Unstable bradycardia will cause irreversible end organ damage or cardiac arrest if not rapidly diagnosed and treated.
Examples of unstable conditions include:
AMS or obtunded state
Symptomatic hypotension
Ischemic chest pain
Dyspnea from pulmonary congestion/acute HF
Bradycardia has intrinsic and extrinsic causes that ultimately impair cardiac conduction and output
If unstable, goal is to increase heart rate to provide adequate cardiac output using atropine, adrenergic medications, and temporary pacing until the etiology is found and treated
Atropine will not work for non-parasympathetic causes of bradycardia or any conduction interruption below the AV node, consider using epinephrine
Treat the patient, not the number
Bradycardia secondary to hyperkalemia, hypothermia, hypoglycemia, or hypothyroidism will not respond effectively to standard ACLS therapies. Treat the underlying cause.
Always consider hyperkalemia and treat with empiric calcium when thought to be a potential cause
Note the atrial rate (P waves), ventricular rate (QRS), and P-QRS association (PR-interval beat to beat). Pay attention to the width of the QRS, and the rate variation (regular or irregular)
Narrow QRS bradycardias are typically vagally induced and have a better prognosis. They rarely require pacing.
Differential for the causes of Bradycardia
Metabolic (acidosis, hyperkalemia, hypermagnesemia, etc.)
Vagal tone (including ischemia)
AV Blocks
Damage to conduction system (CAD, endocarditis, myocarditis, infiltrative diseases, etc.)
Hypothermia
Elevated ICP
Medications
Beta blockers
Calcium channel blockers
Digoxin (Salvador Dali slurred downsloping ST-segment)
Opiates
Clonidine
Cholinergic's (look for associated toxidrome)