Deceased)
The pathology follows a biologically straightforward progression, as documented through study analysis of the living (early/intermediate stages) and forensic analysis of the deceased (final stage):
Stage 1: Spike Exposure (via infection or mRNA vaccination).
Stage 2: Amyloid Microclots Form (Present in 100% of vaccinated subjects).
Stage 3: Large, NET-rich, Fibrinolysis-Resistant Clots Accumulate (Found 20x higher in symptomatic Long VACCINE patients).
Stage 4: Massive, Rubbery, White Fibrous Intravascular Clots (These merge to form the final stage observed postmortem).
6.2 Forensic Validation: The White Fibrous Clots
6.2 Forensic Validation: The White Fibrous Clots
Surveys of embalmers and detailed forensic analysis validate the characteristics of the end-stage clots.
Embalmer Survey (2025 TFDA): A survey of embalmers found that 64% reported white fibrous clots in 2025, and these were found in 17% of all bodies. Furthermore, 70% observed widespread microclotting, described as "coffee-grounds blood".
Forensic Pathology Match: Forensic analysis by Kevin W. McCairn, PhD et al. confirms that these postmortem clots are not normal thrombi but are amyloidogenic fibrin aggregates. Their characteristics—protease-resistant, rubbery, fibrous, and exhibiting $\beta$-sheet structures (ThT-positive)—match exactly the pathological microclots described in the new study, only in a late, aggregated form.
Page 8: Strategic Recommendations & Diagnostic Potential
Page 8: Strategic Recommendations & Diagnostic Potential
7.0 Policy Mandates and Immediate Action
7.0 Policy Mandates and Immediate Action
Given the evidence of widespread, spike protein-induced vascular pathology that raises alarms about cumulative vascular injury globally, immediate strategic action is required:
Ban on Spike Protein Platforms: Any platform delivering spike protein into human circulation must be immediately banned for human use.
Immediate and Transparent Investigation: Federal public health agencies, including the CDC, are required to launch an immediate, transparent investigation into these microclot findings and the white fibrous clot situation. The failure to investigate constitutes a dereliction of duty.
7.1 Novel Diagnostic and Screening Strategy
7.1 Novel Diagnostic and Screening Strategy
The strong quantitative and structural association between microclots and NETs markers suggests a powerful new diagnostic strategy for Long VACCINE pathology.
Combined Biomarker Approach: A detection strategy demonstrating the combined presence of high microclot numbers and elevated levels of NETs markers (MPO, NE, cirDNA) offers an efficient means of addressing the underdiagnosis of the condition.
Machine Learning Validation: Machine learning models were employed to evaluate the diagnostic potential of these markers. The Random Forest (RF) classifier achieved the best performance.
Accuracy: The RF model achieved an accuracy of 0.91 and an Area Under the Curve (AUC) of 0.95.
Key Features: The most important features for discriminating symptomatic (LC) patients from healthy individuals were microclots in the 100–400 $\mu$m² size range and Myeloperoxidase (MPO) concentration.
Screening Power: The combination of an NETs marker (MPO) and a microclot numbering marker (100–400 $\mu$m²) achieved a very high discriminating/screening power, demonstrating clear potential for improved diagnostic accuracy and targeted management strategies.
Page 9: Macro-Strategic Context: Geopolitical & Economic Destabilization
Page 9: Macro-Strategic Context: Geopolitical & Economic Destabilization
8.0 The Global Coup d’État Narrative
8.0 The Global Coup d’État Narrative
Michel Chossudovsky’s analysis provides a crucial strategic lens, arguing that the crisis is an "insidious project" actively destroying people’s lives. His work aims to refute the official narrative used to destabilize countries and impose the COVID-19 "vaccine".
Crisis of Humanity: The crisis is characterized as affecting humanity in its entirety, impacting almost 8 billion people.
Deliberate Economic Shutdown: A central conclusion is that there is no causal relationship between the virus and economic variables. Instead, the shutdown of the global economy was caused by the deliberate implementation of illogical, scientifically baseless lockdowns.
PCR Test as a Weapon: The crisis was fueled by a "false covid pandemic," which was made possible by the widespread use of the Polymerase Chain Reaction (PCR) test, which produces up to 97% proven false positives, combined with a relentless campaign of fear.
8.1 The Great Reset Framework
8.1 The Great Reset Framework
Chossudovsky defines this systemic destabilization as a "global coup d’état under way called ‘The Great Reset’".
War Against Humanity: The crisis is framed as a singular, irregular, and massive assault against liberty and the goodness of people.
Dystopian Future Warning: The critical conclusion drawn from this geopolitical analysis is that if this movement is not resisted and defeated, it will lead to a dystopian future not yet imagined. The author urges the public to understand the wider perspective of how the crisis is contextualized within geopolitics.
Page 10: Conclusion & Call to Action
Page 10: Conclusion & Call to Action
The confluence of biological evidence and geopolitical analysis presented in this report reveals a health catastrophe driven by a documented mechanism of vascular injury, occurring within a systemic framework of global destabilization.
The scientific data demonstrates that the spike protein directly causes amyloid microclot formation. These microclots are now known to be circulating in 100% of the vaccinated population in the analyzed cohort, raising critical alarms about cumulative global vascular damage. Furthermore, these microscopic structures are the antecedent pathology for the large, rubbery, white fibrous clots being found postmortem.
The most severe finding is that damage caused by these microscopic clots in non-regenerative tissues—the brain, heart, spinal cord, and lungs—is permanent. The scale of potential hidden injury across the world population necessitates an immediate and radical shift in global health policy.
The medical necessity, combined with the strategic imperative to resist the systemic destabilization described as the "Global Coup d'État", demands that global policy leaders immediately ban any platform delivering spike protein into human circulation and launch a full, transparent forensic and clinical investigation into this crisis.
(End of Report)
Welcome back to the deep dive. Our mission here is always to take a dense stack of sources, articles, research papers—sometimes some really challenging analyses—and just distill the most important nuggets for you.
Right. To give you a comprehensive understanding without all the information overload.
Exactly. And today's deep dive is, I would say, unusually focused. We're dissecting a single but pivotal analysis, one that claims to synthesize evidence revealing a really widespread—maybe even fundamental—biological alteration that resulted from events of the pandemic era. And our goal has to be rigorous here. We need to unpack the specific claims made by the authors about the prevalence, the nature, and the progression of these anomalous blood structures. And we have to follow the source material's logic precisely.
We do.
Okay, so the core analysis we are focusing on is titled "Amyloid Microclots: Examining the Core Findings and Contextual Implications of Recent Pathology Studies," and it's written by Nicholas Holschuh, Dr. Charles Hoffe, and Prof. Michel Chossudovsky.
And what's really fascinating here—what kind of drew us to this—is the scope of the synthesis, right? The authors take these early, almost anecdotal clinical observations and they bridge them with rigorous peer-reviewed pathological data on the microscopic nature of these clots. And then they connect that all the way to macroscopic findings, things that have been independently observed in forensic settings. It's an attempt to build a really comprehensive biological and observational narrative, and we have to follow that analytical thread all the way through the source material's claims.
Okay, let's unpack this because the implications—I mean, if the authors' synthesis holds true—they're massive. They would demand immediate public health attention.
No question.
So we'll start where the analysis itself begins. That's with the foundational work of Dr. Charles Hoffe from back in early 2021.
Right.
The article highlights his perspective because, well, he reportedly came to very similar conclusions about a kind of silent clotting pathology at the very outset of the vaccine rollout in Canada. This was long before major studies were even published.
So he was one of the first to raise a flag on this specific issue.
Exactly. So what was his specific terminology for these structures, and what was the immediate alarming consequence he thought he was observing?
Dr. Hoffe referred to these specific blood structures as microscopic blood clots, and crucially, he labeled the resulting conditions as hidden COVID-19 vaccine injuries.
Hidden. That word is key.
It's foundational to the article's argument. It posits that the injuries being incurred are often not immediately discernible or even recorded because they're happening at a micro level. They're happening beneath the threshold of standard clinical observation.
That's it precisely.
And that distinction between the visible and the microscopic—it's absolutely critical. It dictates what clinicians and you, the public, are even aware of.
I mean, we all remember the media reporting on those rare visible big blood clots, the ones that cause strokes or severe thrombotic events.
Sure, they were all over the news.
But the authors emphasize that Dr. Hoffe was focused on something fundamentally different and potentially far more widespread.
Exactly. The article quotes Dr. Hoffe making this distinction very explicitly. He contrasted the big blood clots, which are the ones that cause strokes and show up on CT scans, MRI, etc., with the structures he was observing, which he said were what?
He stated they are microscopic and too small to find on any scan.
Okay, so the key takeaway there, according to the source, is that a widespread clotting process could be unfolding silently, right? Operating entirely under the diagnostic radar of standard imaging technology.
So if they're too small for any kind of visual detection with a scan, how did Dr. Hoffe even suggest they could be detected? This must lead us to some kind of diagnostic tool.
It does, and it's a critical detail: the reliance on the D-dimer test.
