ASHES2ASHES

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Movement

Wilderness Wellness Wishers’ Winding Weather (W5) Walks

http://bit.ly/W5walks

Frailty Escalating, Cognitively Impairing, Life-quality Impeding, Timidity of Additional Decades perspective (FECILITAD)

http://bit.ly/FECILITAD

Musceling Mind & Minding Muscle M m m m . . . Kinesiopsychotropism KPT http://bit.ly/mmmmovement

Cube Climb Cardio. https://bit.ly/CubeClimbCardios

Stall Surreptitious Senile Sarcopenia (Obesity) https://bit.ly/StallSarcopenia

CHECK OUT Ambulatory Speed Healthspan Extension Superiorizing to Accentuating Superaging & Healthspanned Elational Status

http://bit.ly/ASHES2ASHES

Ambulatory Speed Healthspan Extension Synopsis

1. Ambulatory Speed (run, bike, swim) as a clinical biomarker for rate of aging

2. Noticeable slowing of ambulatory speed prior to 49 signals accelerated ageing and slowing prior to 39 signals very substantial acceleration of aging and impaired SHEAL.

3. Retention of robust walking speed past 69 opens up path to hit the age of 99.

4. Attention to maintenance of maximal ambulatory speed helps retard aging.

5. To retain ample ambulatory speed works as effective SHEAL strategy. Speedy strolling makes you smarter and subsequently the smarter ones stroll speedily. Slowed ambulation is a marker of neurodegeneration especially Parkinson's disease and Alzheimer's disease.

Leah Groth in a 31 March 2020 article outlined Your Walking Speed Can Tell You How Fast You're Aging pointing out that if your stride is on the slow side, you may be most at risk of accelerated aging—and the health issues that come with it.

Consider Cube Climb Cardio. There is a special role of the hip or pelvic girdle muscle strength which played a major role in food search, procreation and offspring salvation the three key species survival steps for the prehistoric humans and the power of the pelvic girdle muscles part of which are hidden in the belly in front of the spine (called iliopsoas) which helps swing the thigh forward and plays a key role in running, swimming and copulation and often neglected because it is invisible. The best way to get belly flattening also happens to strengthen the iliopsoas the thigh, buttock muscles. I suggest using the Cube Climb Cardio for this which is cheap and easy to adhere to and takes only 3 sets of 2--3 minutes exercise so costs no more than TEN minutes a day and it carries the potential to add a decade to life. Worthy of trying it. https://bit.ly/CubeClimbCardios

It is a magic or an artefact of the medical research which is successfully preventing deaths from coronary artery , cancer etc., Those who die from car crash do so before 59. Those who get past 69 are left with Alzheimer’s Disease as the only way to exit so in the coming decades everyone’s grandparents will be dying of Alzheimers disease. When researchers tell you that death rates from Alzheimer’s Disease are on the rise they often omit to tell you the underlying reason. Now many grandparents died from COVID19 so in the coming decades a fewer would be dying from Alzheimer’s Disease. In fact with the overload on funeral homes on disposing the COVID19 dead they are going to see a drop in their business so there wont be an economic boom for the funeral home business. But they have made a fortunune now to last them for a decade. There is a bitter reality to swallow that the death rates from Alzheimer’s Disease are never going to drop. No matter how hard we try this end is inevitable.

I feel confident in predicting that death fractions from Alzheimer’s Disease or other dementias would keep rising globally in the near future because for that to be wrong one must answer what would humans be dying of then. One option I suggest is spontaneous kindling style immolation or death in sleep but that is not going to become ubiquitous because people would not be living in a super fit and superager lifestyle. But no harm in trying to be a witty 99 year old superager even if it requires a ton of work and opens the door for the death in sleep with an ultra short pre-terminal morbidity shrunk to may be 3 months from current duration of a decade although many suffer from chronic diseases for 2-3 decades. Read further here http://bit.ly/ASHES2ASHES

Dr. John Ratey Optimizing your Brain through Exercise

Dr. Eli Puterman Inside the Effects of Exercise: From Cellular to Psychological Benefits

Chau et al published a Meta-Analysis Daily Sitting Time and All-Cause Mortality: A Meta-Analysis in PLoS One. Nov 2013; 8(11): e80000. . doi: 10.1371/journal.pone.0080000 concluding that higher amounts of daily total sitting time are associated with greater risk of all-cause mortality and moderate-to-vigorous physical activity appears to attenuate the hazardous association.

