This map depicts US Poverty and Political Leanings by counties. It does this by creating a FeatureLayer instance from an existing portal item and adds it to a basic Map in a SceneView. When an item is loaded as a layer, the Renderer and PopupTemplate saved to the layer in the portal are preserved in the application.
Yes, it is possible to transfer data from Portal for ArcGIS to ArcGIS Online. Data can be transferred either by republishing web maps and services to ArcGIS Online or by using ArcGIS Online Assistant.
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To me the esri_apps content is just one big collection of urls managed in the customer's databases.
That just doesn't seem right.
If it is an on premise installation, that should really be optional.
At the very least, there should be a way to hide this content from the users.
We have our own software distribution process and users within our organization are not allowed to install programs on their own.
When test subjects are out of sight from the Turret's presence, they will maintain a standby state and will remain motionless with their guns retracted. Moving into their angle of sight however, will cause Turrets to deploy their guns shortly after greeting the unlucky individual before opening fire. They will continue to fire until the target is dead or moves out of sight. If the latter occurs, Turrets will briefly search for the test subject (as indicated by the movement of the targeting laser), after which they will revert to a standby state.
In Portal, Turrets will shoot at bulletproof glass and through portals when the player is on the other side. In Portal 2, however, Turrets will only follow the player behind glass with their beam and will not shoot through portals, opening fire almost immediately when the player moves out of the glass or through the portal. In addition, Turrets appear to be unable to see through Hard Light Bridges or Excursion Funnels.
The Rocket Turret, also called a Rocket Sentry, is a unique enemy seen at the end of Portal. It appears as a raw personality core outfitted with a rocket launcher. Upon activation, it will slowly lock onto the player and emit a sound before firing a rocket. Unlike Sentry turrets, it cannot be disabled or destroyed by the player; Attempting to redirect a rocket back at the turret with the use of portals will cause the projectile to fizzle upon impact. It plays a vital role in the battle with GLaDOS, as rockets need to be sent through portals in order to hit her, causing personality cores to detach from her.
In the game's co-op campaign, the Defective Turrets appear as obstacles for Atlas and P-body throughout the Mobility Gels testing course. These Turrets appear to be as fully functional as regular Sentry Turrets and possess the innocent and feminine voice tones as they should. The only difference from regular Turrets is that they have been defaced. This is most likely because Aperture Science had not yet developed the modern casing. One Defective Turret in the final test chamber of this test course has evidently been reconfigured by someone as, instead of serving as a typical testing obstacle, before dying, it reveals that it "needs to protect the humans".
Frankenturrets do not speak, and instead emit incomprehensible chirping sounds. They generally serve as Wheatley's replacement for Storage Cubes once Chell is available as his test subject. They are harmless, their the only difference from normal cubes being that Frankenturrets will hop around aimlessly when not placed on a button. When picked up by Chell, they retract their legs inside the cube. If they fall over on their sides or upside down, they will wave their legs, trying to get up.
Apart from the ending, a Prima Donna Turret can also be found in the chapter The Return, at the end of Test Chamber 11, briefly seen taking a lift upwards just as Chell enters the Chamberlock. It can also be found below the Rattmann's den in the same chapter during Test Chamber 16, where it is observing or lecturing a turret quartet.
It can be distinguished from ordinary Turrets by its immense size, crown and leopard skin-detailed chassis. In the context of the slideshow, the Animal King Turret is used as an example of an entity or a form of organism capable of taking control of Earth which "...refuses to, or is incapable of listening to reason".
The web portal for Azure DevOps is organized around a set of services as well as administrative pages and several task specific features such as the search box. The service labels differ depending on whether you work from Azure DevOps Services or Azure DevOps on premises and its version.
Limited access is available to an unlimited number of stakeholders for free. For details, see Work as a Stakeholder. Most regular contributors must have a TFS client access license (CAL). All Visual Studio subscriptions include a TFS CAL. Find out more about licensing from TFS pricing.
Although you can access source code, work items, and builds from both clients, some task specific tools are only supported in the web browser or an IDE but not in both. Supported tasks differ depending on whether you connect to a Git or TFVC repository from Team Explorer.
