The term "hearing impaired" is often used to describe people with any degree of hearing loss, from mild to profound, including those who are deaf and those who are hard of hearing. Many individuals who are deaf or hard of hearing prefer the terms "deaf" and "hard of hearing," because they consider them to be more positive than the term "hearing impaired," which implies a deficit or that something is wrong that makes a person less than whole.

I would imagine that in addition to all the amazing skills that deaf people develop (many simply by virtue of living in a world not designed for them), they can concentrate and sleep better than hearing people, or at least better than hearing people with ADHD who are very easily distracted by sound.


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While it is true about when you said that deaf people like being dead, I am deaf, but I do hope for a miracle cure. I enjoy being deaf at times, like when it comes to sleeping. I often wish that I could speak better. I also wish I could be better engaged in group conversations. I do enjoy enjoy the advantages such as being more attentive, and noticing how hearing people can be so rude at times. But I think that since I have already experienced this, since I already know, it would help me to be an even better person, if there was a miracle cure.

Sometimes, people with SSHL put off seeing a doctor because they think their hearing loss is due to allergies, a sinus infection, earwax plugging the ear canal, or other common conditions. However, you should consider sudden deafness symptoms a medical emergency and visit a doctor immediately. Although about half of people with SSHL recover some or all their hearing spontaneously, usually within one to two weeks from onset, delaying SSHL diagnosis and treatment (when warranted) can decrease treatment effectiveness. Receiving timely treatment greatly increases the chance that you will recover at least some of your hearing.

The USA best chance of the match came early in the second half via another dead-ball situation. Braden Anderson hit a dipping free kick from 45 yards into the area, finding Schmid whose subtle flick was enough to redirect the ball past Diaw before it careened back off the crossbar in the 48th minute.

Deaf soccer is contested by deaf and hard-of-hearing athletes. Under international criteria, players must have a hearing loss of at least 55 decibels in their better ear. Hearing loss below this level has been shown to negatively impact hearing. All players competing in deaf matches must remove all hearing aids before playing.

The quicker you seek treatment, the better your chances are for a full recovery. When hearing can't be fixed with medical treatment, single-sided deafness is often the result, which happens about 15% of the time.

Everyone's hearing naturally declines with age, and people often have one ear that hears better than the other. But if hearing loss appears suddenly in one ear for no apparent reason, you may have experienced sudden sensorineural hearing loss, or SHL, a kind of nerve deafness.

The Survey of Income and Program Participation (SIPP) is one of a few national surveys that regularly collects data identifying the American population of persons with hearing loss or deafness. Estimates from the SIPP indicate that fewer than 1 in 20 Americans are currently deaf or hard of hearing. In round numbers, nearly 10,000,000 persons are hard of hearing and close to 1,000,000 are functionally deaf. More than half of all persons with hearing loss or deafness are 65 years or older and less than 4% are under 18 years of age. However, these findings are limited to those who report difficulty hearing "normal conversation" and do not include the larger population of persons with hearing loss for which only hearing outside the range and circumstances of normal conversation is affected. Policy makers, communications technology manufacturers, health and education service providers, researchers, and advocacy organizations have an interest in these results.

The more time passed, though, the more she started to think he was gone for good. When winter set in, she told herself he was dead because better that than picture Ruger alone and miserable out in the cold.

Congenital deafness has been reported for more than 100 dog breeds, with the list growing at a regular rate (see list); it can potentially appear in any breed but especially in those with white pigmentation of skin and hair. Deafness may have been long-established in a breed but kept hidden from outsiders to protect reputations. The disorder is usually associated with pigmentation patterns, where the presence of white in the hair coat increases the likelihood of deafness. Two pigmentation genes in particular are often associated with deafness in dogs: the merle gene (seen in the collie, Shetland Sheepdog, Dappled Dachshund, Harlequin Great Dane, American Foxhound, Old English Sheepdog, and Norwegian Dunkerhound among others) and the piebald gene (Bull Terrier, Samoyed, Greyhound, Great Pyrenees, Sealyham Terrier, Beagle, Bulldog, Dalmatian, English Setter). However, not all breeds with these genes have been reported to be affected. The deafness, which usually develops in the first few weeks after birth while the ear canal is still closed, usually results from the degeneration of part of the blood supply to the cochlea (the stria vascularis). The sensory nerve cells of the cochlea subsequently die and permanent deafness results. The cause of the vascular degeneration is not known, but appears to be associated with the absence of pigment producing cells known as melanocytes in the stria. All of the functions of these cells are not known, but one role is to maintain high potassium concentrations in the fluid (endolymph) surrounding the hair cells of the cochlea; these pigment cells are critical for survival of the stria and the stria is critical for survival of the hair cells. A different form of congenital hereditary deafness is seen in the Doberman, which is also accompanied by vestibular (balance) disturbance; this deafness results from a different mechanism where hair cell death is not the result of degeneration of the stria but is instead the primary pathology. Deafness may also occur later in life in dogs from other causes such as toxicities, infections, injuries, or due to aging (presbycusis); most of these forms of deafness do not have a genetic cause in animals and thus do not present a concern in breeding decisions, but a newly-identified form of adult-onset hereditary deafness is now recognized in Border Collies and Rhodesian Ridgebacks.

