Post date: May 4, 2016 12:37:53 PM
Developmental Essentials by IYCA
Energy Management and Bioenergetics
Questions
Why do coaches concentrate so much on conditioning?
When is it appropriate to involve adult type training with youth athletes?
Take a current program and modify based on chapter content.
Outline a training day for a pee wee program, high school and college.
How would you integrate principles involved in anaerobic and aerobic metabolism to program work to rest rations for games of tag?
Introduction
Explore various means by which the body obtains energy and effects of training and developmental age.
Energy: carried in adenosine triphosphate when broken down, energy is released.
2 issues
longitudinal change in metabolic efficiency - a power due to growth and maturation
effect of training on these factors
metabolic systems never work in isolation - one system is usually dominant (intensity)
identify principal factors in biological development and effects of training
page 187 - figure 7-1 overview between aerobic and anaerobic
oxidative (steady state) metabolism
aerobic use of oxygen in activity - body’s preferred method of providing energy
fatty acids are major source of fuel along with some carbohydrate and even protein
energy is high yield and steady
kreb’s cycle and electron transport chain represent major metabolic machinery
substrates for Keb cycle include:
blood glucose
glycogen from liver and muscle
protein
fatty acids transformed into acetyl CoA(coenzyme a)
page 187 - description of krebs cycle
yield- slow glycolysis
fatty acid nets hundreds of adenosine triphosphate molecules
Control-
intensity determines aerobic or anaerobic dominance
anaerobic threshold - maximal rate of steady state activity -- limiting factor is availability of substrate
figure 7-2 aerobic metabolism
substrate depletion and recovery
during steady state exercise depletion available substrate will occur at level of muscle and liver
prepubertal child favors fatty acids as a substrate and oxidative mechanisms during exercise
development and aerobic performance
increases with development
factors
mechanisms used
amount of muscle mass
efficiency of internal machinery
changes in cardiorespiratory system
VO2max (highest level of steady state available) improves slightly throughout childhood and significantly during puberty
cardiac output - amount of oxygenated blood pumped out by the heart each minute and oxygen consumption by the body (Vo2) have an intimate relationship
VO2 = CO (aO2 - VO2)
page 189 - Fick equation
related to stroke volume - amount of blood pumped with each beat
VO2max and peak - peak of calculation in a maximum test protocol
VO2max is lower in prepubertal as compared to adults
stroke volume is determining factor in VO2max
increase in muscle in puberty is correlated to increase in physical capacity
training effects on endurance capabilities
VO2 max can be improved in children
most effective is three times /week at 85% of VO2 peak
both high and medium intensity improves movement skills and metabolic efficiency
trainers should spend more time improving running skill, neuromuscular coordination and strength rather than reinforce poor running mechanics
Anaerobic (short burst) activity - high intensity performed less than 3 minutes
duration determined by intensity
energy provided by phosphagen system
jumping, short sprints
energy from fast glycolytic
sprints between 100 and 880 meters begin to bring in
phosphagen - 0-30 seconds, quick energy
overview
rate of adenosine triphosphate supply is high but yield is low
creatines kinase - synthetic enzyme for phosphate system
myokinase reaction is immediate source of energy
control
increase in adenosine diphosphate will increase creatine kinase activity (positive feed forward)
will stay during high intensity to allow time for glycolytic glycolysis
as adenosine triphosphate increases in sarcoplasm (inside muscle cell) it inhibit creatine kinase activity
negative feedback
substrate depletion and recovery
phosphagen system makes use of the donor creatine phosphate to make adenosine triphosphate from adenosine diphosphate and hydrolyzes adenosine triphosphate
the phosphagen system and development
figure 7-3 - phosphagen system in children - page 191
children exhibit less power due to less enzyme activity and less ability to phosphorylate adenosine diphosphate to adenosine triphosphate
special note: creatine is naturally produced. IYCA does not promote supplementation. Long term effects of supplementation are unknown
hormonal changes and increase in body size play a role in power output
glycolysis
glycolytic system provides energy for sustained bouts of high intensity exercise (6 sec to 3 minutes)
fast glycolysis = absence of oxygen
slow glycolysis = presence of oxygen = pyruvate in krebs cycle
slow versus fast
pyruvate converted into lactic acid
slow pyruvate transported into mitochondria where fed into krebs (citric acid ) cycle in presence of oxygen
yield
one molecule of glucose = 2-3 molecules of adenosine triphosphate
control - inhibited
glycolysis is by acidic conditions secondary to ischemia and the production of lactate and positive hydrogen ions/ Possibly inhibited by increases in adenosine triphosphate, CRPO4, citrate and free fatty acids
primary control is achieved via phosphorylation of glucose to hexokinase (type of sugar with different structure)
another factor is the breakdown rate of glycogen (stored form of glucose)
major limiting step is conversion of fructose - 6- phosphate to fructose - 1, 6-bisphosphate by phosphofructokinase
adenosine monophosphate produced by phosphagen energy system will stimulate glycolysis via an increase in phosphofructokinase activity
presence of secondary to deamination of proteins will also increase phosphofructokinase activity - - breakdown of proteins (and possibly muscle tissue - after 2 hours of exercise amino acids are consumed as fuel
development and the anaerobic system
lactate threshold is the point at which the body begins to rely on anaerobic mechanisms preferentially
increase in intensity puts the body closer to onset of blood lactate accumulation
point at which lactate is produce at a rate faster than the body can buffer and clear waste
children experience lower peaks in lactic acid
active recovery speeds lactate clearance - lactate not just a waste product but also an energy source
in children lactate threshold occurs at an increased percentage of peak oxygen consumption than in non athletic adults - behave more like athletically trained adults)
relationship between sedentary and active children not yet studied
increase in activity of glycolytic enzymes during growth and maturation
page 193, Figure 7-4. Anaerobic glycolysis in children
training effects on short burst activities and anaerobic metabolism
circumpubertal boys (11-13) - increase in activity of phosphofructokinase post training
increase in maximum anaerobic power range from 4% to 14%
neuromuscular coordination and muscular strength will determine amount of improvement in training
selected special populations
movement should be a part of any medical prescription
asthma - dyspnea and bronchospasm secondary to exercise induced hyperventilation
exercise reduces incidence and dependence on medication
deconditioned athletes more prone to attack
importance of accurate assessment and appropriate progressions
start with short burst activity with appropriate rest
obesity
young children can lose weight rapidly due to increased propensity to lose weight than adults
avoid high impact activity
deconditioned - non-obese - no research
avoid indoor entertainment
send kids outside more
go for a walk, play, slowly build work capacity
conclusion
focus on acquisition of basic movement skills
move well, then move often
as technique improves, slowly look to build work capacity
important to consider all forms of varied training - aerobic and anaerobic demands when programming training
ages 6-11 respond best to game situations (tag related)
practice skills through games