Portal and hepatic indicator dilution curves (IDC) were obtained after injection of a mixture of 51Cr-labeled red blood cells ([51Cr]RBC) and 125I-albumin microaggregates (125I-AMA) into the cranial mesenteric artery in dogs. The extraction (E) of 125I-AMA from portal blood was measured during one passage through the hepatic reticuloendothelial system. Using [51Cr]RBC as a vascular reference substance, E-125I-AMA was calculated by comparing simultaneous [51Cr]RBC and 125I-AMA portal and hepatic IDC, and was expressed as percentage of 125I-AMA flowing through the portal vein. In 44 experiments (15 dogs), the colloid was almost completely extracted (E-125I-AMA = 92.3 +/- 1.0% (mean +/- SE)). This approach was applied in 15 patients with severe portal hypertension undergoing combined umbilicoportal, hepatic vein, and superior mesenteric artery catheterization. Eleven patients had alcoholic cirrhosis (AC) and 4 patients had idiopathic noncirrhotic portal hypertension (IPH). Using [51Cr]RBC-IDC, the portal fraction of hepatic blood flow varied between 34.1 and 100% (mean 62.6%) in AC patients and between 56.5 and 91.2% (mean 74.2%) in IPH patients. E-125I-AMA varied from 5.2 to 100% (mean 45.1%) in AC patients, although normal values were obtained in IPH patients (mean 93.2%). In all patients the extraction of Indocyanine green (E-ICG) was calculated using a continuous infusion for the estimation of hepatic blood flow. E-ICG was decreased in AC patients (mean 22.1%), although normal values were obtained in IPH patients (mean 49.5%). A highly significant correlation was found between E-125I-AMA and E-IGC (r = 0.977, P less than 0.001). Also, a significant correlation was found in all patients between E-125I-AMA and the relative clearance of ICG (r = 0.906, P less than 0.001). The correlations between the extraction or clearance of substances removed by two different cell population suggest that their decreases are mainly due to changes in liver microcirculation. In cirrhotics, the decreased E-125I-AMA can be related to part of portal blood bypassing Kupffer cells (intrahepatic portohepatic shunts) and/or to sinusoidal changes responsible for ineffective phagocytosis. Thus, E-125I-AMA can be used as an estimation of the functional portal blood supply to the liver in cirrhotics. Using portal and hepatic IDC after injection of [51Cr]RBC and 125I-AMA into the superior mesenteric artery, the portal fraction of hepatic blood flow and the functional portal blood supply can be estimated simultaneously in patients with portal hypertension before portacaval shunts.
Background/aims: Venous hyporesponsiveness in portal hypertension has been reported previously by us. The present study was undertaken to investigate possible changes of phosphoinositide signal transduction pathway in the portal veins from portal hypertensive rats
Methods: Portal hypertension was induced by partial portal vein ligation. Fourteen days after surgery, portal veins were removed for measurement of [3H]inositol phosphate responses to both receptor- and nonreceptor-mediated stimuli.
Results: Basal [3H]inositol phosphate formation was similar between the two groups. Both phenylephrine and angiotensin II stimulated [3H]inositol phosphate formation in portal veins, but the responses were attenuated in the portal hypertensive group. In contrast, the [3H]inositol phosphate formation by nonreceptor-mediated stimuli (GTP gamma S, NaF/AlCl3, and phospholipase C) was similar between the two groups.
Conclusion: Our results showed that the receptor-mediated [3H]inositol phosphate formation was attenuated, while the non-receptor-mediated formation was unaltered, in the portal vein from portal hypertensive rats.
Diagrammatic representation of the embryological development of the PV. a The vitelline venous system arrives at the primitive liver as two paired veins (right and left), branches into the hepatic sinusoids, and then coalesce, pierce the septum tranversum (primitive diaphragm) and drain into the sinus venosus (primitive heart). These two vitelline veins communicate through three pre-hepatic anastomoses around the developing duodenum (cranial-ventral, dorsal, and caudal-ventral). b Over time, a selective involution occurs, involving the caudal part of the right vitelline vein, the cranial part of the left vitelline vein, and the caudal-ventral anastomosis. The dorsal and cranial-ventral anastomoses persist and give rise to the main PV and to the left PV, respectively. Initially, the paired umbilical veins lie more lateral than the vitelline ones, and also pierce the septum tranversum and drain into the sinus venosus. With the development of the liver, the umbilical veins fragment and connect to the hepatic sinusoids. Over time, a selective involution of the right umbilical vein and cranial portion of the left umbilical vein also occurs. c The remnant left umbilical vein cranially bifurcates, forming two new communications: one with the IVC through the ductus venosus, carrying oxygenated blood from the placenta directly to the fetus; and another with the left PV, supplying directly the liver. After birth, the ductus venosus and the left umbilical vein involute and become the ligamentum venosum and ligamentum teres, respectively ff782bc1db