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Recent interesting articles :

[1] Begum NA, Izumi N, Nishikori M, Nagaoka H, Shinkura R, Honjo T. Requirement of non-canonical activity of uracil DNA glycosylase for class switch recombination.J Biol Chem. (2007);282:731-42.

[2] Nguyen DH, Gummuluru S, Hu J.Deamination-independent inhibition of Hepatitis B Virus Reverse Transcription by APOBEC3G.J Virol. (2007); 81:4465-72.

[3Holmes RK, Koning FA, Bishop KN, Malim MH. APOBEC3F can inhibit the accumulation of HIV-1 reverse transcription products in the absence of hypermutation. Comparisons with APOBEC3G. J Biol Chem. (2007);282:2587-95.

[4] Pham P, Chelico L, Goodman MF. DNA deaminases AID and APOBEC3G act processively on single-stranded DNA.DNA Repair (Amst). (2007);6:689-92.

[5] Lee JH, Goulian M, Boder ET. Autocatalytic activation of influenza hemagglutinin.J Mol Biol. (2006);364:275-82.

[6] Ledford H.Virus paper reignites prion spat.Nature.(2007);445:575.

[7] Baenziger S, Tussiwand R, Schlaepfer E, Mazzucchelli L, Heikenwalder M, Kurrer MO, Behnke S, Frey J, Oxenius A, Joller H, Aguzzi A, Manz MG, Speck RF.Disseminated and sustained HIV infection in CD34+ cord blood cell-transplanted Rag2-/-gamma c-/- mice.Proc Natl Acad Sci U S A. (2006);103:15951-6.

[8Lötscher M, Recher M, Lang KS, Navarini A, Hunziker L, Santimaria R, Glatzel M, Schwarz P, Böni J, Zinkernagel RM.Induced prion protein controls immune-activated retroviruses in the mouse spleen. PLoS ONE. (2007);2(11):e1158.

[9] Hou F, Sun L, Zheng H, Skaug B, Jiang QX, Chen ZJ. MAVS Forms Functional Prion-like Aggregates to Activate and Propagate Antiviral Innate Immune Response. Cell. (2011);146(3):448-61.