The prevalence of congenital deafness in different breeds is seldom known because of the limited number of studies (see table). In the Dalmatian, where the prevalence is highest, 8% of all dogs in the US are bilaterally deaf and 22% are unilaterally deaf; rates are lower in Europe. In the English Setter, English Cocker Spaniel, Australian Cattle Dog, and Bull Terrier, where fewer numbers of dogs have been hearing tested, the prevalence appears to be about one third to one half that of Dalmatians. Unilateral or bilateral deafness is found in 75% of all white Norwegian Dunkerhounds, but the prevalence in normal-color dogs is unknown. Other breeds with a high prevalence are the Catahoula and Australian Shepherd. The prevalence of all types of deafness in the general dog population is low, reported to be 2.56 to 6.5 cases per 10,000 dogs seen at veterinary school teaching hospitals, but these data predate the availability of hearing testing devices and so are much lower that actual values. Recognition of affected cases is often difficult, because unilaterally deaf dogs appear to hear normally unless a special test (the brainstem auditory evoked response, BAER) is performed; facilities to perform the BAER are usually only available at veterinary schools (see list). It should be noted that a unilaterally deaf dog can be as great a genetic risk for transmission of deafness to its offspring as is a bilaterally deaf dog, so BAER testing of puppies from affected breeds is important.

If instead the deafness is carried as a simple autosomal dominant gene (D), the breeding of an affected dog (Dd) to a free dog (dd) (Table 3) would result on average in 50% affected and 50% free. Dogs with the genotype DD would be unlikely to occur unless two deaf dogs had been bred. All of the above assumes that incomplete penetrance is not acting. If more than one gene (recessive and/or dominant) is involved in producing the deafness, the possible combinations become much more complicated. In humans more than 50 different autosomal recessive or dominant deafness genes or loci have been identified. The children of two deaf parents with two different recessive deafness can be unaffected but carry both genes. If deafness in dogs results from more than one recessive gene, the possible outcomes of breedings are more numerous and determination of the mechanisms of transmission will be difficult.

So what should breeders do when deafness crops up? The most conservative approach would be to not breed the affected animal and not repeat the breeding that produced deafness. It is frequently recommended (i.e. Dalmatian Club of America) that bilaterally deaf puppies should be euthanatized by breeders, since they can make poor pets, are difficult to train, can be prone to startle biting, may die from misadventure (cars), and require excessive care. There is considerable controversy on this point, and there is no question that many people have successfully raised deaf dogs. For every story of a problem deaf dog there seems to be a story of one that was successfully raised. Unfortunately, there is no way to predict how a deaf puppy will turn out. Unilaterally deaf dogs can make good pets but should not be bred. When deafness is uncommon in a breed, affected dogs should not be bred, but this does not mean that all related dogs are a risk and must be retired from breeding. An understanding of simple autosomal recessive and dominant patterns, as explained above, can allow the breeder to make better informed decisions (even though we do not yet know the mechanism of inheritance) and likely avoid future deaf animals without sacrificing a breeding line that has been shaped over many years. However, extreme caution must be used when line breeding of dogs related to deaf dogs, whether the deafness is unilateral or bilateral. To make these decisions in an informed manner for breeds with known deafness, it is important that advantage be taken of hearing testing facilities at veterinary schools. Unilaterally deaf dogs cannot be detected by other means, and these dogs will pass on their deafness genes. be457b7860

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