The D-dimer test. Can you break that down for us?
Of course. For anyone unfamiliar, the D-dimer test measures fibrin degradation products. Fibrin is the protein matrix of a clot.
Okay.
So when the body forms a clot and then tries to break it down—a process called fibrinolysis—these little fragments of the dissolved clot, the D-dimers, get released into the bloodstream.
So a high D-dimer level signals that there's significant clotting.
Mhm.
And also the attempt to break down those clots happening in the body.
Exactly. It suggests a large volume of clotting activity is taking place—activity that the person might be completely unaware of.
That's the chilling implication, and it's reinforced throughout the article that many people who receive the vaccine might be walking around with these elevated clotting signals, suggesting widespread but silent clot formation and breakdown attempts.
Yes. Without ever experiencing acute immediate symptoms. The injury is hidden. It's ongoing, and it's only visible through this single blood marker.
The discussion then moves pretty quickly to the implications for tissue damage. This is where a silent injury becomes a permanent one, right?
The source material really emphasizes the authors' statement that certain critical tissues—specifically listing the brain, spinal cord, heart, and lungs—have very little, if any, ability to regenerate once they're damaged.
And this is where the concern for cumulative damage becomes overwhelming. Even if a microscopic clot is tiny and doesn't cause immediate catastrophic damage, the source material focuses on the long-term impact.
What's the quote they use?
The quote provided in the analysis states: "When those tissues are damaged by blood clots, they are permanently damaged."
That is a stark assessment. So if the clotting is silent but it's persistent, then the microscopic damage to essential organs is also persistent and non-reversible.
That's the implication. It suggests that even a low level of persistent microscopic clotting over time could accumulate into severe irreversible loss of function in essential non-regenerative organs like heart muscle or brain tissue.
Precisely. And analyzing that conclusion further, the article emphasizes Dr. Hoffe's stark warning from that very early period. It acts as a sort of prophetic introduction to the more rigorous pathology they analyze later.
And what was that warning?
"These shots are causing huge damage, and the worst is yet to come."
So this precursor discovery—it establishes the foundational claim.
Yes. That a widespread hidden microscopic clotting process was already suspected very, very early in the rollout. It really sets the baseline for everything that follows, right? We move from this early clinical suspicion and the D-dimer test to the findings of a massive pathology study that appears to confirm and quantify this microscopic damage.
Exactly.
Okay. So, now let's dive into the core study discussed in the article. The authors of our analysis claim that this new peer-reviewed study, once you reexamine it, reveals a foundational biological finding of the pandemic era. What is the single central finding about clot prevalence?
The central finding, at least according to the analysis' reinterpretation, is that every single vaccinated participant in the study had specific types of microclots circulating in their blood.
Every single one.
Every single one. The authors frame this as a universal biological alteration, a baseline state for the vaccinated population they studied. And it's a finding they contend the original research didn't fully appreciate or frame correctly.
Okay, before we even get to the prevalence, we have to define the specific characteristics that make these structures anomalous because this is where the technical clarity is vital. These are not just normal run-of-the-mill blood clots.
Not at all. And the study defined these structures using two specific non-negotiable chemical properties that distinguish them from a healthy thrombus or clot.
What's the first one?
The first feature is that they were identified as fibrinolysis resistant.
Fibrinolysis resistant. Let's break that down.
Sure. Fibrinolysis is the body's natural recycling system for blood clots. It ensures that temporary plugs are dissolved once their job is done. A structure that is resistant to this process actively resists the body's natural cleanup mechanisms.
And why is that resistance such a critical pathological feature?
Because it means the structures are persistent. A normal clot—it dissolves in hours or days. A fibrinolysis-resistant clot persists indefinitely. It becomes a semi-permanent obstacle in the microvascular system.
So it transforms a temporary physiological response into a chronic pathological problem, a blockage the body just can't clear.
Exactly. Leading to chronic low-level injury and accumulation.
And the second defining characteristic, the one related to their composition that really defines their pathological nature.
They were ThT positive.
ThT positive, which means they bound the fluorogenic dye thioflavin T. This dye is the crucial chemical marker because it specifically binds to a very particular type of protein structure: the beta-sheet structure.
And the article specifies that a structure was only counted as a microclot if it showed this ThT positivity.
That's right. That was the rule.
And based on that specific binding, the authors make a definitive technical statement about the composition of these things.
They do. This binding means, by chemical definition, that every single microclot they counted was amyloidogenic.
Amyloidogenic. That's the key term.
It is. Amyloid means the protein is misfolded. It's adopted this highly stable, typically insoluble configuration that is extremely resistant to natural breakdown.
When most of us hear the word amyloid, we think of diseases like Alzheimer's or Parkinson's—you know, conditions defined by misfolded protein deposits in the brain. So the article is claiming that this fundamental chemically resistant misfolding is happening, but it's happening in the circulating blood of the study participants.
That's exactly right. It uses that powerful, almost intuitive analogy: normal proteins are flexible and soluble; amyloid proteins are like a complex, rigid, sticky structure—almost like microscopic rebar or cement—functionally insoluble.
And the body has a very poor mechanism for clearing them. So the finding here is that this kind of structure isn't just a localized brain issue but a circulating blood issue.
Okay. So let's drill down into the demographic and the statistical conclusion that the authors draw from the study's supplementary tables—S1, I believe—because this is where the most staggering claim about prevalence comes from.
Right. So the analysis highlights the cohort demographics: 83 of 88 study participants—that's a massive 94% of the entire group—were documented as vaccinated.
94%.
And crucially, the analysis then asserts that 100% of these vaccinated individuals had amyloid microclots. This is presented as an inescapable statistical link.
And this 100% prevalence included every person labeled as a healthy control in the original study. This is the moment where the authors argue the original study completely missed the core biological shift.
That's their central argument. The source material explicitly states that the control group, who were defined as healthy and asymptomatic, were statistically indistinguishable from the symptomatic group regarding the mere presence of these small amyloid microclots.
So everyone had them.
Everyone vaccinated in this cohort had them. The authors synthesize this data to argue that the presence of these specific anomalous fibrinolysis-resistant clots is not a feature of severe illness but rather a universal underlying biological alteration, present in everyone vaccinated in this cohort regardless of their current symptom status.
If that biological alteration—this presence of insoluble misfolded protein clots—is universally linked to the vaccinated population within the study, what does that do to the definition of health in this population group?
According to the source material, it changes everything. It suggests a fundamental shift has occurred in the body's vascular homeostasis. If 100% of the vaccinated population has these clots, then the absence of these clots is no longer the metric for health.
So clinicians who are relying on previous baseline metrics might miss this universal underlying vascular stress.
That's the argument. The authors are arguing for a fundamental redefinition of the post-vaccination state as one characterized by universal persistent low-level microclotting.
So to sum up this section, the core finding the authors are pushing is that this amyloid pathology isn't some occasional adverse event.
No, it's a universal baseline biological change in the vaccinated population of the study, defined by the chemical resistance and the misfolded nature of the clots.
The source's primary and most consequential claim.
Absolutely. The presence of these specific anomalous fibrinolysis-resistant amyloidogenic microclots represents a fundamental and widespread post-vaccination state, according to their analysis.
So if 100% of the vaccinated individuals had these microclots as a baseline, let's move from that universal prevalence to pathological severity. What distinguished those people who were categorized as long COVID from the asymptomatic controls?
The difference was profound, and it was quantitative. It wasn't the presence of the microclots that was unique to the sick people—because everyone had them, right? It was their size and, crucially, their quantity. While the baseline alteration was universal, the symptomatic pathology, according to the source material, was dictated by the sheer volume and accumulation of these structures.
Let's put a number on that difference for the listener. How much greater was the physical burden in the highly symptomatic patients?
The total microclot burden was estimated to be approximately 20-fold higher in patients categorized as long COVID.
20-fold. We should probably pause on that. What are the functional implications of a 20-fold jump in clot burden?
Well, if you have 20 times the amount of insoluble permanent microvascular blockages, this immediately translates to vastly reduced circulation efficiency, increased resistance, and critically, chronic widespread ischemia.
Ischemia meaning a lack of blood flow and oxygen to tissues.
Precisely.
So if you think of the vascular system as the body's delivery network, a 20-fold increase in permanent sludge means the entire network is constantly fighting extreme traffic congestion, especially in the smallest capillaries.
That's the perfect analogy. This constant high-volume obstruction doesn't just cause acute events; it starves tissue slowly.
And the authors argue this is the mechanism that explains the chronic fatigue, the brain fog, the multi-organ symptoms that are characteristic of the condition. It's chronic widespread low-level oxygen deprivation caused by systemic microvascular compromise.
The analysis also drilled down into the specific size ranges where this pathological burden was concentrated, using metrics from table S1 of the original research. This highlights the kind of large-scale aggregation that was happening in symptomatic people.
Yes, the aggregation was significant. They observed that 98% of the symptomatic patients had large microclots in the 900 to 1,600 square micrometer range, which—to put that into perspective—is large enough to cause serious issues in smaller arterioles and capillaries.
