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Even mild Covid does what to the heart?

Heart-disease risk soars after COVID — even with a mild caseMassive study shows a long-term, substantial rise in risk of cardiovascular disease, including heart attack and stroke, after a SARS-CoV-2 infection.

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COVID and the Heart: It Spares No One | Johns Hopkins Bloomberg School of Public HealthNew evidence suggests anyone infected with COVID is at higher risk for heart issues—a risk that persists even in relatively healthy people long after the illness has passed.

Excess risk for acute myocardial infarction mortality during the COVID‐19 pandemicJournal of Medical Virology is a clinical virology journal focused on the diagnosis, epidemiology, immunology and pathogenesis of human viral infections & diseases.

Covid-19 infections leave an impact on the heart, raising concerns about lasting damageIt is too soon to say if the heart damage in patients recovering from Covid-19 is transient or permanent, but cardiologists are worried.

COVID’s Damage Lingers in the HeartResearchers increasingly find that the effects of infection by SARS-CoV-2 extend to the cardiovascular system

Long-Term Adverse Effects of Mild COVID-19 Disease on Arterial Stiffness, and Systemic and Central Hemodynamics: A Pre-Post StudyCOVID-19-associated vascular disease complications are primarily associated with endothelial dysfunction; however, the consequences of disease on vascular structure and function, particularly in the long term (>7 weeks post-infection), remain unexplored. Individual pre- and post-infection changes in arterial stiffness as well as central and systemic hemodynamic parameters were measured in patients diagnosed with mild COVID-19. As part of in-laboratory observational studies, baseline measurements were taken up to two years before, whereas the post-infection measurements were made 2–3 months after the onset of COVID-19. We used the same measurement protocol throughout the study as well as linear and mixed-effects regression models to analyze the data. Patients (N = 32) were predominantly healthy and young (mean age ± SD: 36.6 ± 12.6). We found that various parameters of arterial stiffness and central hemodynamics—cfPWV, AIx@HR75, and cDBP as well as DBP and MAP—responded to a mild COVID-19 disease. The magnitude of these responses was dependent on the time since the onset of COVID-19 as well as age (pregression_models ≤ 0.013). In fact, mixed-effects models predicted a clinically significant progression of vascular impairment within the period of 2–3 months following infection (change in cfPWV by +1.4 m/s, +15% in AIx@HR75, approximately +8 mmHg in DBP, cDBP, and MAP). The results point toward the existence of a widespread and long-lasting pathological process in the vasculature following mild COVID-19 disease, with heterogeneous individual responses, some of which may be triggered by an autoimmune response to COVID-19.

Cardiovascular effects of the post-COVID-19 condition | PDF HostPDF Host read free online - Nature Cardiovascular Research, doi:10.1038/s44161-023-00414-8

SARS-CoV-2 infects coronary arteries, increases plaque inflammationNIH-funded research sheds light on link between COVID-19 infection and increased risk of cardiovascular disease and stroke.

COVID-19 sharply boosts risk for blood-fat disordersA new study led by researchers at Albert Einstein College of Medicine involving more than 200,000 adults found that the COVID-19 pandemic caused a 29% increase in risk for developing dyslipidemia, a condition involving abnormal lipid (fat) levels in the blood. Seniors and people with type 2 diabetes were even more strongly affected, experiencing an approximately two-fold increased risk for developing dyslipidemia, which is a major risk factor for cardiovascular diseases such as heart attack and stroke. The research was published today in the print edition of The Journal of Clinical Investigation.

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