Chapter 14: ALS and Parkinson's disease

    When I undertook my studies of sleeping practices related to Alzheimer's disease, I also questioned those who were inquiring about Parkinson's Disease(PD), MS, and ALS (amyotrophic lateral sclerosis) (Lou Gehrig's Disease). I really didn't expect that there would be a connection to depleted rebreathing, but I did investigate anyway. Since their cause is still unknown, I thought that perhaps I could find something valuable, some relationship. After a short while I discovered that there didn't appear to be any connection between sleeping under the covers to PD or ALS; therefore I discontinued the study of these outcomes. It doesn't mean that they are not related, my study was quite small. They could be related to some neurological damage that was suffered near birth through depleted rebreathing (particularly Parkinson's disease), which would be particularly difficult to test for. 
ALS seems more like MS as an autoimmune reaction to an allergy or virus. But those are just my first thoughts. I am continuing my research in that area, focussing on possible injuries or their treatment, on sleeping position (like MS, sleeping with a kinked neck could cause nerve damage), and on food allergies that may be relevant to MS. If you wish to participate in this short survey, please browse to ALS Survey.

I do have some thoughts on Parkinson's.
    Individuals with PD often will walk with a shuffling, short step, gait. I believe that this can be avoided or improved in the early stages of the disease. Due to the pain of arthritis and other age-related aches and pains, individuals may begin taking shorter steps. This may be due to weaker balance and/or joint pain. When this takes place the brain, which is always learning, adjusts to the shorter steps, and starts making this shorter step normal, regardless of any pain. However, as aches and pains continue, there will be an additional shortening of steps, and this too becomes internalized and normalized in the brain. This process continues, regardless of the presence of any pain, to the point that the individual's gait is ratcheted down to that of being a shuffle. Their gait or shuffle was the result of the brain's ability to learn new movements. Observers have noticed that those suffering PD had not lost their ability to do other physical movements, such as riding a bike. My opinion here is that they didn't go through the same learning process that they went through with their walking. I would suggest that individuals, who are beginning or show signs of PD, daily practice making longer strides in their walking. They should make a deliberate attempt to avoid restricting their stride. Will this prevent PD. I don't believe so. But it should help reduce the symptoms of the disease. Other practices that may be helpful are dance, Tai Chi and yoga. 
Barry Stanley