Definitely. And furthermore, 60% of these patients had very large microclots, which were defined as those measuring over 1,600 square micrometers.
So these structures are functionally significant in blocking small vessels. But what's equally important is what these larger pathogenic microclots... The analysis stresses they were far more complex than just misfolded fibrin.
Oh, much more complex.
What components suggest an ongoing, almost chaotic immunological response? This is where the narrative shifts from simple physical blockage to what you could almost call an immunological war zone. These larger clots were densely packed with components that reflect a severe localized immunological event centered around the blockage.
Like?
The analysis lists several components. First, neutrophil extracellular traps, or NETs. These are essentially the internal contents of neutrophils—a type of white blood cell—that have been violently ejected in an attempt to contain a threat. They form a sticky, web-like mesh.
Okay. So the immune system is throwing out its own guts to trap something.
In a sense, yes. Then you have myeloperoxidase, or MPO, and neutrophil elastase. These are powerful destructive enzymes released by the neutrophils as part of their defensive mechanism.
And what else?
Extracellular DNA, which forms the literal structural core of the NETs, and of course, the misfolded amyloid fibrin at the center of it all.
This concentration of immune components—it suggests an active, unresolved inflammatory process localized around the amyloid structure. If NETs are the immune system throwing out its DNA and its chemical weapons to trap something, but that structure is fibrinolysis resistant...
It suggests a continuous failed attempt by the body to neutralize an insoluble problem.
That's the core interpretation the authors present.
Exactly. The immune system recognizes the amyloid clot as a threat that needs to be contained and eliminated. It deploys its most aggressive tools—the NETs, MPO, elastase—but because the clot is chemically designed to resist enzymatic breakdown, the immune attack fails to clear it.
And instead, the debris—the NETs, the DNA, the enzymes—just compounds the mass, making the clot larger, stickier, and more persistent. It turns a simple physical obstruction into a site of chronic immunological engagement, which contributes to the systemic symptoms.
So, the symptomatic people aren't just experiencing more clots. They're experiencing clots that are actively engaging and consuming the immune system, leading to a much higher burden of toxic inflammatory byproducts.
Exactly. This is why the quantification is so important. The pathology isn't just about the presence of a few microscopic particles. It's about a massive, complex, and persistent accumulation that drains the body's resources and constantly compromises microvascular integrity.
Okay, so section four is the crucial link here, attempting to establish a direct causal mechanism between the pathology they saw in patients and the spike protein. The study authors performed an in vitro mechanistic experiment to isolate the role of the spike protein itself.
Right? And this experiment is absolutely foundational to the article's core thesis. It attempts to remove all other confounding factors—other proteins, immune responses, infections—and test the ability of the spike protein alone to initiate the precise pathologies seen in the patients.
And the methodology was pretty simple, right?
Elegantly simple. The authors added purified spike protein alone to fibrinogen—which is the precursor protein that circulates in the blood and forms fibrin, the backbone of a clot—in a controlled test tube environment.
Now, in a conversational context, you have to ask: how robust is a claim of direct causation based only on an in vitro—a test tube—experiment? I mean, that's a massive leap from a lab dish to the complexity of the human body.
It is. And while the analysis acknowledges that in vivo—in body—conditions are vastly more complex, they argue the in vitro findings provide compelling evidence of potential causation. The results demonstrate the intrinsic capability of the spike protein to act as a pro-amyloidogenic agent.
So it shows what the spike protein can do on its own.
Exactly. And the source material outlines three major outcomes, confirming that the intervention directly produced structures that were identical in their key pathological features to those found in the patient's samples.
Let's detail those outcomes to really reinforce the direct causal link the article claims.
Okay, first: the intervention successfully produced insoluble ThT-positive amyloid microclots, which for the authors is the smoking gun.
It is. It proves that the spike protein alone—without any input from other viral components or a complex immune response—has the intrinsic chemical ability to generate that misfolded amyloid structure.
So the spike protein doesn't just trigger a cascade that leads to clotting. It physically interacts with the clotting precursor to force it into a resistant misfolded shape.
Precisely. This structural coercion is key. Secondly, the reaction generated misfolded fibrin structures identical to those found in patient samples. This confirms the structural and chemical resemblance between the lab-generated clots and the real-world pathology, which validates the in vitro model as relevant.
Yes. And the third result is what confirms the functional pathology—that these clots are designed to stick around, making them relevant to vessel obstruction.
What was that result?
The final critical result was that the intervention yielded fibrinolysis-resistant aggregates compatible with vessel obstruction, which confirms that the spike protein not only creates the structure but also imparts that defining pathological characteristic: the resistance to breakdown that makes microclots persistent and dangerous in the human body.
That's right. The analysis concludes that this mechanistic experiment confirmed that spike protein directly induces amyloid microclot formation. The single intervention, the authors contend, corroborates the core thesis.
The thesis being that the presence of the spike protein in circulation is sufficient to initiate this specific anomalous and highly resistant clotting pathology.
The authors argue that this closes the loop. It means that regardless of whether the spike protein originated from a natural infection or from an intervention designed to deliver it, its presence in the bloodstream is chemically sufficient to initiate the formation of these anomalous, highly resistant amyloid clots—the very same clots found universally in the study's vaccinated participants.
Very sad.
This is a major inflection point in the article. The authors pivot—and they pivot sharply—against the study's original framing. They present a very forceful argument that the condition being observed is not long COVID but fundamentally long vaccine pathology.
It's a huge reframing, and it's based on a specific set of biological and statistical observations within the study cohort.
What's the critical evidence they use to support this?
It's twofold. First, the overwhelming vaccination status of the cohort. Second, the complete lack of standard biological confirmation for prior SARS-CoV-2 infection among the participants.
So they're arguing that the label "long COVID" requires evidence of COVID-19 infection, and they claim that evidence is completely absent in the data.
The source material is very specific about the diagnostic measures that were not performed in the original study, which the authors used to undermine that long COVID diagnosis.
That's right. They meticulously point out that the original study performed no standard antibody testing to confirm past exposure, no PCR testing, no sequencing, and no neutralizing antibody assays.
So, how was the status of long COVID even assigned?
According to the analysis, it was assigned purely based on symptoms and the clinician's impression.
Not on biological data.
Not on biological data. So, the authors argue forcefully that scientifically, there is no biological evidence provided in the study that any participant was positive for prior infection, which means the clotting abnormalities cannot be definitively or specifically attributed to the virus itself. So without that biological evidence of a past infection, the overwhelming statistical link falls back to the one common biological event shared by almost the entire cohort, both symptomatic and asymptomatic.
And that event is vaccination. Given the 94% vaccination uptake in the study cohort, the biological signal—the universal presence of the amyloid microclots—is claimed to be overwhelmingly linked to spike protein exposure in a vaccinated population.
The authors contend that labeling a pathology found in 100% of a 94% vaccinated cohort as long COVID is a profound error of interpretation, naturally when the spike protein has been mechanistically proven to cause the pathology in vitro.
So the source material is essentially arguing that when you have a universal pathology—the amyloid microclots—in a heavily vaccinated group, and a proven mechanism linking that pathology directly to the spike protein delivered by the vaccine platform, the logical conclusion is that the pathological finding is intrinsic to the shared intervention, not some speculative antecedent infection.
They believe the original study just failed to properly synthesize its own findings.
That's the argument. By focusing on the symptomology as the primary classifier, they argue the researchers ignored the fundamental biological observation of universal amyloid microclot presence, which is linked directly to the mechanism of spike protein exposure within a population that shared a massive history of the intervention.
And the authors insist the data compels the label "long vaccine pathology."
It does. And this shift in nomenclature—it has immense clinical and policy ramifications. It moves the causality argument away from the infection and directly onto the intervention delivering the spike protein.
It forces a completely different investigative pathway.
It does. If the pathology is fundamentally long vaccine, it necessitates an investigation into the manufacturing stability and distribution of the spike protein itself rather than focusing solely on the long-term effects of viral remnants.
Okay, this next section is critical. It bridges the microscopic pathology observed in the living to the macroscopic visible findings reported in the deceased. The article introduces observational reports that seem to align the study's core findings with the emergence of these large rubbery white fibrous clots that have been increasingly reported by forensic professionals since 2021.
The authors use this observational data to corroborate the end stage of their proposed pathology. They cite a specific and very compelling data set: a survey conducted by former USAF Major Tom Haviland.
And this was presented at the 2025 Tennessee Funeral Directors Association convention.
That's right. The survey attempted to quantify the prevalence of these unusual postmortem findings in the embalming industry.
Let's go through those specific data points. They suggest a major shift in what forensic professionals are encountering.
The survey results are specific and, according to the source material, quite alarming. First, 64% of embalmers reported finding these white fibrous clots in 2025. This suggests a really widespread geographical observation, not just an isolated phenomenon.
Not at all. Furthermore, these specific clots were found in 17% of all bodies processed.
17%. That is a massive proportion of all deceased individuals displaying this anomalous finding.
