Dreams
By: Madison Hahn
By: Madison Hahn
Most dreams occur in REM sleep
vivid, sensorimotor hallucinations with a narrative structure
fully disconnected from the external environment
sleeping person has a "high arousal threshold"
olfactory stimuli are shut off
primary visual cortex (V1)
neuromodulators
necessary for external stimuli
in REM they are greatly reduced
norepinephrine, serotonin, histamine, hypocretin
histamine helps incorporate sensory stimuli into consciousness
low-level sensory areas or high-order sensory areas
dreams may be more closely related to the imagination than perception
Lesion studies- have shown that the tempero-parieto-occipital junction must be intact
region effects imagery in wakefulness
dreams are created in a way of imagery
this accounts for strange properties in dreams
ex: scene switches, blended characters, weird plot
Waking- watching the live news
Dreaming- watching a movie being created by an interesting and creative director
nightmares can be characterized as repeated occurrences of extremely dysphoric and well-remembered dreams that usually involve a threat to one's survival, physical integrity, or security.
most result in early awakening, and worsened sleep quality
people generally wake up restless, and alert, but with obvious emotional and physical signs of stress
fear, tachycardia, tachypnea, cold sweats
may be recurrent in people with PTSD, depression, or anxiety
Idiopathic vs posttraumatic nightmares
idiopathic- unknown etiology, and is unrelated to other disorders
content is generally unspecific
failure, being chased, helplessness, evil force, disaster
they can serve as an adaptation to any stressful event in currently happening in life
posttraumatic- part of the stress reaction following a traumatic event
suicidal dreams
nightmares in turn can create more anxiety in the waking life
depression: recurrent nightmares can worsen depression as it can create melancholy after awakening which can last the rest of the day
dreams are not only generated by the brainstem during REM sleep but by complex forebrain mechanisms independently of REM
IMPAIRED FEAR EXTINCTION IS CONTINUOUSLY ACTIVATED IN PEOPLE WITH NIGHTMARE DISORDER
RESULTS IN PEOPLE PROCESSING NEGATIVE AND POSITIVE SEMANTIC STIMULI MORE HEAVILY
Lucid Dreaming- the person has control over their dream, they can even control their own plot
this could in turn help people with recurring nightmares by actively having them change the course of their dream
Neurobiological mechanisms
frontal activation > inhibits the limbic system
in REM frontal activity is usually decreased
in lucid dreams (LD) frontal gamma activity is increased (~40 Hz)
frontal region deals with executive control, rational judgment, attention, and working memory
In REM there is also an increase in dopamine levels which generally occurs in the nucleus accumbens
in theory, activation of the frontal lobe will suppress the limbic system during LD which could decrease the intensity and frequency of nightmares
There are limitations since lucid nightmares can also occur
this type of psychotherapy would happen in a 6-week period with therapists there to guide patients through techniques
techniques would then be used in the dreams to help decrease the intensity of the nightmares
may be efficient
even when lucidity is not achieved, the induction exercises help patients develop critical thinking over their dreams
Hippocampal damage pts Dreams
less episodic
lacked content
dreamed less
struggled with scene imagery
Hippocampus causes dreams to be compromised
networks in the brain that recall memory and construct images in real life are also used in dreams
human participants with bilateral hippocampus damage were compared in a sleep study to healthy participants
hippocampus linked with sleep and memory
damages memory processing
specifically episodic memory
might be necessary for typical dreaming