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Many people say the first step to quitting smoking successfully is to make a firm decision to quit and pick a definite date to stop. Make a plan to deal with the situations that trigger your urge to smoke and to cope with cravings. You may need to try many approaches to find what works best for you. For example, you might:

Some people think smokeless tobacco (chewing tobacco and snuff), pipes, and cigars are safe alternatives to cigarettes. They are not. Smokeless tobacco causes cancer of the mouth and pancreas. It also causes precancerous lesions (known as oral leukoplakia), gum problems, and nicotine addiction. Pipe and cigar smokers may develop cancer of the mouth, lip, larynx, esophagus, and bladder. Those who inhale when smoking are also at increased risk of getting lung cancer as well as heart disease, chronic bronchitis, chronic obstructive pulmonary disease, and emphysema. Using a hookah to smoke tobacco poses many of the same health risks as cigarette smoking.

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Secondhand smoke created by cigarettes, cigars, and pipes can cause serious health problems for family, friends, and even pets of smokers. Secondhand smoke is especially dangerous for people who already have lung or heart disease. In adults, secondhand smoke can cause heart disease and lung cancer. In babies, it can increase the risk of sudden infant death syndrome (SIDS), which is the unexplained death of a baby younger than 1 year of age. Children are also more likely to have lung problems, ear infections, and severe asthma if they are around secondhand smoke.

On Thursday, smoky and hazy conditions will be most noticeable in areas west and south areas in New York State. There is some good news for Eastern New York as an Air Quality Alert continues today; the smoky skies will gradually decrease from north to south because of a trough moving down from New England.

A trough of low pressure has backed down over the state bringing clouds and occasional rain showers. Any rain showers that transpire are a benefit in multiple ways, as they not only moisten the air and ground, but they also aid in cleaning the air of smoke particulates. Showers will be scattered through the next couple of days.

A northerly breeze and another plume of smoke headed down from thenorth is forecast to impact o Western and Southern New York today then continue to head south through Pennsylvania to the Mid Atlantic States, while lesser smoke conditions develop in Eastern New York and in the Adirondacks to New England.

Unlike the plumes of smoke way up high that we saw a few weeks back, the current downwind features of our weather pattern are drawing the smoke to the surface. Wildfire smoke that can blow overhead with little fanfare has now taken a dive from 2,000 feet in the sky to the ground level.

There is some good news about a gradual shift in the steering winds of the jet stream going from north to west by Friday and especially over the weekend. This subtle shift in wind direction with the next trough that brings additional showers back to the northeast United States will benefit us, as greatly less smoky air returns.

Good News: A new trough slips over New York from the Great Lakes on Monday and Tuesday. This system brings rain showers and a wind shift that could keep the smoke away for a short time. A few chances for rain showers also roll through Canada, but there is concern that it may not be enough widespread rainfall to make a big impact on tampering out the hundreds of brush fires.

Corwin analyzed direct and diffuse light hitting different regions of the United States. Even in areas in the West and Southwest blanketed by smoke in 2020, the monthly global horizontal irradiance for each region decreased minimally on average from the year prior.

Corwin and her colleagues plan to look at solar photovoltaic plant data collected by the U.S. Energy Information Administration. She hopes to describe how much energy a solar plant can produce in smokey conditions compared with normal conditions.

Each pair of factors proved to be linked: history of smoking and brain volume; genetic risk for smoking and history of smoking; and genetic risk for smoking and brain volume. Further, the association between smoking and brain volume depended on dose: The more packs a person smoked per day, the smaller his or her brain volume.

And unfortunately, the shrinkage seems to be irreversible. By analyzing data on people who had quit smoking years before, the researchers found that their brains remained permanently smaller than those of people who had never smoked.

News from California throughout the 2020 harvest season was overwhelmingly focused on devastation: record heat waves, uncontrollable wildfires, burned vineyards, smoke tainted wines, and shuttered tasting rooms.

The biggest challenge for California vintners, particularly for those that were directly impacted by wildfires and smoke, will likely be overcoming consumer perceptions. However, winery principals remain undaunted.