It's huge. And in addition to the large clots, 70% of embalmers also observed widespread microclotting, which was vividly described as "coffee grounds blood."
Coffee grounds blood.
Yes, which aligns with the idea of widespread non-cleared microvascular pathology.
And the survey noted other demographic shifts as well.
It did. It reported that 39% of respondents noted rising infant deaths, specifically a 14% increase.
The observation of coffee grounds blood strongly supports the earlier claims about widespread microclotting that the body can't resolve, while the large rubbery clots represent the final aggregated structure.
That's the connection they're making.
So, do these macroscopic postmortem clots share the precise pathological characteristics of the microscopic ones we discussed earlier—the amyloid nature, the resistance?
Well, the article cites forensic analysis by Kevin W. McKernan, PhD, and others, claiming that these analyses confirm the physical and chemical characteristics do match the pathology found in the living.
What are the specific points of that match?
The source material emphasizes several. They are confirmed to be amyloidogenic fibrin aggregates, not normal thrombi. Normal clots are red, jelly-like, and easily broken down. These are white, rubbery, and highly resistant.
Okay.
They exhibit beta-sheet structures, meaning they bind ThT and are chemically amyloid. They are physically protease resistant, rubbery, and fibrous, confirming their high resistance to breakdown. And they show a dense fibrillar ultrastructure on scanning electron microscopy (SEM), indicating a highly organized pathological aggregation.
And is there any direct link back to the intervention?
The forensic analysis found that they contain human genetic material and show preliminary plasmid spike-associated markers, which directly links the end-stage pathology back to the components involved in the intervention platform.
The authors are essentially using this evidence to construct a comprehensive biological progression—a narrative.
That's right.
Let's summarize the proposed four-step progression outlined in the source material, linking the initial exposure to the final end-stage finding.
Okay. The source material proposes a clear progressive pathological pathway. One: spike exposure—either from infection or mRNA vaccination—initiates the process by entering the bloodstream.
Step one.
Two: amyloid microclots form. These are the small fibrinolysis-resistant structures documented as present in 100% of the vaccinated subjects.
Step two.
Three: large NET-rich fibrinolysis-resistant clots accumulate. This is the intermediate high-burden stage observed to be 20 times higher in symptomatic patients, causing chronic ischemia and severe symptoms.
And the final step.
Four: these merge into massive rubbery white fibrous intravascular clots. This is the final end-stage form seen in the deceased and corroborated by the embalmer surveys.
So this narrative argues that the study on the living documented the early and intermediate stages of the pathology, while the forensic analysis and the embalmer surveys reveal the final catastrophic aggregated stage of a continuous vascular disease process. It creates this powerful and frankly disturbing narrative that links microscopic chemistry to macroscopic death, suggesting a continuous progressive vascular pathology initiated by the spike protein.
That is the claim.
The final section of the article then translates these pathological findings into immediate policy and public health demands. Given the observation of amyloid microclots in every single vaccinated individual in the study cohort, the conclusion drawn by the authors carries serious global public health implications.
The primary alarm they raise is extremely broad: cumulative vascular injury across the entire globe.
Across the globe.
Yes. Since the pathology was found in 100% of vaccinated participants, including the healthy controls, the authors extrapolate this finding. They argue that it suggests this microclotting is not a rare side effect, but a fundamental biological alteration across the world's vaccinated population.
They're contending this represents a global ticking clock of vascular compromise.
That's the essence of it.
That extrapolation, if true, means that billions of people might have a compromised microvascular system right now, facing long-term consequences far beyond just acute adverse events. What explicit demands are directed at regulatory bodies like the CDC based on these findings?
The demands are presented as non-negotiable and urgent. The authors state that the CDC and federal public health agencies must launch an immediate transparent investigation into these findings.
An investigation into what specifically?
Specifically focusing on the large white fibrous clots being reported in the deceased. The authors claim that failing to investigate the white fibrous clot situation, given the pathological evidence of amyloid formation in the living, constitutes a dereliction of duty.
A dereliction of duty by those charged with protecting public health.
That's the language they use.
And based on the claimed mechanism that the spike protein alone induces these amyloid clots and the universal prevalence found in the study, what is the definitive policy recommendation provided in the source material?
The policy recommendation is unequivocal and absolute: any platform delivering spike protein into human circulation must be immediately banned for human use.
Immediately banned.
Yes. This recommendation is the direct logical consequence of the authors' central finding. If the spike protein is the initiator of a widespread persistent and non-reversible pathological clotting cascade, then the continued use of any delivery system for that protein must cease immediately to prevent further global vascular injury. So the article concludes by reframing the findings not merely as some medical discovery that requires further study but as a critical immediate public health emergency—one that necessitates severe regulatory action against the claimed causal agent: the spike protein.
Okay. So we've established the intense biological and clinical claims from the analysis of the pathology studies. Now, adhering to our commitment to discuss all the provided source material, we have to shift...
A big shift.
A very big shift from this highly focused microscopic view to the broadest possible context claimed by one of the co-authors, Professor Michel Chossudovsky, in his related source material for the book, "The Worldwide Corona Crisis: Global Coup d'État Against Humanity." And this transition moves us entirely away from clinical pathology and squarely into the realm of geopolitical and economic claims.
Right?
The source material establishes the book's stated objective, which is to inform people worldwide and refute the official narrative—a narrative the author claims was used to destabilize the economic and social fabric of entire countries, followed by the imposition of the deadly COVID-19 vaccine.
And the focus here is on the massive scale of this alleged crisis.
It is. It's claimed to affect almost 8 billion people.
The scope of the book's analysis is claimed to be comprehensive, arguing that this alleged crisis is not just confined to medicine. What areas does the book claim to cover?
The book is claimed to cover the pandemic from its medical dimensions all the way to its economic and social repercussions, its political underpinnings, and its mental and psychological impacts.
So everything.
Everything. The overarching narrative from the author is that this structure, framed by the official narrative, destroys people's lives. The author states his objective is to provide a comprehensive tool for resistance against this perceived global architecture of control.
So diving into the book's specific mechanism for the alleged global crisis, how was the crisis engineered? According to the source material's description of the book's claims...
The book uses the framing of a false COVID pandemic and a plandemic.
Okay.
The operational tool, as summarized in the source material, is centered on the polymerase chain reaction, or PCR, test. This test is described in the source as a DNA-modifying test which allegedly produced an astronomical figure of up to 97% proven false positives.
97% false positives due to what?
Due to manipulation of the cycle threshold, or CT, rates.
So the core argument is that the health crisis—the pandemic itself—was manufactured using a faulty or deliberately misused diagnostic tool that massively inflated case numbers, thus creating the necessary perception of catastrophe.
Precisely. The book claims that this highly unreliable PCR test, combined with a relentless global 24/7 fear campaign conducted by media and governments, was the critical enabling factor for the entire event, allowing the worldwide panic-laden plandemic to be executed.
The source material quotes the author's conclusion that this global operation would never have been possible without the infamous DNA-modifying polymerase chain reaction test.
The book then addresses the devastating economic analysis. The author directly challenges the conventional understanding, noting the claim that there is no causal relationship between the virus and economic variables.
This is a really key distinction in the book's geopolitical argument. The book concludes that the economic crisis and the subsequent collapse were not a necessary consequence of the virus itself, but rather the intended result of specific deliberate policy decisions.
What decisions?
The book argues that the shutdown of the global economy was caused by the deliberate implementation of illogical, scientifically baseless lockdowns. So the lockdowns themselves were the mechanism of economic destabilization, not the viral threat they were supposedly meant to contain.
That is the claim. The economic fallout is presented as evidence of intent—a deliberate destabilization tactic rather than just a side effect of a health measure.
The authors frame the economic collapse as a policy choice designed to achieve a larger structural goal.
Yes, reinforcing the perception of a managed crisis.
And finally, what is the highest-level, broadest claim the source material asserts? The book aims to educate the reader about what is the ultimate alleged goal of this entire structure.
The book is claimed to be a comprehensive lesson on the premise that there is a global coup d'état underway called the Great Reset.
The Great Reset.
The authors argue this agenda—if not resisted and defeated immediately by freedom-loving people everywhere—will result in a dystopian future not yet imagined.
So the book is a call to action.
It explicitly encourages readers to disseminate this information rapidly, emphasizing the urgency of resisting this alleged global agenda before it is too late.
This transitions the scope from specific molecular pathology—the amyloid microclot—to a global structure where that pathology is presented as an intentional medically imposed consequence of a geopolitical agenda.
Yes, it connects the medical claims that a widespread potentially fatal pathology has been imposed on billions to the claims that this was executed through a manufactured health crisis, which itself was enabled by a faulty diagnostic test and destructive lockdowns. It is a unified narrative of control and consequence.
This has been an intensive deep dive into highly specific and consequential claims derived from the source material. We have spanned the entire spectrum, starting with the microscopic findings in the living: the universal prevalence of those fibrinolysis-resistant amyloid microclots in the vaccinated cohort, including those initially labeled as healthy controls.
