ECG plays a key role in screening for and diagnosing cardiac conditions in children and teens. It's often utilized to investigate potential congenital heart disease, suspected arrhythmias, and symptoms like chest pain and syncope. But pediatric ECG interpretation presents some unique challenges.

Small children may not sit still, creating artifacts that could hamper interpretation. And even when a good reading can be obtained, what is normal across various ECG measures depends on a patient's age as well as physiological and anatomical changes over time.


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It is critical to get an artifact-free reading for any ECG, but doing so can be particularly challenging in working with small children, so steps must be taken to minimize movement. "In our practice, we use cartoons, movies, stickers, and bubbles as distractions," Stephanie A. Escamilla, MD wrote in a primer for Consultant360.1 "Because the ECG can be completed relatively quickly, these usually suffice."

We do not ask for any personal information except a current email address. The emails are kept in our secure REDCap database and are not used for any purpose except 1) to send the participants the ECG links and response links and 2) to identify their ECG reading in the report that is sent to the facilitator weekly. Participant specialty and stage of training is requested during the registration process. No other PHI/PII data is collected.

Electrocardiography is typically the first cardiac test ordered in adult and pediatric patients presenting with potential cardiac abnormalities. Pediatric patients have unique physiology that rapidly changes from fetal to infant ages and then throughout childhood. These physiologic changes are reflected by changes in the electrocardiogram. In this lesson, we will review the basic principles and findings on the pediatric electrocardiogram.

This is Part 1 of a two-part series about the approach to pediatric ECGs. This series was developed by Eric King, a medical student at the University of Alberta. This podcast was developed with the help of pediatric hospitalist and medical educator Dr. Karen Forbes, pediatric cardiologist Dr. Joseph Atallah and the help of the PedsCases team. This series will cover how to systematically interpret a pediatric ECG, how the forces within the heart change and progress after birth, how to recognize a normal pediatric ECG at different points in childhood, and how to recognize common pediatric ECG abnormalities. Part 1 will discuss rate, rhythm, axis, and intervals, and will work through a case of a 4-week-old boy with a new murmur.

In contrast to adult ECGs, pediatric ECGs exhibit key differences. Fortunately, the majority of these differences are predictable based on normal development and physiology. The neonatal heart is smaller (smaller volume chambers and lower muscular mass) and is more dependent on the right ventricle than the adult heart. The pulmonary vascular resistance is higher in-utero and drops as the child transitions from neonatal to adult physiology. Normal Pediatric ECG parameters will vary by age.


The left ventricle (LV) in the adult is larger than the right ventricle (RV) and the LV thus accounts for the majority of the electrical forces. In the pediatric patient, the RV is relatively larger (see table), and is most dominant at 36 weeks of gestational age. The LV to RV ratio reaches adult proportions by 1-2 months of age. These dynamic changes lead to the changes in the morphology of ECG tracings as the patient ages.

Finally, in the appropriate clinical scenario, pathology that is classically thought of as limited to adults can present in pediatric patients and manifest on ECGs. For example, acute MI, T wave changes secondary to an intracranial process, and changes secondary to electrolyte abnormalities can all be seen on the ECG of a pediatric patient.


Chapter contents Show Definition of normal and pathological pediatric and neonatal ECG Heart rate Rhythm Normal rhythm Arrhythmias Sinus tachycardia Sinus arrhythmia Sinus pause Ectopic atrial rhythm Other tachyarrhythmias Administration of adenosine in pediatric patients Synchronized cardioversion Sinus bradycardia Premature supraventricular and ventricular beats P-wave Abnormal P waves: atrial abnormality 4. PR interval Pre-excitation and atrioventricular (AV) block Accessory pathways and pre-excitation Wolff-Parkinson-White syndrome Causes of varying PR interval The QRS complex Electrical axis (heart axis) QRS duration R-wave and S-wave Q-waves Right ventricular hypertrophy Left ventricular hypertrophy Biventricular hypertrophy Hypertrophic obstructive cardiomyopathy (HOCM) ECG characteristics of HCM/HOCM The ST-segment T-wave U-wave QT interval (QTc) Causes of prolonged QTc interval

The neonatal and pediatric electrocardiogram (ECG) is interpreted using the same principles as in ECG interpretation in adults. The ECG must always be interpreted using a systematic approach in order to minimize the probability of missing significant abnormalities. The following parameters must be assessed, in chronological order:

Sinus arrhythmia occurs in the majority of healthy pediatric patients. P-waves precede all QRS complexes and the PR interval is constant. Although sinus arrhythmia can be pronounced in pediatric patients, it is considered a benign finding.