Studies about e-cigarettes and smoking behavior show conflicting results. E-cigarettes were mildly helpful in kicking the habit in one clinical trial. In other studies, e-cigarette use did not increase quit rates, or was even associated with a higher risk of continuing to smoke. A recent review concluded that real-world use of e-cigarettes is associated with lower quit rates.

Now the bad news. Nicotine in e-cigarettes may have several negative health effects. Chronic nicotine exposure may lead to insulin resistance and type 2 diabetes, although this risk may be offset by the well-known appetite suppressant effects of nicotine. Inhaled nicotine increases heart rate and blood pressure. Nicotine is highly addictive in its own right, and it may lead to changes in the brain that increase the risk of addiction to other drugs, especially in young people. Nicotine may also impair prefrontal brain development in adolescents, leading to attention deficit disorder and poor impulse control. These potential harms of nicotine are particularly worrisome in view of soaring rates of e-cigarette use in U.S. teenagers.

There is good news to tell about Aboriginal and Torres Strait Islander tobacco control: smoking prevalence is falling and government funding has been secured in a climate of under-investment in prevention.

All this health promotion activity complements the individual cessation support provided to smokers by ACCHOs. A national study found that more Aboriginal and Torres Strait Islander smokers recalled having been advised to quit by a health professional in the past year than a similar sample of all Australian smokers.13

A study published in 2012 in The Journal of the American Medical Association (JAMA) had some good news for people who smoke marijuana: smoking at a rate of one joint a day for as long as seven years doesn't seem to affect lung function adversely.

And then there was the really good news for pot smokers: smoking marijuana in moderation may actually improve lung function, perhaps because inhaling gives the lungs and the chest muscles a bit of a workout.

Because the smoke from marijuana and tobacco contains many of the same substances, including toxic gases and tar, health experts have suspected that the risk of developing COPD from smoking marijuana might be similar to that of smoking tobacco. Yet the results of earlier studies on pot smoking had yielded puzzling results. Many short-term studies demonstrated that while inhaling pot led to coughing and phlegm production, there was no evidence that it decreased lung function.

The researchers suggested that the way pot is smoked may be the reason marijuana could have some pulmonary benefits. The deep, sucking inhalation may stretch lung tissue to expand lung volume. It may also strengthen the muscles of the chest wall, enabling pot users to inhale and exhale air more forcefully.

Hammond Mayor Thomas McDermott Jr. had good news Thursday for fellow marijuana smokers in the city when the comment was made during his Left of Center podcast that a lot of people wind up arrested after lighting up, like himself, to celebrate 420 Day.

"It's something you have to be very careful with," McDermott said. "Edibles are very strong. It would be hard to smoke yourself to death. But you can eat an edible and you will have to go to the emergency room."

Interestingly, nAChRs regulate cell proliferation and inhibit apoptosis (48). For instance, Maouche et al. found that 7 nAChRs were enriched in basal lung epithelia and that, during development, 7 regulated basal cell proliferation (98), which is important for the maintenance of epithelial cell turnover and differentiation. It is well established that smoking is linked to lung cancer, and a hallmark of lung cancer is uncontrolled cell proliferation. West et al. reported that both nicotine and its metabolite (nicotine-derived nitrosamine ketone) stimulated Akt signal transduction downstream of nAChR activation, which altered cell proliferation and apoptosis in bronchial epithelia (156). Specifically, 3, 5, and 4 were identified as candidate genes for a potential role in lung cancer from genome-wide association studies (26, 75, 130, 139). Additionally, Lam et al. found different nAChR subunit gene expression profiles between nonsmokers and smokers with nonsmall cell lung cancer (86). In the same study, human bronchial epithelial cultures (HBEC) were exposed to nicotine, and expression was compared before and after removal of nicotine. Interestingly, exposing HBECs briefly upregulated nAChR 1, 5, and 7 expression at 72 h that returned to baseline levels after removal of nicotine. While all classes of nAChRs are capable of desensitization through chronic agonist exposure, there are definite immediate effects of nicotine on nAChRs in a subunit-dependent manner. Although it is currently unknown whether chronic exposure of nAChR to nicotine via E-Cigs can cause lung cancer, the role of nAChR 7 in contributing to nonsmall cell lung cancer by altering cell proliferation and apoptotic resistance has been reported (86, 113). e24fc04721