We meticulously followed the mechanistic evidence demonstrating the direct role of the spike protein in producing these anomalous structures in vitro.
And we synthesized the observational link to the macroscopic postmortem findings, mapping out that proposed progression from microscopic persistence to massive rubbery white fibrous clots.
And finally, we addressed the broader context provided by co-author Michel Chossudovsky, detailing the claims that this medical crisis was facilitated by a manufactured health threat, enabled by high false-positive PCR tests and subsequent scientifically baseless lockdowns—all framed within the context of an urgent global agenda labeled the Great Reset.
So I have to leave you with one final thought here. If the prevalence of these amyloid microclots is indeed as universal as the study's reanalysis claims—present even in asymptomatic healthy vaccinated individuals—and if they do lead to irreversible damage to non-regenerative tissues as the authors contend, then the essential question becomes: what is the long-term ethical and legal liability for institutions that continue to promote this platform, knowing that a low-level silent biological alteration may have been occurring in the majority of the population? This persistent low-level damage, according to the source material, is the true hidden crisis.
A profound question that demands continuous focus and rigorous investigation based on the evidence presented in the source material. Thank you for listening to another session of the NotebookLM.news series. You can find the full archive of all my podcasts at notebooklm.news.
TRANSCRIPT OF LM AUDIO PODCAST
Welcome back to the deep dive. Our mission here is always to take well a dense stack of sources, articles, research papers, sometimes some really challenging analyses and just distill the most important nuggets for you.
Right. To give you a comprehensive understanding without all the information overload.
Exactly. And uh today's deep dive is, I would say, unusually focused. We're dissecting a single but pivotal analysis,
one that claims to synthesize evidence revealing a really wide spread maybe even fundamental biological alteration that resulted from events of the pandemic era
and our goal has to be rigorous here. We need to unpack the specific claims made by the authors about the you know the prevalence the nature and the progression of these anomalous blood structures
and we have to follow the source materials logic precisely
we do
okay so the core analysis we are focusing on is titled amalloid microclots examining the core findings and contextual implications of recent pathology studies and it's written by Nicholas Holshire Dr. Charles Hoff and Prof. Mikl Chzadovski.
And what's really fascinating here, what kind of drew us to this is the scope of the synthesis, right?
The authors take these early almost anecdotal clinical observations and they bridge them with rigorous peer-reviewed pathological data on the microscopic nature of these clots.
And then they connect that all the way to macroscopic findings, things that have been independently observed in forensic settings.
It's an attempt to build a really comprehensive biological and observation narrative and we have to follow that analytical thread all the way through the source material's claims.
Okay, let's unpack this because the implications I mean if the author synthesis holds true they're massive. They would demand immediate public health attention.
No question.
So we'll start where the analysis itself begins. That's what the foundational work of Dr. Charles Hoff from back in early 2021.
Right.
The article highlights his perspective because well he reportedly came to very similar conclusions about a kind of silent clotting pathology. at the very outset of the vaccine roll out in Canada. This was long before major studies were even published.
So he was one of the first to raise a flag on this specific issue
exactly. So what was his specific terminology for these structures and what was the you know the immediate alarming consequence he thought he was observing?
Dr. Hoff referred to these specific blood structures as microscopic blood clots
and uh crucially he labeled the resulting conditions as hidden coid9 vac. seen injuries
hidden. That word is key.
It's foundational to the article's argument. It posits that the injuries being incurred are often not immediately discernible or even recorded because they're happening at a micro level.
They're happening beneath the threshold of standard clinical observation.
That's it precisely.
And that distinction between the visible and the microscopic, it's absolutely critical. It dictates what clinicians and you, the public, are even aware of.
I mean, we all remember the media reporting on those rare visible big blood clots. the ones that cause strokes or severe thrombotic events.
Sure, they were all over the news.
But the authors emphasize that Dr. Hoff was focused on something fundamentally different and potentially far more widespread.
Exactly. The article quotes Dr. Hoff making this distinction very explicitly. He contrasted the big blood clots, which are the ones that cause strokes and show up on CT scans, MRI, etc., with the structures he was observing,
which he said were what
he stated they are microscopic and too small to find on any scan.
Okay, so the key takeaway there according to the source is that a widespread clotting process could be unfolding silently,
right? Operating entirely under the diagnostic radar of standard imaging technology.
So if they're too small for any kind of visual detection with a scan, how did Dr. Hoff even suggest they could be detected? This must lead us to some kind of diagnostic tool.
It does, and it's a critical detail. The reliance on the D-dimer test.
The d-dimer test. Can you break that down for us?
Of course. For anyone unfamiliar, The d-dimer test measures fibbrin degradation products. Fibbrin is uh it's the protein matrix of a clot.
Okay.
So when the body forms a clot and then tries to break it down a process called fibbronolysis, these little fragments of the dissolved clot, the d-dimer get released into the bloodstream.
So a high d-dimer level signals that there's significant clotting.
Mhm.
And also the attempt to break down those clots happening in the body.
Exactly. It suggests a large volume of clotting activity is taking place. adding activity that the person might be completely unaware of.
That's the chilling implication and it's reinforced throughout the article that many people who receive the vaccine might be walking around with these elevated clotting signals suggesting widespread but silent clot formation
and breakdown attempts.
Yes. Without ever experiencing acute immediate symptoms. The injury is hidden. It's ongoing and it's only visible through the single blood marker.
The discussion then moves pretty quickly to the implications for tissue damage. This is where a silent injury becomes a permanent one. Right?
The source material really emphasizes the author's statement that certain critical tissues, specifically listing the brain, spinal cord, heart, and lungs, have very little, if any, ability to regenerate once they're damaged.
And this is where the concern for cumulative damage becomes well overwhelming. Even if a microscopic clot is tiny and doesn't cause immediate catastrophic damage, the source material focuses on the long-term impact.
What's the quote they use
the quote provided in the analysis states when those tissues are damaged by blood clots they are permanently damaged
that is a stark assessment so if the clotting is silent but it's persistent then the microscopic damage to essential organs is also persistent and non-reversible
that's the implication it suggests that even a low level of persistent microscopic clotting over time could accumulate into severe irreversible loss of function
in essential non-regenerative organs like heart muscle or brain tissue.
Precisely. And analyzing that conclusion further, the article emphasizes Dr. Hoff's stark warning from that very early period. It acts as a sort of prophetic introduction to the more rigorous pathology they analyze later.
And what was that warning?
These shots are causing huge damage and the worst is yet to come.
So this precursor discovery, it establishes the foundational claim.
Yes. That a widespread hidden microscopic clotting process was already suspected very very early in the roll out. It really sets the baseline. for everything that follows,
right? We move from this early clinical suspicion and the D-dimer test to the findings of a massive pathology study that appears to confirm and quantify this microscopic damage.
Exactly.
Okay. So, now let's dive into the core study discussed in the article. The authors of our analysis claim that this new peer-reviewed study, once you reexamine it, reveals a foundational biological finding of the pandemic era. What is the single central finding about clot prevalence? The central finding, at least according to the analysis reinterpretation, is that every single vaccinated participant in the study had specific types of microclots circulating in their blood.
Every single one.
Every single one. The authors frame this as a universal biological alteration, a baseline state for the vaccinated population they studied. And it's a finding they contend the original research didn't fully appreciate or frame correctly.
Okay, before we even get to the prevalence, we have to define the specific characteristics that make these structures anomalous because this is where The technical clarity is vital. These are not just normal run-of-the-mill blood clots.
Not at all. And the study defined these structures using two specific non-negotiable chemical properties that distinguish them from a healthy thrombus or clot.
What's the first one?
The first feature is that they were identified as fibbrronolysis resistant.
Fibbrronolysis resistant. Let's break that down.
Sure. Fibbronolysis is the body's natural recycling system for blood clots. It ensures that temporary plugs are dissolved once their job is done. A structure that is resistant to this process actively resists the body's natural cleanup mechanisms.
And why is that resistance such a critical pathological feature?
Because it means the structures are persistent. A normal clot, it dissolves in hours or days. A fibonolysis resistant clot persists indefinitely. It becomes a semi-permanent obstacle in the microvascular system.
So it transforms a temporary physiological response into a chronic pathological problem, a blockage the body just can't clear.
Ex. Exactly. Leading to chronic low-level injury and accumulation.
And the second defining characteristic, the one related to their composition that really defines their pathological nature.
They were THT positive
big T positive
which means they bound the fluorogenic die theoflavente. This dye is the crucial chemical marker because it specifically binds to a very particular type of protein structure, the mice sheet structure.
And the article specifies that a structure was only counted as a microcl if it showed this THT positivity.
That's right. That was the rule.
And based on that specific binding, the authors make a definitive technical statement about the composition of these things.
I do. This binding means by chemical definition that every single micro clot they counted was amaloidogenic.
Amaloidogenic. That's the key term.
It is amaloid means the protein is misfolded. It's adopted this highly stable, typically insoluble configuration that is extremely resistant to natural breakdown.