Tachyarrhythmias in pediatric patients are diagnosed in the same way as in adults (see Diagnosis and Management of Tachyarrhythmias/Tachycardia). AVNRT and AVRT are, second to sinus tachycardia, the most common causes of narrow complexes tachycardia (QRS duration

In total, approximately 90% of all tachyarrhythmias in pediatric patients are supraventricular. The remaining 10% are ventricular and, as in adults, are more serious. Approximately half of the patients with recurring tachyarrhythmias are shown to have a structurally normal heart. Among the rest, structural heart disease and Wolff-Parkinson-White syndrome (pre-excitation) are the most common causes.

Premature supraventricular beats occur already from birth. Supraventricular beats are more common than ventricular beats in the first year of life. Most healthy children exhibit only one or a few premature beats per hour and they usually disappear during physical activity (indicating that they are benign).

Assessment of the PR interval is the same in pediatric and adult patients. Short PR interval (i.e. PR interval shorter than 2nd percentile, see Normal values for Pediatric ECG) suggests pre-excitation. The principles of pre-excitation in children are the same as for adults. For details, refer to Pre-excitation and Wolff-Parkinson-White Syndrome.

As in adults, pediatric patients with accessory pathways only exhibit pre-excitation when impulses are actually conducted over the accessory pathway. In the majority of patients, conduction over the accessory pathway is intermittent, meaning that pre-excitation may not be seen at all times.

Conduction via the accessory pathway may be present already at birth. Conduction is typically intermittent, meaning that delta waves are not always visible. Moreover, some accessory pathways are only capable of conducting from the ventricles to the atria; these patients show no evidence of pre-excitation on resting ECG but may experience tachyarrhythmias. This type of WPW is referred to as concealed WPW syndrome.

The prevalence of WPW syndrome among pediatric patients is approximately 0.1% to 0.2%. Prevalence is higher among children with congenital heart disease. It is recommended that echocardiography be performed in children with evidence of pre-excitation.

ECG has low sensitivity (approximately 20%), but high specificity (approximately 85%) for hypertrophy in pediatric patients (Rivenes et al, Am Heart J, 2003). This implies that the ECG detects 20% of cases with hypertrophy, and 85% of those who meet ECG criteria for hypertrophy actually have hypertrophy. Below follows a list of ECG changes seen in right ventricular hypertrophy.

HCM/HOCM can manifest already in childhood. Dyspnea, chest pain, palpitations, pre-syncope/syncope are common symptoms. These patients have an increased risk of sudden cardiac death, heart failure, and atrial fibrillation. ECG is a relatively sensitive instrument for the detection of HCM/HOCM, since only 5-10% of patients have normal ECG at onset (Veselka et al).

It should also be noted that all children with prolonged QTc interval on one ECG tracing do not necessarily have LQTS. The management of pediatric patients with prolonged QTc interval is discussed in detail in the European guidelines available here.

We describe a simplified method for interpreting a pediatric electrocardiogram (EKG). The method uses 4 steps and requires only a few memorized rules, and it can aid health care providers who do not have immediate access to pediatric cardiology services. Most pediatric EKGs are normal. However, both abnormal and normal EKGs should be sent to a pediatric cardiologist for later, confirmatory interpretation.

When interpreting a pediatric EKG, we must follow a sequence similar when we interpret an electrocardiogram of an adult, but taking into account the differences related to the child's age (see pediatric EKG).

Introducing a new edition of the popular text for medical students, residents, and practitioners on interpreting electrocardiograms in children. Pediatric cardiologists Dr. Myung Park and Dr. Warren Guntheroth teach the vectorial approach to pediatric ECG interpretation in a simple and practical way. How to Read Pediatric ECGs contains over 200 actual size ECG tracings, review questions, case studies for board review.

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