When dealing with a WIDE complex (QRS > 120ms) tachycardia with REGULAR rhythm, the classic teaching is to consider Ventricular tachycardia (VT) until proven otherwise. However, it is important to recognize that there are several rhythms that may mimic VT. Here is how to break it down conceptually:
Remember there are only 3 main causes of narrow & regular tachycardias, aberrant conduction will cause these rhythms to have a wide complex QRS
Remember there are only 3 main causes of narrow & irregular tachycardias, aberrant conduction will cause these rhythms to have a wide complex QRS
Looks closely for P waves in all leads (especially V1 & II) to differentiate between the different potential rhythms
Anything that causes aberrant conduction will turn your narrow complex DDx into your wide complex DDx. Main causes of aberrancy include:
Bundle branch blocks, pacemakers, LVH
Electrolyte, metabolic causes (Hyperkalemia, acidosis, etc.)
Toxicologic causes, medications (e.g. TCA's)
Accessory pathways
Ventricular rhythms
Wide & regular complex tachycardias should be considered to be VT until proven otherwise
Use the VTACH mnemonic below to remember potential VT mimics that may change your management
Do not rely on the VT vs. SVT criteria
Rate usually > 120-130 bpm, and not too wide (QRS < 200 ms)
Should be regular in rhythm
Look carefully for P-QRS complexes, often really wide (QRS > 200 ms)
May have Brugada patterns in aVR and V1
No clear P-waves, consider VT!
The ECG can not reliably distinguish between VT and SVT with aberrant conduction
The ECG is reliable at ruling in VT
The ECG is unreliable at ruling out VT (i.e. at ruling in SVT with AC)
Look carefully for P-QRS complexes in all leads
Look carefully for P-QRS complexes, often really wide (QRS > 200 ms)
May have Brugada patterns in aVR and V1
Look carefully for P-QRS complexes, confirm that QRS is wide in all leads
Rightward axis weighs against STEMI
When dealing with a regular REALLY wide complex tachycardia (RRWCT), it is critical to pause and consider the following:
DDx for RRWCT (QRS > 200 ms, or > 1 big box)
Toxicological and metabolic disturbances like hyperkalemia and Na+ channel blocker toxicities can cause RRWCT’s that mimic Ventricular Tachycardia
Lidocaine, amiodarone, and procainamide all have Na+ channel blocking properties and can worsen toxicities
Consider empiric treatment with calcium and bicarb before antiarrhythmics which can kill these patients!
Check out the following link for more about ventricular tachycardia