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Decamethylcyclopentasiloxane (Cyclomethicone)

VOC Exempt Solvents/ Carrying agents are Cyclic Silicone fluids that are exempt from federal VOC regulations including California (CARB) regulations and OTC regulations. They are HAP-free and do not contribute to ozone-depletion and global warming. In addition, they do not have the odor that many petroleum based solvents possess, making them much more worker-friendly.

Cyclo-2245 (D5) VOC Exempt Solvent is a polydimethylcyclosiloxane that is composed of Decamethylcyclopentasiloxane (CAS#541-02-6). The product is clear, tasteless & odorles. Cyclo2245 is being widely accepted as a VOC-exempt replacement for non-compliant solvents, carrying agents and cleaning agents.

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VOC Exempt Carring Agent

As a carrying agents, Cyclo-2245 has comparable evaporation rates to naptha solvents, odorless mineral spirits (OMS) as well as some of the isoparaffinic solvents. It is compatible with most substrates, including metals and plastics.

Use: Cyclo-2245 Cyclomethicone is a volatile polydimethylcyclosiloxane that is composed of Decamethylcyclopentasiloxane (CAS#541-02-6). The product is clear, tasteless, essentially odorless, non-greasy and non-stinging.

Due to their volatility (varying rates of evaporation), low surface tensions (high Spreadability), and non-greasy feel, Cyclomethicones are used as base fluids, carrying agents and wetting agents in a wide range of personal care products. Cosmetic applications include: room sprays, body sprays, antiperspirants, skin creams, skin lotions, suntan lotions, bath oils, hair care products etc. see also Cosmetic Grade Silicones

Cyclomethicone Fluids are exempt from Federal VOC regulations (including CARB and OTC) and will not contribute to ozone-depletion and global warming. They are being widely accepted as replacements for non-VOC complaint petroleum-bases solvents as both carrying agents as well as for cleaning solvents. Cyclo-2245 is especially effective for applications where low to medium solvency power is desireable. As carrying agents, Cyclo-2244 and Cyclo-2245 have comparable evaporation rates to naptha solvents, odorless mineral spirits (OMS) as well as some of the isoparaffinic solvents. They are compatible with most substates, including metals and plastics.

In addition, D5 Cyclomethicone is being used as a Federal VOC Exempt cleaning agent in dry cleaning applications. It serves to act as a carrying agent for the detergent and will not degrade the fabric or colors of the linens and clothing like other stronger solvents.

Cyclosarin or GF (cyclohexyl methylphosphonofluoridate) is an extremely toxic substance used as a chemical weapon.[1] It is a member of the G-series family of nerve agents, a group of chemical weapons discovered and synthesized by a German team led by Dr. Gerhard Schrader. The major nerve gases are the G agents, sarin (GB), soman (GD), tabun (GA), and the V agents such as VX. The original agent, tabun, was discovered in Germany in 1936 in the process of work on organophosphorus insecticides. Next came sarin, soman and finally, cyclosarin, a product of commercial insecticide laboratories prior to World War II.

At room temperature, cyclosarin is a colorless liquid whose odor has been variously described as sweet and musty,[2] or resembling peaches or shellac. Unlike sarin, cyclosarin is a persistent liquid, meaning that it has a low vapor pressure and therefore evaporates relatively slowly, at only about 1/69th the rate of sarin and 1/20th that of water.

First synthesized during World War II as part of Nazi Germany's chemical weapons research on organophosphate compounds after their military potential was recognized, cyclosarin was also studied later in the United States and Great Britain in the early 1950s as part of a systematic study of potential nerve agents. It was never selected for mass production, however, due to its precursors being more expensive than those of other G-series nerve agents such as sarin (GB).

Evonik offers a wide range of modified aliphatic and cycloaliphatic curing agents with varying pot-life, viscosity, cure speed, and chemical resistance.

Ancamine curing agents are used across many different applications, including flooring, concrete repair, adhesives, chemically resistant linings and secondary containment, solvent-free and high- solids coatings, lay-up laminating, electrical encapsulation, and filament winding.

Blowing agent plays a fundamental role in the production of polystyrene (PS), polyurethane (PU) and polyisocyanurate (PIR) insulations foam. A small quantity of blowing agent indirectly provides important performance characteristics to these foams as great thermal insulation properties.

Introduction: C-X-C Chemokine receptor type 4 (CXCR4) is often overexpressed oroveractivated in different types and stages of cancer disease. Therefore, it is considered a promisingtarget for imaging and early detection of primary tumors and metastasis. In the presentresearch, a new cyclo-peptide radiolabelled with 99mTc, 99mTc-Cyclo [D-Phe-D-Tyr-Lys (HYNIC)-D-Arg-2-Nal-Gly-Lys(iPr)], was designed based on the parental LY251029 peptide, as apotential in vivo imaging agent of CXCR4-expressing tumors.

