Publications

Posters and Presentations:

Davis J, Palpant N, Stevenson TK, Converso K, and Metzger J. Diastolic Dysfunction Caused by Excitation-Contraction Uncoupling in a Restrictive Cardiomyopathy Mutant Troponin I Mouse Model. Keystone Symposia: Pathological and Physiological Regulation of Cardiac Hypertrophy, Copper Mountain, CO, January 2008.

Wu H, Stevenson TK, Robinson DA, Romanchuk GL, and Westfall MV. Contractile Response to Endothelin in Myocytes Expressing Troponin Ser43/45 Substitution Mutants. Moses Gunn Symposia, Ann Arbor, MI, March 2009.

Stevenson TK, Moore S, Fredriksson L, Murphy G, and Lawrence DA. Assessment of low Mg2+/High K+-induced synchronous activity in mouse CA3 hippocampal neurons. 2013 Molecular and Integrative Physiology Symposium, University of Michigan, Ann Arbor, MI, April 2013.

Stevenson TK, Moore S, Fredriksson L, Murphy G, and Lawrence DA. Low Mg2+/high K+ model of synchronous activity in the CA3 region of ex vivo hippocampal brain slices. XIVth International Workshop on the Molecular and Cellular Biology of Plasminogen Activation, University of Notre Dame, South Bend, IN, June 2013.

Abstracts:

Stevenson TK, Kampert SE, Romanchuk G, Steggerda J, Keyes EM, and Westfall MV. Phosphomimetic Substitutions in One or Both Ser 43/45 Residues of Cardiac Troponin I Produce Comparable Changes in Contractile Performance. Biophysical Society: 54th Annual Meeting, San Francisco, CA, February 2010.

Stevenson TK, Kampert SE, Romanchuk G, Steggerda J, Keyes EM, and Westfall MV. Cardiac Troponin I Ser 43 and Ser 45 Independently Decrease Ventricle Myocyte Contractile Performance. Myofilament Control Mechanisms in Contraction and Relaxation, Madison, WI, May 2010.

Kampert SE, Stevenson TK, Jensen MA, Keyes EM, Romanchuk GL, and Westfall MV. Combination of Phosphomimetic Substitutions within Cardiac Troponin I Cause Functional Cross-talk. Biophysical Society: 55th Annual Meeting, Baltimore, MD, March 2011.

Book Chapters:

Stevenson TK and Westfall MV. “Tissue Engineering of Fully Differentiated Tissue: Lessons From Work on Cardiac Myocytes.” In Handbook of Molecular and Cellular Methods in Biology and Medicine, edited by Cseke LJ, Kirakosyan AK, Kaufman PB, and Westfall MV. CRC Press, pp 593-606 (2011).

Journal Articles:

Davis J, Yasuda S, Palpant NJ, Martindale J, Stevenson TK, Converso K, and Metzger JM. Diastolic dysfunction and thin filament dysregulation resulting from excitation-contraction uncoupling in a mouse model of restrictive cardiomyopathy. Journal of Molecular and Cellular Cardiology. 53(3): 446-57, 2012.

Hwang H, Robinson DA, Stevenson TK, Wu HC, Kampert, S, Pagani FD, Dyke B, Martin JL, Sadayappan S, Day SM, and Westfall MV. "PKCbetaII modulation of myocyte contractile performance." Journal of Molecular and Cellular Cardiology. 53(2): 176-86, 2012.

Hwang H, Robinson DA, Rogers JB, Stevenson TK, Lang SE, Sadayappan S, Day SM, Sivaramakrishnan S, Westfall MV. "Agonist Activated PKCbeta Translocation and Modulation of Cardiac Myocyte Contractile Function." Scientific Reports. 12(3): 1971, 2013.