Kamil Koziński, PhD ENG

• Molecular mechanisms of diabetes and mental disorders co-occurrence
• Metabolic and behavioral characterization of mice with a conditional knockout of Tcf7l2, a risk gene for diabetes and schizophrenia
• Neuronal energetic metabolism

EDUCATION

01.12.2010 – 30.06.2015

PhD in biology

Nencki Institute of Experimental Biology

Polish Academy of Sciences, Warsaw, Poland

03.10.2005 – 30.06.2010

MSc Eng in biotechnology

Warsaw University of Life Sciences, Warsaw, Poland

Interdisciplinary Department of Biotechnology

07.02.2008 – 30.06.2008

Erasmus Student Exchange

Gent University, Gent, Belgium

Faculty of Science

RESEARCH EXPERIENCE

14.08.2016- till now

Center of New Technologies

Warsaw University, Warsaw, Poland

Laboratory of Molecular Neurobiology

Project title: Metabolic and behavioral characterization of mice with a conditional knockout of Tcf7l2, a risk gene for diabetes and schizophrenia.

01.01.2016 – 01.01.2017

Nencki Institute of Experimental Biology

Polish Academy of Sciences, Warsaw, Poland

Department of Molecular and Cellular Neurobiology

Labolatory of Neurobiology

Project title: Novel technologies for pharmacological stimulation of regeneration.

01.12.2010 – 30.06.2015

Nencki Institute of Experimental Biology

Polish Academy of Sciences, Warsaw, Poland

Department of Biochemistry

Labolatory of Cell Signaling and Metabolic Disorders

Project title: Molecular mechanisms of insulin resistance and beta-cell failure in type 2 diabetes.

01.04.2009-30.11.2010

Institute of Genetics and Biotechnology

Warsaw University, Warsaw, Poland

Faculty of Biology

Laboratory of Eukaryotic RNA metabolism

Project title: Integration of approaches to study RNA quality control pathways in plants.

11.02.2008 – 01.06.2008

VIB Department for Molecular Biomedical Research, Gent University, Gent, Belgium

Molecular Cell Biology Unit

Project title: NBPF1 as a putative tumour suppressor gene of Neuroblastoma.

CONFERENCE ATTENDED

28.08.2017 - 31.08.2017

13th International Congress of Polish Neuroscience Society, Warsaw, Poland.

poster presentation – “Role of the TCF7L2 transcription factor in regulating the development of thalamus and growth of thalamic axons”

28.06.2014 – 02.07.2014

The 11th Biennial International Society for the Study of Fatty Acids and Lipids Congress, Stockholm, Sweden

poster presentation – “Wnt signalling increases rate of fatty acid oxidation in pancreatic beta cells through specific activation of PPARalpha pathway”

16-18.10.2013

1st Nencki Institute Phd Student Conference, Warsaw, Poland

oral presentation – “Adipose-derived Wnts modulate beta cell adaptation during progression of type 2 diabetes”

17-21.09.2013

54th International Conference on Bioscience of Lipids: Linking Transcription to Phisiology in Lipidomics, Bari, Italy

poster presentation – “The role of adipose-derived Wnts in beta cell adaptation during progression of type 2 diabetes”

23-26.09.2012

10th Euro Fed Lipid Congress, Cracow, Poland

poster presentation – “Adipose-derived Wnt activators contribute towards adaptation of pancreatic beta cells to systemic insulin resistance”

21-23.11.2008

X Seminar of Biotechnology Students In Poland, Gdańsk, Poland

oral presentation – “NBPF1 as a putative tumour suppressor gene of Neuroblastoma”

23-25.11.2007

IX Seminar of Biotechnology Students In Poland, Łódź, Poland

poster presentation – “Bacterial biodegradation of petroleum”

TRAININGS AND COURSES

25-30.08.2014

FEBS workshop “Lipids as molecular switches”, Spetses, Greece

poster presentation – “Regulation of lipid metabolism in pancreatic beat cells: role of Wnt signalling”

26-30.05.2013

EMBO Workshop on Liver and Pancreas Development, Function and Disease, Athens, Greece

poster presentation – “Adipose-derived Wnts trigger beta cell adaptation towards systemic insulin resistance”

23-29.08.2012

FEBS workshop “Lipids: from Lipidomics to Disease and Green Energy”, Spetses, Greece

poster presentation – “Role of adipose-derived Wnt activators in beta cell adaptation to insulin resistance”