Abstract: Introduction: C-X-C Chemokine receptor type 4 (CXCR4) is often overexpressed oroveractivated in different types and stages of cancer disease. Therefore, it is considered a promisingtarget for imaging and early detection of primary tumors and metastasis. In the presentresearch, a new cyclo-peptide radiolabelled with 99mTc, 99mTc-Cyclo [D-Phe-D-Tyr-Lys (HYNIC)-D-Arg-2-Nal-Gly-Lys(iPr)], was designed based on the parental LY251029 peptide, as apotential in vivo imaging agent of CXCR4-expressing tumors.

For bicycle riders, Cyclomundo is the one-stop travel center when traveling to France, Italy, Spain, Portugal or Switzerland. Cyclomundo offers guided and self-guided cycling tours for riders of all levels: from novice to seasoned riders. We also arrange logistics and full packages around cyclosportive events like the Etape du Tour and La Marmotte.

In this review, no clear benefit for COX inhibitors was shown over placebo or any other tocolytic agents. While some benefit was demonstrated in terms of postponement of birth for COX inhibitors over placebo and betamimetics and also maternal adverse effects over betamimetics and MgSO4, due to the limitations of small numbers, minimal data on safety, lack of longer-term outcomes and generally low quality of the studies included in this review, we conclude that there is insufficient evidence on which to base decisions about the role of COX inhibition for women in preterm labour. Further well-designed tocolytic studies are required to determine short- and longer-term infant benefit of COX inhibitors over placebo and other tocolytics, particularly CCBs and ORAs. Another important focus for future studies is identifying whether COX-2 inhibitors are superior to non-selective COX inhibitors. All future studies on tocolytics for women in preterm labour should assess longer-term effects into early childhood and also costs.

To assess the effects on maternal and neonatal outcomes of COX inhibitors administered as a tocolytic agent to women in preterm labour when compared with (i) placebo or no intervention and (ii) other tocolytics. In addition, to compare the effects of non-selective COX inhibitors with COX-2 selective inhibitors.

Epidemiological evidence links diets rich in quercetin with decreased incidence of cardiovascular and neoplastic diseases (Hertog et al, 1993, 1994, 1995; Hertog and Hollman, 1996; Keli et al, 1996; Le Marchand et al, 2000). From the mechanistic standpoint, quercetin has been shown to interact with cellular processes in numerous ways (for a review see Nijveldt et al, 2001). Recently, cyclo-oxygenase (COX) enzymes, especially COX-2, have been causally implicated in the early changes associated with carcinogenesis in a number of tissues, among which the colorectal tract has been studied most extensively (Marnett and Dubois, 2002). A noteworthy mechanistic facet of quercetin is its ability to interfere with COX by inhibiting COX-2 promoter activity (Mutoh et al, 2000), COX-2 protein expression (Raso et al, 2001) and COX enzyme activity (Formica and Regelson, 1995). In the light of its interesting biological properties germane to anticarcinogenesis, quercetin has been subjected to a phase I clinical trial in cancer patients, with the aim to develop it as a cancer chemopreventive or antineoplastic agent (Ferry et al, 1996). With respect to potential detrimental effects on health by quercetin, it has been suggested to possess mutagenic and carcinogenic properties (MacGregor and Jurd, 1978; Dunnick and Hailey, 1992), and at high doses there were indications of toxicity in humans (Ferry et al, 1996).

In the light of the interest in quercetin as a potential cancer chemopreventive or chemotherapeutic agent, we reinvestigated its metabolism in the rat using mass spectrometric methods of chemical identification. A particular aim of the study was to compare the qualitative pattern observed in rat plasma with that found in a human who had undergone treatment with quercetin in a clinical trial (Ferry et al, 1996). In the light of the indications of its potential nephrotoxicity demonstrated in that trial, we tested the potential of quercetin to undergo biotransformation via conjugation with glutathione (GSH) to a proximate nephrotoxicant, analogous to the nephrotoxicity associated with hydroquinone (Peters et al, 1997), bromohydroquinone (Monks et al, 1985), 17tag_hash_116-estradiol (Butterworth et al, 1997) or haloalkenes (Iverson et al, 1996). Previous reports have indicated that GSH conjugates are formed in vitro (Awad et al, 2000, 2001; Boersma et al, 2000; Galati et al, 2001). Furthermore, mindful of the fact that COX activity is a potential mechanistic target of quercetin, we compared its effect on cellular prostaglandin E-2 (PGE-2) production with that of representative quercetin metabolites. Overall the work was designed to contribute to the database required for the optimisation of the clinical development of quercetin as a potential cancer chemopreventive and/or chemotherapeutic agent. 006ab0faaa