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After updating today, my mouse cursor seems to be four times as large as normal. This has gotta be due sync today because I synced yesterday at 8:00 PM eastern and this was not present. Is there any way to debug this or fix this? I'm sure I can't be the only one that sees this as well.

Just as a quick question, are all the people here having problems people with multiple monitors or using some sort of login service that doesn't have good support for multi monitors? I think my issues was that I use slim, and slim doesn't have true multimonitor support, so it essentially saw my two monitors as being one huge resolution -- so it loaded in the HiDIP mouse cursor because it doesn't know any better.

Just as a quick question, are all the people here having problems people with multiple monitors or using some sort of login service that doesn't have good support for multi monitors? I think my issues was that I use slim, and slim doesn't have true multimonitor support, so it essentially say my two monitors as being one huge resolution -- so it loaded in the HiDIP mouse cursor because it doesn't know any better.

If you see Toshiba Service Station in the list of programs and the installed version is V2.6.14 or lower, click here for instructions to download and install the updated program. You can view the version number by left mouse clicking on TOSHIBA Service Station one time.

If you see Toshiba Service Station in the list of programs and the installed version is V2.2.14 or lower, click here for instructions to download and install the updated program. You can view the version number by left mouse clicking on TOSHIBA Service Station one time.

EDIT - I've struck this problem with BIOS firmware 2.14 & 2.15, but going to the next version back (2.06) results in success. Still not a fix as we need the higher BIOS level to overcome a USB keyboard/mouse lockout problem during preboot.

Hello friends, can you help me with writing a simple script? Because something doesn't work at all The task is as follows. This is necessary so that when you click (with a valid left mouse button (we are talking about an FPS controller) on a specific object, something happens (something, for example, node.Enabled = false)Like a light switch

Aldosterone, which plays a central role in the regulation of blood pressure, is produced by zona glomerulosa (ZG) cells of the adrenal gland. When dysregulated, aldosterone is pathogenic and contributes to the development and progression of cardiovascular and renal disease. Although sustained production of aldosterone requires persistent Ca2+ entry through low-voltage activated Ca2+ channels, isolated ZG cells are considered nonexcitable, with recorded membrane voltages that are too hyperpolarized to permit Ca2+ entry. Here, we show that mouse ZG cells within adrenal slices spontaneously generate membrane potential oscillations of low periodicity. This innate electrical excitability of ZG cells provides a platform for the production of a recurrent Ca2+ signal that can be controlled by Ang II and extracellular potassium, the 2 major regulators of aldosterone production. We conclude that native ZG cells are electrical oscillators, and that this behavior provides what we believe to be a new molecular explanation for the control of Ca2+ entry in these steroidogenic cells. 5376163bf9

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