Introduction – Metacarpal fractures represent approximately 40% of all injuries to the hand. When these fractures occur over the neck of the 4th or 5th metacarpals, they are said to be “Boxer’s Fractures.” The metacarpal neck is the most common metacarpal segment to be fractured. The 5th metacarpal is the most commonly fractured. Mechanism of fracture is most commonly due to direct blow (axial load) to a clenched fist (punching a wall or fixed object).
Epidemiology – Most common among males, ages 10-29
Anatomy – The metacarpals are concave over the palmar surface and have several distinct regions: a head that articulates with the phalanges at the MCP joint, and narrower neck that connects the head to the shaft, and a base that articulates with the carpals (CMC joint). The 2nd and 3rd metacarpals are slightly more rigid with regard to bony and ligamentous attachments, making it less likely for these to be fractured.
Diagnosis – Physical exam will demonstrate swelling and pain on palpation over the affected metacarpal. There may be a palpable “bump” over the dorsum of the affected hand, representing fractured bone tenting the skin. There may be a laceration over the fracture if the fracture protruded through the skin – an open fracture. If there is rotation of the fracture segments, there may be overlapping of fingers when the patient is asked to partially and/or fully flex the fingers (see diagram, showing overlapping 4th and 5th digits).
X-ray is used to characterize the injury. Three views are obtained—standard AP, lateral, and oblique.
Treatment – If the fracture is open (protruded through the skin), immediate surgery is indicated to wash the wound and repair the fracture. If not open, patient’s are typically splint-immobilized. Reduction (manual pushing/movement by physician) may be attempted if there is significant angulation in the AP or radio-ulnar planes; this may be difficult if there is rotation of the bones. Once swelling has subsided (1-2 weeks post-injury), the patient may be casted for 4 weeks, or may need surgery if the splint or attempted reduction failed to align the fracture to acceptable angulation and/or shortening of the bone segments. Surgery includes pinning the fracture using small Kirschner wires (K-wires) or screws.
Introduction – Carpal tunnel syndrome is a compression neuropathy of the median nerve as it traverses through the carpal tunnel of the volar aspect (flexor surface) of the wrist, and causes pain, paresthesias (tingling), numbness, and motor deficits over the median nerve distribution of the hand. It typically presents with frequent nighttime awakening with hand pain, numbness, and tingling. It appears to have both genetic and environmental causes and is often seen in chronically ill patients, and those who perform repetitive, manual labor.
Epidemiology – Women are more affected than men. Associated conditions include obesity, diabetes, hypothyroid, rheumatoid arthritis, pregnancy, and amyloidosis.
Anatomy – The median nerve travels along the flexor (volar) surface of the wrist and provides motor innervation to the Thenar eminence at the base of the thumb (abductor pollicis brevis, flexor pollicis brevis, opponens pollicis) and sensory innervation to the volar thumb, index, middle and radial half of the ring fingers. The median nerve travels through the carpal tunnel alongside the eight flexor digitorum superficialis/profundus tendons and the flexor pollicis longus. The borders of the carpal tunnel include the scaphoid tubercle and trapezium radially, the hook of the hamate and pisiform ulnarly, the transverse carpal ligament palmarly (roof), and the proximal carpal row dorsally (floor).
Diagnosis – Several physical exam tests may be performed:
Carpal Tunnel compression test (Durkan’s Test) – most sensitive; physician uses thumbs to compress the median nerve for 30 seconds. Positive if elicits pain/paresthesias/numbness.
Phalen’s Test – less sensitive; patient flexes wrist and extends elbow for 60 seconds.
Tinnel’s Test – less sensitive, more specific; physician taps the volar wrist overlying the Carpal Tunnel.
Physicians may also order an electromyography (EMG) to assess the conduction speed of the median nerve. Slower conduction times suggest compression and CTS.
Treatment – Performed step-wise in an algorithmic approach:
Nighttime wrist splints, NSAIDs, activity modification – immobilize affected wrist(s) during sleep, when symptoms are most active. NSAIDs are helpful to reduce inflammation. Activity modification may reduce repetitive stress/compression
Corticosteroid injection of the carpal tunnel – second-line treatment. 80% have transient improvement, but nearly all will have return of symptoms within 1 year.
Carpal Tunnel Release surgery – Incision and release of the transverse carpal ligament, with care to incise the most ulnar aspect of the ligament (so as to avoid transection of the recurrent motor branch of the median nerve).
Introduction – Brachial plexus injuries (BPI) can occur at any level of the plexus and can be functionally devastating. They are classified as:
Traumatic - associated with high energy injuries (vehicle/motorcycle)
Obstetric - complicated child birth (shoulder dystocia).
Locations of injury can be either:
Preganglionic - avulsion proximal to dorsal roots
Postganglionic - distal to dorsal roots
Postganglionic lesions have three distinct patterns: 1. complete plexus injuries, 2. upper plexus injuries (Erb’s palsy), and 3. lower plexus injuries (Klumpke’s palsy).