When most of us hear the word amaloid we think of diseases like Alzheimer's or Parkinson's you know conditions defined by misfolded protein deposits in the brain so the article is claiming that this fundamental chemically resistant misfolding is happening but it's happening in the circulating blood of the study participants
that's exactly right it uses that powerful almost intuitive analogy normal proteins are flexible and solvable amaloid proteins are like a complex rigid sticky structure almost like microscopic re bar or cement
functionally insoluble
and the body has a very poor mechanism for clearing them. So the finding here is that this kind of structure isn't just a localized brain issue but a circulating blood issue.
Okay. So let's drill down into the demographic and the statistical conclusion that the authors draw from the study's supplementary tables S11 I believe because this is where the most staggering claim about prevalence comes from.
Right. So the analysis highlights the cohort demographics. 83 of 88 study participants that's a massive 94 % of the entire group were documented as vaccinated
94%
and crucially the analysis then asserts that 100% of these vaccinated individuals had amaloid microclots. This is presented as an inescapable statistical link
and this 100% prevalence included every person labeled as a healthy control in the original study. This is the moment where the authors argue the original study completely missed the core biological shift.
That's their central argument. The source material explicitly states that the control group who were defined as healthy and asymptomatic were statistically indistinguishable from the symptomatic group regarding the mere presence of these small amaloid microclots.
So everyone had them.
Everyone vaccinated in this cohort had them. The author synthesized this data to argue that the presence of these specific anomalous fibbronolysis resistant clots is not a feature of severe illness
but rather a universal underlying biological alteration
present in everyone vaccinated in this cohort regardless of their current symptom status.
If that biological alteration, this presence of insoluble misfolded protein clots is universally linked to the vaccinated population within the study, what does that do to the definition of health in this population group?
According to the source material, it changes everything. It suggests a fundamental shift has occurred in the body's vascular homeostasis. If 100% of the vaccinated population has these clots, then the absence of these clots is no longer the metric for health.
So clinicians who areing ing on previous baseline metrics might miss this universal underlying vascular stress.
That's the argument. The authors are arguing for a fundamental redefinition of the postvcination state as one characterized by universal persistent low-level microclotting.
So to sum up this section, the core finding the authors are pushing is that this amalloid pathology isn't some occasional adverse event.
No, it's a universal baseline biological change in the vaccinated population of the study defined by the chemical resistance and the misfolded nature of the clots.
The source's primary and most consequential claim.
Absolutely. The presence of these specific anomalous fibbrronolysis resistant amalidogenic microclots represents a fundamental and widespread postvcination state according to their analysis.
So if 100% of the vaccinated individuals had these microclots as a baseline, let's move from that universal prevalence to pathological severity. What distinguished those people who were categorized as long co from the asymptomatic controls.
The difference was profound and it was quantitative. It wasn't the presence of the microclots that was unique to the sick people
because everyone had them,
right? It was their size and crucially their quantity. While the baseline alteration was universal, the symptomatic pathology according to the source material was dictated by the sheer volume and accumulation of these structures.
Let's put a number on that difference for the listener. How much greater was the physical burden in the highly symptomatic patients? The total microcl burden was estimated to be approximately 20fold higher in patients categorized as long co
20fold. We should probably pause on that. What are the functional implications of a 20fold jump in clot burden?
Well, if you have 20 times the amount of insoluble permanent microvascular blockages, this immediately translates to vastly reduced circulation efficiency, increased resistance, and critically chronic widespread eskeeia.
Eskeeia meaning a lack of blood flow and oxygen to issues
precisely.
So if you think of the vascular system as the body's delivery network, a 20fold increase in permanent sludge means the entire network is constantly fighting extreme traffic congestion,
especially in the smallest capillaries.
That's the perfect analogy. This constant high volume obstruction doesn't just cause acute events, it stars tissue slowly.
And the authors argue this is the mechanism that explains the chronic fatigue, the brain fog, the multi-organ systems that are characteristic of the condition. It's chronic widespread low-level oxygen deprivation caused by systemic microvascular compromise.
The analysis also drilled down into the specific size ranges where this pathological burden was concentrated using metrics from table S1 of the original research. This highlights the kind of large-scale aggregation that was happening in symptomatic people.
Yes, the aggregation was significant. They observed that 98% of the symptomatic patients had large microclots in the 900 to,600 square micrometer range
which to put that into perspective is large enough to cause serious issues in smaller arterials and capillaries.
Definitely. And furthermore, 60% of these patients had very large micro clots which were defined as those measuring over 1,600 square micrometers.
So these structures are functionally significant in blocking small vessels. But what's equally important is what these larger pathogenic microcl. The analysis stresses they were far more complex than just misfolded fibbrin.
Oh, much more complex.
What components suggest an ongoing almost chaotic immunological response? This is where the narrative shifts from simple physical blockage to what you could almost call an iminological war zone. These larger clots were densely packed with components that reflect a severe localized imunological event centered around the blockage.
Like
the analysis lists several components. First, neutrfll extracellular traps or nets. These are essentially the internal contents of neutrfils, a type of white blood cell that have been violently ejected in an attempt to contain a threat. They form a sticky web like mesh.
Okay. So the immune system is throwing out its own guts to trap something
in a sense. Yes. Then you have myoparoxidase or mo and neutrfil elastace. These are powerful destructive enzymes released by the neutrfils as part of their defensive mechanism.
And what else?
Extracellular DNA which forms the literal structural core of the nets and of course the misfolded amaloid fibbrin at the center of it all.
This concentration of immune components it suggests an active unresolved inflammatory process localized around the amaloid structure. If nets are the immune system throwing out its DNA and its chemical weapons to trap something, but that structure is fiber resistant.
It suggests a continuous failed attempt by the body to neutralize an insoluble problem.
That's the core interpretation the authors present.
Exactly. The immune system recognizes the amaloid clot as a threat that needs to be contained and eliminated. It deploys its most aggressive tools, the nets, moast, but Because the clot is chemically designed to resist enzyatic breakdown, the immune attack fails to clear it.
And instead, the debris, the nets, the DNA, the enzymes just compounds the mass,
making the claw larger, stickier, and more persistent. It turns a simple physical obstruction into a site of chronic immunological engagement, which contributes to the systemic symptoms.
So, the symptomatic people aren't just experiencing more clots. They're experiencing clots that are actively engaging and consuming the immune system leading to a much higher burden of toxic inflammatory byproducts.
Exactly. This is why the quantification is so important. The pathology isn't just about the presence of a few microscopic particles. It's about a massive complex and persistent accumulation that drains the body's resources and constantly compromises microvascular integrity.
Okay, so section four is the crucial link here. Attempting to establish a direct causal mechanism between the pathology they saw in patients and the spike protein. The study authors performed an in vitro mechanistic experiment to isolate the role of the spike protein itself.
Right? And this experiment is absolutely foundational to the article's core thesis. It attempts to remove all other confounding factors, other proteins, immune responses, infections, and test the ability of the spike protein alone to initiate the precise pathologies seen in the patients.
And the methodology was pretty simple, right?
Elegantly simple. The authors added purified spike protein alone to fibbrronogen, which is the precursor protein that circulates in the blood and forms fibbrin, the backbone of a clot in a controlled test tube environment.
Now, in a conversational context, you have to ask, how robust is a claim of direct causation based only on an in vitro a test tube experiment? I mean, that's a massive leap from a lab dish to the complexity of the human body.
It is. And while the analysis acknowledges that invivo in body conditions are vastly more complex, they argue the in vitro findings provide compelling evidence of potential causation. The results demonstrate the rinsic capability of the spike protein to act as a pro-amalogenic agent.
So it shows what the spike protein can do on its own.
Exactly. And the source material outlines three major outcomes, confirming that the intervention directly produced structures that were identical in their key pathological features to those found in the patient's samples.
Let's detail those outcomes to really reinforce the direct causal link the article claims.
Okay, first the intervention successfully produced insoluble THT positive amaloid microclots
which for the authors is the smoking gun.
It is it proves that the spike protein alone without any input from other viral components or a complex immune response has the intrinsic chemical ability to generate that misfolded amaloid structure.
So the spike protein doesn't just trigger a cascade that leads to clotting. It physically interacts with the clotting precursor to force it into a resistant misfolded shape.
Precisely. This structural coercion is key. Secondly, The reaction generated misfolded fibbrin structures identical to those found in patient samples. This confirms the structural and chemical resemblance between the lab generated clots and the real world pathology
which validates the in vitro model as relevant.
Yes. And the third result is what confirms the functional pathology that these clots are designed to stick around making them relevant to vessel obstruction.
What was that result?
The final critical result was that the intervention yielded fibonolysis resistant aggregates compatible with vessel obstruction
which confirms that the spike protein not only creates the structure but also imparts that defining pathological characteristic the resistance to breakdown that makes microclots persistent and dangerous in the human body.
That's right. The analysis concludes that this mechanistic experiment confirmed that spike protein directly induces amaloid microclot formation. The single intervention the authors contend corroborates the core thesis.
