Adipose Tissue regeneration/Scar formation

Project aims:

We investigate the mechanisms by which adipose tissues reconstruct after injury. By extending the morphogenesis model [1] to account for tissue reconstruction (ECM insemination, inflammation phase), we show that the model successfully generates regeneration or scar formation as functions of few key parameters, and seems to indicate that the fate of injury outcome could be mainly due to ECM rigidity (crosslink amount and remodelling speed) [2].


Based on these model predictions, we perform a combined in-vivo / in-silico study by interfacing model simulations to experiments on mice tissues. 

We discover in-silico that a transient an early decrease in ECM cross-linking guides tissue repair towards regeneration. Consistent with the computational model, transient inhibition or stimulation of fiber cross-linking for the first six days after subcutaneous adipose tissue (AT) resection in adult mice led to regenerative or scar healing, respectively. Therefore, this work positions the computational  model as a predictive tool for tissue regeneration that behaves as a digital twin of our in vivo model. In addition, it opens new therapeutic approaches targeting ECM cross-linking to induce tissue regeneration in adult mammals. 

Collaborations:

A. Pacary, J. Paupert, A. Lorsignol, M. Ousset, L. Casteilla (RESTORE, Toulouse)

 P. Degond (IMT, Toulouse)

Related publications: 

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