Erb’s palsy – caudally forced shoulder (compression) or stretch injury to neck (burner-stinger).
Klumpke’s palsy – forced arm abduction (grabbing while falling).
Epidemiology – Complete involvement of all roots (C5-T1) – 75-80%
C5 and C6 injury (Erb’s palsy) – 20-25%
C8 and T1 injury (Klumpke’s palsy) – 0.6 - 3%
Anatomy –See figures for detailed anatomy.
Diagnosis – Can be made on the basis of physical exam. Often EMG is used to confirm palsy.
Preganglionic lesions: flail arm, Horner’s syndrome (involvement of sympathetic chain), medially winged scapula (loss of serratus anterior/rhomboids; long thoracic/dorsal scapular nn, respectively)
Complete postganlionic lesions: flacid arm, motor and sensory deficits
Upper lesion (Erb’s palsy): arm adduction and internal rotation, elbow extension, pronation
C5 – Axillary n. def. weak deltoid (abduction), teres minor (external rotation)
Suprascapular n. def. weak supraspinatus (abduction), infraspinatus (external rotation)
Musculocutaneous n. def. weak biceps (arm flexion, supination)
C6 – Radial n. def. weak brachioradialis, supinator, extensors
Lower lesion (Klumpke’s palsy): results in “claw hand” deformity (wrist extension, MCP extension, IP flexion)
C8-T1– Median and ulnar nn. def. weak FCU (wrist flexion) and lumbricals (MCP flexion, IP extension)
Treatment – Typically watchful waiting, unless mechanism of injury allows for nerve repair.
Introduction – Fractures involving the humerus can occur in the proximal, middle, and distal portions of the bone, with each fracture location representing a different potential nerve palsy syndrome..
Epidemiology – All of these fracture locations have a bimodal distribution of occurrence, both in the young (often males) and elderly (often females).
Anatomy – The location of the axillary nerve near the surgical neck of the humerus makes deltoid muscle paralysis most common in proximal humerus fractures. Patients may present with a “flat” deltoid and weak arm abduction. Because the radial nerve courses along the spiral groove of the humerus, a midshaft fracture can lead to “Saturday Night” palsy of the wrist extensors. Also may be caused by compression by crutches or in an individual who “falls asleep” with his or her arm draped over a chair, hence “Saturday Night”. Patients will have poor wrist extension, leading to “wrist drop”. Median nerve (near supracondylar humerus) and the ulnar nerve (cubital tunnel) palsies are most common in fractures of the distal humerus and medial epicondyle, respectively.
Diagnosis – Patients present with pain, bruising, swelling, and possibly mal-alignment of the upper arm. Younger patients will often report a history of higher-energy falls or trauma, while elderly patients will report a history of low energy falls (fracture is due to osteoporotic bone in this case). The diagnosis is made using radiographs of the upper arm. Look for cortical interruption and evidence of angulation or displacement. In patients with supracondylar (a sub-type of proximal humerus fracture), compartment syndrome of the forearm may result due to vascular compromise of the brachial artery. Distal pulses and neuro status must be carefully monitored.
Treatment – Patients make undergo operative fixation or non-operative treatment (casting) to reduce the fracture and stabilize the bone so that it may properly heal.
Introduction – Rotator cuff injury describes a spectrum of disease that includes both impingement and tears. The rotator cuff is a common tendinous sheath that covers the humeral head and facilitates shoulder stability and motion. It includes 4 muscles: supraspinatus, infraspinatus, teres minor, and subscapularis. Tears may be caused by impingement of cuff muscles during motion (especially overhead motion involving the supraspinatus), shoulder instability, or trauma (especially avulsion involving the subscapularis).
Epidemiology – Tears are especially common in overhead throwing athletes. Chronic tears are characteristic of older patients, while traumatic tears may occur in younger patients.
Anatomy – The supraspinatus lies over the top of the humeral head and is responsible for arm abduction. It is most predisposed to impingement due to its location between the humeral head and the acromion, which compresses the tendon during shoulder movement. The infraspinatus and teres minor cover the back of the humeral head and are external rotators of the arm. The subscapularis crosses over the front of the joint and internally rotates the arm.
Diagnosis – Patients present with insidious shoulder pain exacerbated by overhead activities and nighttime pain. Diagnosis can be made using provocative maneuvers on exam that test resisted elevation, external rotation, and internal rotation of the arm. Definitive diagnosis is usually made with a shoulder MRI, which can demonstrate a tear or tendinopathy.
Treatment – Decisions regarding treatment depend upon age and activity of the patient, tear mechanism, and characteristics of tear (size, partial vs. full). Non-operative treatment with physical therapy to strengthen the cuff, NSAIDs, and corticosteroid injections are first line for most tears. If non-op management fails, patients may undergo arthroscopic or open cuff repair.