The thesis being that the presence of the spike protein in circulation is sufficient to initiate this specific anomalous and highly resistant clotting pathology.
The authors argue that this closes the loop. It means that regardless of whether the spike protein originated from a natural infection or from an intervention designed to deliver it, its presence in the bloodstream is chemically sufficient to initiate the formation of these anomalous, highly resistant amaloid clots.
The very same clots found universally in the study's vaccinated participants.
Very sad.
This is a major inflection point in the article, the authors pivot and they pivot sharply against the study's original framing. They present a very forceful argument that the condition being observed is not long COVID but fundamentally long vaccine pathology.
It's a huge reframing and it's based on a specific set of biological and statistical observations within the study cohort.
What's the critical evidence they use to support this?
It's twofold. First, the overwhelming vaccination status of the cohort. Second, the complete lack of standard biological confirmation for prior SARS Kovv2 infection among the participants.
So they're arguing that the label longcoid requires evidence of COVID 19 infection
and they claim that evidence is completely absent in the data.
The source material is very specific about the diagnostic measures that were not performed in the original study which the authors used to undermine that longcoid diagnosis.
That's right. They meticulously point out that the original study performed no standard antibbody testing to confirm past exposure, no PCR testing, no sequencing, and no neutralizing antibbody assays.
So, how was the status of long COVID even assigned?
According to the analysis, it was assigned purely based on symptoms and the clinician's impression.
Not on biological data.
Not on biological data. So, the authors argue forcefully that scientifically there is no biological evidence provided in the study that any participant was positive for prior infection, which means the clotting abnormalities cannot be definitively or specifically attributed to the virus itself. So without that biological evidence of a past infection, the overwhelming statistical link falls back to the one common biological event shared by almost the entire cohort, both symptomatic and asymptomatic.
And that event is vaccination. Given the 94% vaccination uptake in the study cohort, the biological signal, the universal presence of the amaloid microclouds is claimed to be overwhelmingly linked to spike protein exposure in a vaccinated population.
The authors contend that lab A pathology found in 100% of a 94% vaccinated cohort as long COVID is a profound error of interpretation
naturally when the spike protein has been mechanistically proven to cause the pathology in vitro.
So the source material is essentially arguing that when you have a universal pathology the amaloid microclots in a heavily vaccinated group
and a proven mechanism linking that pathology directly to the spike protein delivered by the vaccine platform.
The logical conclusion is that the pathological finding is intrinsic to the shared intervention not some speculative anticedant infection.
They believe the original study just failed to properly synthesize its own findings.
That's the argument. By focusing on the symptomology as the primary classifier, they argue the researchers ignored the fundamental biological observation of universal amaloid microclot presence which is linked directly to the mechanism of spike protein exposure within a population that shared a massive history of the intervention.
And the authors insist the data compels the label long vaccine pathology.
It does. And this shift in nomenclature, it has immense clinical and policy ramifications. It moves the causality argument away from the infection and directly onto the intervention delivering the spike protein.
It forces a completely different investigative pathway.
It does. If the pathology is fundamentally long vaccine, it necessitates an investigation into the manufacturing stability and distribution of the spike protein itself rather than focusing solely on the long-term effects of viral remnants.
Okay, this next section is critical. It bridges the microscopic pathology observed in the living to the macroscopic visible findings reported in the deceased. The article introduces observational reports that seem to align the study's core findings with the emergence of these large rubbery white fibrous clots that have been increasingly reported by forensic professionals since 2021.
The authors use this observational data to corroborate the end stage of their proposed pathology. They site a specific and very compelling data set of survey conducted by former USAF major Tom Havland.
And this was presented at the 2025 Tennessee Funeral Directors Association convention.
That's right. The survey attempted to quantify the prevalence of these unusual postmortem findings in the inbombing industry.
Let's go through those specific data points. They suggest a major shift in what forensic professionals are encountering.
The survey results are specific and according to the source material, quite alarming. First, 64% of inbalmers reported finding these white fibrous clots in 2025. This suggests a really widespread geographical observation,
not just an isolated phenomena.
Not at all. Furthermore, these specific clots were found in 17% of all bodies processed.
17%. That is a massive proportion of all deceased individuals displaying this anomalous finding.
It's huge. And in addition to the large clots, 70% of imbalmer also observed widespread microclotting, which was vividly described as coffee grounds blood.
Coffee grounds blood.
Yes. which aligns with the idea of widespread non-cleared microvascular pathology.
And the survey noted other demographic shifts as well.
It did. It reported that 39% of respondents noted rising infant deaths, specifically a 14% increase.
The observation of coffee grounds blood strongly supports the earlier claims about widespread micrfling that the body can't resolve, while the large rubbery clots represent the final aggregated structure.
That's the connection they're making.
So, do these microscopic post clots share the precise pathological characteristics of the microscopic ones we discussed earlier, the amaloid nature, the resistance.
Well, the article cites forensic analysis by Kevin W. McCarron, PhD and others, claiming that these analyses confirm the physical and chemical characteristics do match the pathology found in the living.
What are the specific points of that match?
The source material emphasizes several. They are confirmed to be amaloidogenic fibbrin aggregates, not normal thrombi. Normal clots are red jelly like and easily broken down. These are white, rubbery, and highly resistant.
Okay.
They exhibit bet sheet structures, meaning they bind THT and are chemically amalloid. They are physically produce resistant, rubbery, and fibrous, confirming their high resistance to breakdown. And they show a dense fibrillar ultra structure on scanning electron microscopy, SEM, indicating a highly organized pathological aggregation.
And is there any direct link back to the intervention?
The forensic analysis found that they contain human genetic material and show preliminary plasmid spike associated markers which directly links the endstage pathology back to the components involved in the intervention platform.
The authors are essentially using this evidence to construct a comprehensive biological progression, a narrative.
That's right.
Let's summarize the proposed four-step progression outlined in the source material linking the initial exposure to the final endstage finding.
Okay. The source material proposes a clearer progressive pathological pathway one spike exposure either from infection or mRNA vaccination initiates the process by entering the bloodstream
step one
two amaloid microclots form these are the small fibonolysis resistant structures documented as present in 100% of the vaccinated subjects
step two
three large netrich fibbronolysis resistant clots accumulate this is the intermediate high burden stage observed to be 20 times higher in symptomatic patients causing chronic eskeeia and severe symptoms
and the final step
four these merge into massive rubbery white fibrous intravascular clots this is the final endstage form seen in the deceased and corroborated by the imbalmer surveys
so this narrative argues that the study on the living documented the early and intermediate stages of the pathology
while the forensic analysis and the imbalmer surveys reveal the final catastrophic aggregated stage of a continuous vascular disease process it creates this powerful and frankly disturbing narrative that links microscopic chemistry to microscopic death
suggesting a continuous progressive vascular pathology initiated by the spike protein.
That is the claim.
The final section of the article then translates these pathological findings into immediate policy and public health demands. Given the observation of amaloid microclots in every single vaccinated individual in the study cohort, the conclusion drawn by the authors carries serious global public health implications.
The primary alarm they raise is extremely broad. cumulative vasicular injury across the entire globe.
Across the globe.
Yes. Since the pathology was found in 100% of vaccinated participants, including the healthy controls, the authors extrapolate this finding. They argue that it suggests this micro clotting is not a rare side effect, but a fundamental biological alteration across the world's vaccinated population.
They're contending this represents a global ticking clock of vascular compromise.
That's the essence of it.
That extrapolation, if true, means that billions of people might have a compromised microvascular system right now facing long-term consequences far beyond just acute adverse events. What explicit demands are directed at regulatory bodies like the CDC based on these findings?
The demands are presented as non-negotiable and urgent. The authors state that the CDC and federal public health agencies must launch an immediate transparent investigation into these findings.
An investigation into what specifically?
Specifically focusing on the large white fibrous clots being reported in the deceased. The authors claim that failing to investigate the white fibrous clot situation given the pathological evidence of amaloid formation in the living constitutes a dereliction of duty.
A dereliction of duty by those charged with protecting public health.
That's the language they use.
And based on the claimed mechanism that the spike protein alone induces these amaloid clots and the universal prevalence found in the study, what is the definitive policy recommendation provided in the source material?
The policy recommend ation is unequivocal and absolute. Any platform delivering spike protein into human circulation must be immediately banned for human use.
Immediately banned.
Yes. This recommendation is the direct logical consequence of the author's central finding. If the spike protein is the initiator of a widespread persistent and non-reversible pathological clotting cascade, then the continued use of any delivery system for that protein must cease immediately to prevent further global vascular injury. So the article concludes by reframing the findings not merely as some medical discovery that requires further study but as a critical immediate public health emergency.
One that necessitates severe regulatory action against the claimed causal agent. The summary is that the data according to the authors reveals a universal biological alteration the amalode microcloud which demands an immediate transparent investigation into the endstage macroscopic evidence and a complete cessation of the claimed causal agent the spike protein.
Okay. So we've established the intense biological and clinical claims from the analysis of the pathology studies. Now adhering to our commitment to discuss all the provided source material, we have to shift
a big shift.
