Images & Videos

morphine-dependent increases in calcium (alone or in combination with Tat) are mediated by morphine opioid receptors

Fitting, S., Knapp, P. E., Zou, S., Marks, W. D., Bowers, M. S., Akbarali, H. I., & Hauser, K. F. (2014). Interactive HIV-1 Tat and morphine-induced synaptodendritic injury is triggered through focal disruptions in Na⁺ influx, mitochondrial instability, and Ca²⁺ overload. The Journal of neuroscience : the official journal of the Society for Neuroscience, 34(38), 12850–12864. https://doi.org/10.1523/JNEUROSCI.5351-13.2014 

neuroinflammation assessed by in vivo PET imaging in Tat+ transgenic mice in the presence/absence of morphine treatment

Hermes, D. J., Jacobs, I. R., Key, M. C., League, A. F., Yadav-Samudrala, B. J., Xu, C., McLane, V. D., Nass, S. R., Jiang, W., Meeker, R. B., Ignatowska-Jankowska, B. M., Lichtman, A. H., Li, Z., Wu, Z., Yuan, H., Knapp, P. E., Hauser, K. F., & Fitting, S. (2020). Escalating morphine dosing in HIV-1 Tat transgenic mice with sustained Tat exposure reveals an allostatic shift in neuroinflammatory regulation accompanied by increased neuroprotective non-endocannabinoid lipid signaling molecules and amino acids. Journal of neuroinflammation, 17(1), 345. https://doi.org/10.1186/s12974-020-01971-6 

presence of dystrophic microglia (MIP-1α) and microglia (Iba1) in basolateral nucleus of the amygdala (BLNa) of a Tg26(–/–) female mouse with chronic THC history

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