I did that, however, it didn't work out. I will be grateful if you help me clearly. I just read README file, but it's not clear to understand and there is no other instructions online on the internet.

For the time being, it does not matter where you put that folder. You'll have to compile the Matlab-MEX files, from the source code that resides in the matlab subfolder of your extracted libsvm package.


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Now, in the ROOT/matlab folder, you should have obtained two MEX files (with the .mexw32 or .mexw64 extension). These are the files that you will use from now on. You can discard the rest, except those files (and, maybe, some other generated libraries).

With the aid of @axeoth, here is the solution. I followed instruction files in matlab subfolder of extracted libsvm package. I downloaded and installed Microsoft Visual Studio 2010 and wrote 'mex -setup' on command window of MATLAB to choose a suitable compiler for mex (I use MATLAB R2012b in 32-bit PC, so I found supported compilers in this link ) After then, I followed @axeoth's instructions. I changed the name of the folder to ROOT and moved it to Documents/MATLAB. I started MATLAB and on command window, I wrote these below "one by one"

then I copied everything obtained in ROOT/matlab subfolder. I created a new folder named libsvm under MATLAB/toolbox folder and pasted everything there. and then, I added that libsvm/ folder to the Matlab Path by using "Set Path" button on MATLAB R2012b. After then I wrote "svmtrain" and "svmpredict" so I got the helps of these two commands.

and then I used matlab compiler to compile this function using matlab GUI compiler (File-> new -> Deployment Project and then choose C++ shared Library). It produces this files 2 folders: distrib and src.

I want to use this file in a C++ application. I tried many times and I didn't find a way. All the ways I found over the internet are using old matlab compiler which produces different files or works on an old version of visual studio.

For linker in Additional Library Directories show the way to your matlab libs(C:\Program Files (x86)\MATLAB\R2009b\extern\lib\win32\microsoft for 32bit ver (matlab and VS versions should be the same. I had to install the second Matlab 32bit version.)).

I install the Matlab2009a to my computer successfully and when I try to activate it without using the internet (Activate manually without the Internet) and provide the path to license file. Then an error coming out

If the computer that is running MATLAB cannot connect to the internet, you can download a license file manually from the MathWorks website on another computer and move the file to the target computer.

In this study, we used resting-state fMRI data to map differences in functional connectivity between a comprehensive set of 90 distinct cortical and subcortical brain regions in healthy individuals and adolescents with internet addiction, focusing on individuals engaging in excessive online gaming among the proposed subtypes of this disorder [27]. We also conducted an analysis of network topological disturbances [28] to assess whether any between-group differences in connectivity strength were further associated with a global reconfiguration of functional interactions [26], as has been reported in many other psychiatric disorders [29], [30].

In this study, the NBS was used with network size measured based on its extent; that is, the total number of connections comprising the network. This size measure is not suited to detecting focal effects involving single, isolated connections that do not collectively form a network. To test for these kinds of focal between-group differences, the NBS analysis was repeated testing for differences in component mass rather than size. The mass statistic provides greater sensitivity to focal, intense effects than testing for differences in component size. In addition, we also thresholded edge-wise comparisons using the FDR, which will be very sensitive to high-intensity, focal effects. No significant between-group differences were apparent with either the FDR or mass statistics, suggesting that aberrant connectivity in internet addiction encompasses a distributed network involving several cortical and subcortical regions.

In contrast to the decreased strength of functional connectivity, topological parameters revealed no significant group differences. Even when we conducted further exploratory analyses based on less stringent control against type I error, the results indicated possible topological difference mainly involving the occipital lobe, which was unaffected in the NBS analysis. Thus, while internet addiction was associated with a widespread and significant decrease of functional connectivity in cortico-subcortical circuits, this decrease was not associated with a global disruption in brain functional network topology. This study demonstrates that widespread differences in functional connectivity can exist in the absence of alterations in basic topological measures. It may seem remarkable that differences in connectivity strength were so widespread in the absence of any significant topological differences. However, it is important to remark that topology and connectivity strength are distinct properties of the connectome and abnormalities in one need not imply abnormalities in the other. Similar findings have been observed in other disorders [52], [58]. We note however, that group differences in some topological properties trended towards statistical significance. Analysis of a larger sample may afford the necessary power to declare these effects significant. Our results suggest that topological changes may be more subtle than those observed for functional connectivity measures.

Among the 59 connections included in the altered network, 25 were interhemispheric connections and 34 were intrahemispheric, pointing to involvement of long-range as well as short-range connections throughout the brain. Given that internet addiction is a newly recognized mental health condition, with its concept and diagnostic criteria still elusive and undetermined, perhaps it might be surprising to find such an extensively affected network in the brain of these subjects. Recently, Lin et al. (2012) investigated brain white matter integrity in adolescents with internet addiction using diffusion-tensor imaging, and found a widespread decrease in fractional anisotropy (FA) throughout the brain with no area of higher FA in internet addiction group [13]. Such results implicate a possible anatomical basis for the functional disturbances observed in our sample, a hypothesis that could be tested using combined fMRI and diffusion-weighted imaging in the same participants [59].

Interestingly, a similar pattern of decreased resting-state functional connectivity between frontal and parietal regions was reported both in cocaine- and heroin-dependent individuals [64], [65]. In a recent review, Sutherland et al. (2012) suggested that decreased connectivity in the fronto-parietal circuits might be a central component in the impaired cognitive control network of drug-addicted populations [54]. Our finding in internet addiction also supports the notion that decreased functional connectivity between frontal and parietal regions might be a common characteristic across different types of addiction, suggesting the presence of a shared phenotype that is not a secondary consequence of drug use. In addition, a recent investigation of whole-brain functional connectivity in schizophrenia showed prominent fronto-temporal rather than fronto-parietal or fronto-striatal changes [52], consistent with classical pathophysiological models of the disorder [66]. Of course, the most remarkable finding of the present study is that internet addiction was associated with pathology of striatal circuits in particular, a system commonly implicated in other addictive disorders, suggesting a shared neurobiological phenotype. Identifying altered striatal circuits overlapping with those of well-established addictive disorders might be useful for testing whether the addiction model is an appropriate theoretical framework for understanding the disorder [67], [68]. However, whether a relatively stronger impairment of fronto-parietal and fronto-striatal functional connectivity could be largely specific to addiction disorders still remains in question. Future studies directly comparing different disorders are needed in order to establish specificity.

Proportion of connections affected in internet addiction linking distinct pairs of broad cerebral divisions (detailed for subcortical regions).The number of links involving each pair of divisions is normalized by total number of pair-wise links.

To register and activate your license, start the FEATool GUI on the computer system you wish to activate (the number of systems that can be active at the same time depends on your license type). Make sure your system is connected to the internet and not blocked by firewalls, then select the Activate FEATool License... option from the Help menu.

If you want to de-activate and move an active license to a new computer/system, make sure your computer is connected to the internet, start FEATool Multiphysics, and select Deactivate FEATool License... from the Help menu. This will deactivate, remove, and release the license from your current/old system and generate a new activation token for use with a different system.

If Gmsh or Triangle is selected as grid generation algorithm and FEATool cannot find the corresponding binaries, they will automatically be downloaded, and installed when an internet connection is available. Alternatively, the mesh generator binaries can downloaded from the external grid generators repository and/or compiled manually and installed into any of the directories available on the MATLAB paths (external binaries are typically placed in the bin folder of the FEATool installation directory). be457b7860

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