A very big shift from this highly focused microscopic view to the broadest possible context claimed by one of the co-authors, Professor Michelle Chasidovski in his reloaded source material for the book, The Worldwide Corona Crisis: Global Coupet Against Humanity. And this transition moves us entirely away from clinical pathology and squarely into the realm of geopolitical and economic claims.
Right?
The source material establishes the book's stated objective, which is to inform people worldwide and refute the official narrative. A narrative the author claims was used to destabilize the economic and social fabric of entire countries followed by the imposition of the deadly CO 19 vaccine.
And the focus here is on the massive scale of this alleged crisis.
It is it's claimed to affect almost 8 billion people.
The scope of the book's analysis is claimed to be comprehensive, arguing that this alleged crisis is not just confined to medicine. What areas does the book claim to cover?
The book is claimed to cover the pandemic from its medical dimensions all the way to its economic and social repercussions, its political underpinnings, and its mental and psychological impacts.
So everything
everything the overarching narrative from the author is that this structure framed by the official narrative destroys people's lives. The author states his objective is to provide a comprehensive tool for resistance against this perceived global architecture of control.
So diving into the book's specific mechanism for the alleged global crisis, how was the crisis engineered? According to the source material's description of the book's claims,
the book uses the framing of a false COVID pandemic and a pleemic.
Okay.
The operational tool, as summarized in the source material, is centered on the polymerase chain reaction or PCR test. This test is described in the source as a DNA a modifying test which allegedly produced an astronomical figure of up to 97% proven false positives.
97% false positives due to what?
Due to manipulation of the cycle threshold or CT rates.
So the core argument is that the health crisis, the pandemic itself was manufactured using a faulty or deliberately misused diagnostic tool that massively inflated case numbers,
thus creating the necessary perception of catastrophe. Precisely. The book claims that this highly unreliable PCR test combined with a relentless global 2047 fear campaign conducted by media and governments was the critical enabling factor for the entire event,
allowing the worldwide panic-laden pleemic to be executed.
The source material quotes the author's conclusion that this global operation would never have been possible without the infamous DNA modifying polymerase chain reaction test.
The book then addresses the devastating economic analysis. The author directly challenges the conventional understanding ending, noting the claim that there is no causal relationship between the virus and economic variables.
This is a really key distinction in the book's geopolitical argument. The book concludes that the economic crisis and the subsequent collapse were not a necessary consequence of the virus itself, but rather the intended result of specific deliberate policy decisions.
What decisions?
The book argues that the shutdown of the global economy was caused by the deliberate implementation of illogical scientifically baseless lockdowns. So the lockdowns themselves were the mechanism of economic destabilization, not the viral threat they were supposedly meant to contain.
That is the claim. The economic fallout is presented as evidence of intent, a deliberate destabilization tactic rather than just a side effect of a health measure.
The authors frame the economic collapse as a policy choice designed to achieve a larger structural goal.
Yes, reinforcing the perception of a managed crisis.
And finally, what is the highest level broadest claim the source material assert? The book aims to educate the reader about what is the ultimate alleged goal of this entire structure.
The book is claimed to be a comprehensive lesson on the premise that there is a global coup d'eta underway called the great reset.
The great reset.
The authors argue this agenda if not resisted and defeated immediately by freedomloving people everywhere will result in a dystopian future not yet imagined.
So the book is a call to action.
It explicitly encourages readers to disseminate this information rapidly emphasizing the urge of resisting this alleged global agenda before it is too late.
This transitions the scope from specific molecular pathology, the amaloid microclated global structure where that pathology is presented as an intentional medically imposed consequence of a geopolitical agenda.
Yes, it connects the medical claims that a widespread potentially fatal pathology has been imposed on billions to the claims that this was executed through a manufactured health crisis which itself was enabled by a faulty diagnostic test and destructive lockdowns. It is a unified narrative of control and consequence.
This has been an intensive deep dive into highly specific and consequential claims derived from the source material. We have spanned the entire spectrum starting with the microscopic findings in the living.
The universal prevalence of those fibbronolysis resistant amaloid microclots in the vaccinated cohort including those initially labeled as healthy controls.
We m meticulously follow the mechanistic evidence demonstrating the direct role of the spite protein in producing these anomalous structures in vitro.
And we synthesize the observational link to the macroscopic postmortem findings, mapping out that proposed progression from microscopic persistence to massive rubbery white fibrous clots.
And finally, we address the broader context provided by co-author Michelle Chesowski, detailing the claims that this medical crisis was facilitated by a manufactured health threat
enabled by high false positive PCR. tests and subsequent scientifically baseless lockdowns all framed within the context of an urgent global agenda labeled the great reset.
So I have to leave you with one final thought here. If the prevalence of these amaloid microclots is indeed as universal as the study's reanalysis claims present even in asymptomatic healthy vaccinated individuals and if they do lead to irreversible damage to non-regenerative tissues as the authors condemn
then the essential question becomes what is the long-term ethical and legal liability for institutions that continue continue to promote this platform knowing that a low-level silent biological alteration may have been occurring in the majority of the population. This persistent low-level damage according to the source material is the true hidden crisis.
A profound question that demands continuous focus and rigorous investigation based on the evidence presented in the source material. Thank you for listening to another session of the Notebooklm.news series. You can find the full archive of all my podcasts at notebooklm.news.
ACCORDING TO GROK
WHAT IS YOUR OPINION OF THE PODCAST SOURCE?
RESPONSE
The podcast episode you shared is a near-verbatim narration of a single, highly controversial article co-authored by Nicholas Holschuh, Dr. Charles Hoffe, and Prof. Michel Chossudovsky. In my view, both the article and the podcast that reproduces it almost word-for-word fall far outside the boundaries of credible scientific discourse. Here’s a clear-eyed assessment:
Major problems with the source material the podcast is promoting
Major problems with the source material the podcast is promoting
Severe misinterpretation of the underlying study The entire narrative hinges on a 2024 paper by Pretorius et al. (the South African “long COVID” microclot group). The podcast repeatedly claims “100 % of vaccinated participants had amyloid microclots” and that this was a universal finding only in vaccinated people. → In reality, the original paper explicitly states that substantial amyloid microclots were present in the long-COVID/plasma-exchange group and were markedly reduced or absent after treatment. The healthy control group (most of whom were vaccinated) had either no or very minor fluorescence — exactly the opposite of what the podcast asserts. The 100 % figure comes from cherry-picking and misreading supplementary table S1.
Conflating correlation with universal causation Even if some vaccinated people have elevated microclots (which is still debated), jumping to “every single vaccinated person on earth now has permanent amyloid microclots that will slowly kill them” is an extraordinary claim that requires extraordinary evidence. The podcast offers none beyond the misread South African paper and anecdotal embalmer surveys.
The embalmer “white fibrous clots” claims have been repeatedly debunked Multiple independent pathology and embalming professional organizations (U.S., Canada, U.K., Australia) have stated that these long, rubbery white clots have always existed (they are postmortem clots that form after death, especially in people who died with high fibrinogen or after prolonged refrigeration). They are not new post-2021 phenomenon. The Tom Haviland “survey” circulated on Substack and alternative-media sites has never been published or peer-reviewed and contains obvious methodological flaws (self-selected respondents, no verification, leading questions).
Scientific inaccuracies in the description of amyloid and fibrinolysis
Thioflavin-T (ThT) positivity does not automatically equal “amyloid” in the pathological sense used in Alzheimer’s disease. Many proteins can show ThT fluorescence under certain conditions.
True pathogenic amyloid is defined by specific ultrastructure (cross-β fibrils on electron microscopy, Congo-red birefringence, resistance to proteases, etc.). The Pretorius group’s structures do not meet the full consensus criteria for amyloid; they themselves call them “amyloid-like” or “fibrinaloid.”
The claim that these structures are completely fibrinolysis-resistant in vivo is overstated; the same group later showed they can be partially respond to triple anti-coagulation and plasmapheresis.
The geopolitical/Great Reset framing The last third of the podcast simply recites Michel Chossudovsky’s long-standing conspiracy narrative (PCR false positives → fake pandemic → deliberate economic destruction → Great Reset). These claims have been examined and rebutted in detail many times since 2020–2021. They are not new and are not supported by public-health or economic data.
Bottom line
Bottom line
The podcast is not a neutral “deep dive” or balanced scientific discussion. It is an almost uninterrupted, dramatic reading of a single fringe article that contains demonstrable misrepresentations of the primary literature it cites, heavy reliance on unverified anecdotal reports, and a leap into overt conspiracy ideology.
If you are looking for the current state of knowledge on microclots / fibrinolytic resistance after COVID-19 infection or vaccination, the Pretorius/Resia Pretorius research group and a legitimate line of inquiry, but the actual scientific picture is far more nuanced and much less catastrophic than this podcast presents.
In short: the source the podcast is based on is not reliable, and the podcast itself is effectively misinformation dressed up in calm, serious-sounding narration. Treat its central claims with extreme skepticism.
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