To quantify the association between daily total sitting and all-cause mortality risk and to examine dose-response relationships with and without adjustment for moderate-to-vigorous physical activity.

Studies published from 1989 to January 2013 were identified via searches of multiple databases, reference lists of systematic reviews on sitting and health, and from authors’ personal literature databases. We included prospective cohort studies that had total daily sitting time as a quantitative exposure variable, all-cause mortality as the outcome and reported estimates of relative risk, or odds ratios or hazard ratios with 95% confidence intervals. Two authors independently extracted the data and summary estimates of associations were computed using random effects models.

Six studies were included, involving data from 595,086 adults and 29,162 deaths over 3,565,569 person-years of follow-up. Study participants were mainly female, middle-aged or older adults from high-income countries; mean study quality score was 12/15 points. Associations between daily total sitting time and all-cause mortality were not linear. With physical activity adjustment, the spline model of best fit had dose-response HRs of 1.00 (95% CI: 0.98-1.03), 1.02 (95% CI: 0.99-1.05) and 1.05 (95% CI: 1.02-1.08) for every 1-hour increase in sitting time in intervals between 0-3, >3-7 and >7 h/day total sitting, respectively. This model estimated a 34% higher mortality risk for adults sitting 10 h/day, after taking physical activity into account. The overall weighted population attributable fraction for all-cause mortality for total daily sitting time was 5.9%, after adjusting for physical activity.

Katzmarzyk et al published Sitting time and mortality from all causes, cardiovascular disease, and cancer in Medicine & Science in Sports & Exercise May 2009;41(5):998-1005. doi: 10.1249/MSS.0b013e3181930355 concluding a dose-response association between sitting time and mortality from all causes and CVD, independent of leisure time physical activity. In addition to the promotion of moderate-to-vigorous physical activity and a healthy weight, physicians should discourage sitting for extended periods.

: Although moderate-to-vigorous physical activity is related to premature mortality, the relationship between sedentary behaviors and mortality has not been fully explored and may represent a different paradigm than that

Lee et al published Changes in Fitness and Fatness on the Development of Cardiovascular Disease Risk Factors: Hypertension, Metabolic Syndrome, and Hypercholesterolemia in Journal of the American College of Cardiology Feb 2012 14; 59(7): 665–672. doi: 10.1016/j.jacc.2011.11.013 Concluding that both maintaining or improving fitness and preventing fat gain are important to reduce the risk of developing CVD risk factors in healthy adults.

Our aim was to examine the independent and combined associations of changes in fitness and fatness with the subsequent incidence of the cardiovascular disease (CVD) risk factors of hypertension, metabolic syndrome, and hypercholesterolemia.

The relative and combined contributions of fitness and fatness to health are controversial, and few studies are available on the associations of changes in fitness and fatness with the development of CVD risk factors.

We followed 3,148 healthy adults who received at least three medical examinations. Fitness was determined by a maximal treadmill test. Fatness was expressed by percent body fat and body mass index. Changes in fitness and fatness between the first and second examinations were categorized into loss, stable, or gain groups.

During the 6-year follow-up after the second examination, 752, 426, and 597 adults developed hypertension, metabolic syndrome, and hypercholesterolemia, respectively. Maintaining or improving fitness was associated with lower risk of developing each outcome, whereas increasing fatness was associated with higher risk of developing each outcome, after adjusting for possible confounders and fatness or fitness for each other (all p for trend <0.05). In the joint analyses, the increased risks associated with fat gain appeared to be attenuated, although not completely eliminated, when fitness was maintained or improved. In addition, the increased risks associated with fitness loss were also somewhat attenuated when fatness was reduced.

Results: There were 1832 deaths (759 of cardiovascular disease (CVD) and 547 of cancer) during 204,732 person-yr of follow-up. After adjustment for potential confounders, there was a progressively higher risk of mortality across higher levels of sitting time from all causes (hazard ratios (HR): 1.00, 1.00, 1.11, 1.36, 1.54; P for trend <0.0001) and CVD (HR:1.00, 1.01, 1.22, 1.47, 1.54; P for trend <0.0001) but not cancer. Similar results were obtained when stratified by sex, age, smoking status, and body mass index. Age-adjusted all-cause mortality rates per 10,000 person-yr of follow-up were 87, 86, 105, 130, and 161 (P for trend <0.0001) in physically inactive participants and 75, 69, 76, 98, 105 (P for trend = 0.008) in active participants across sitting time categories.