A hair analysis requested from your doctor can be used to determine 

the levels 

of aluminium in your body

A lethal poisoning in a healthy woman caused by deliberate ingestion of aluminium phosphide (AlP), a pesticide used to kill rodents and insects. Toxicity of AlP and review of cases reported to the National Poisons Information Service (London)...

Aluminium is abundant in nature. Even our drinking water is treated with the use of aluminium. Our salt is laced with aluminium (anti-caking agent 554), so is baking powder. Some Deodorant and lots of cosmetics contain aluminium.  How many baking products do you consume ? How much salt you consume every single day? How many cosmetics do you use. Aluminium is widely used along with thousands of other chemicals, preservatives, colorants and chemical agents.

Aluminum build up in the body can be toxic in excessive amounts. Even in small amounts it can cause health problems, if it is deposited in the brain. Many of the symptoms of aluminum toxicity mimic those of Alzheimer’s disease and osteoporosis. Colic, rickets, gastrointestinal problems, interference with the metabolism of calcium, extreme nervousness, anemia, headaches, decreased liver and kidney function, memory loss, speech problems, softening of the bones, and aching muscles can all be caused by aluminum toxicity.

Aluminum is excreted by the kidneys. Toxic amounts can impair kidney function. Aluminum can also accumulate in the brain causing seizures and reduced mental alertness. The brain is normally protected by a blood-brain barrier, which filters the blood before it reaches it. Elemental aluminum does not pass easily through this barrier, but certain compounds contained within aluminum, such as aluminum fluoride do. Interestingly, many municipal water supplies are treated with both aluminum sulfate and aluminum fluoride.

These two chemicals can also combine easily in the blood. Aluminum fluoride is also poorly excreted in the urine.

It is estimated that the normal person takes in between 3 and 10 milligrams of aluminum per day. It can be absorbed into the body through the digestive tract, the lungs and the skin, and is also absorbed by and accumulates in the bodies tissues. Aluminum is found naturally in our air, water and soil. It is also used in the process of making cooking pots and pans, utensils and foil. Other items such as over the counter pain killers, anti-inflammatory products, and douche preparations can also contain aluminum. Aluminum is also an additive in most baking powders, is used in food processing, and is present in antiperspirants, toothpaste, dental amalgams, bleached flour, grated cheese, table salt (anti-caking agent 554 ), and beer, (especially when the beer is in aluminum cans). 

Excessive use of antacids is also a common cause of aluminum toxicity in this country, especially for those who have kidney problems. Many over the counter type antacids contain amounts of aluminum hydroxide that may be to much for the kidneys to handle properly.

  How to prevent or reduce aluminum toxicity 

   from happening to ourselves and our families?

1. Beware of any product containing aluminum ( or aluminium (something) or aluminium dihydroxyaluminum))

2. Read ingredient label; do not use products with the word “aluminium” something. Lots of the time aluminium is used with another chemical

3. Eat a diet that is high in fiber and includes apple pectin

4. Use water filters 

5. Use stainless steel, iron and glass cookware. Do not use aluminium pots. Dispose of pots and pans once the enamel coating has worn off and you can see the silver color surface of aluminium

6. Avoid food or drinking from aluminium cans

7. Use ganoderma to improve your health. The germanium in Ganoderma binds to toxic compounds in the body and is carried out of the body with urine and stool

There is huge amount of research about chemicals and aluminium which we use on everyday basis and the health conserns associated with these chemicals. Start reading ingredient labels and try to use natural products. There is lots of healthy alternatives. 

                                                                      HEALTH TOPICS


THE ROOT CAUSE of CANCER-Nobel Prize Winner Dr.Otto H Warburg 
Avoid these Food Additives 


Despite its natural abundance, aluminium has no known function in biology. It is remarkably nontoxic, aluminium sulfate having an LD50 of 6207 mg/kg (oral, mouse), which corresponds to 500 grams for a 80 kg person.[5] Despite the extremely low acute toxicity, the health effects of aluminium are of interest in view of the widespread occurrence of the element in the environment and in commerce.
Some toxicity can be traced to deposition in bone and the central nervous system, which is particularly increased in patients with reduced renal function. Because aluminium competes with calcium for absorption, increased amounts of dietary aluminium may contribute to the reduced skeletal mineralization (osteopenia) observed in preterm infants and infants with growth retardation. In very high doses, aluminium can cause neurotoxicity, and is associated with altered function of the blood-brain barrier.[66] A small percentage of people are allergic to aluminium and experience contact dermatitisdigestive disorders, vomiting or other symptoms upon contact or ingestion of products containing aluminium, such as deodorants or antacids. In those without allergies, aluminium is not as toxic as heavy metals, but there is evidence of some toxicity if it is consumed in excessive amounts.[67] Although the use of aluminium cookware has not been shown to lead to aluminium toxicity in general, excessive consumption of antacids containing aluminium compounds and excessive use of aluminium-containing antiperspirants provide more significant exposure levels. Studies have shown that consumption of acidic foods or liquids with aluminium significantly increases aluminium absorption,[68] and maltol has been shown to increase the accumulation of aluminium in nervous and osseus tissue.[69] Furthermore, aluminium increases estrogen-related gene expression in human breast cancer cells cultured in the laboratory.[70] The estrogen-like effects of these salts have led to their classification as a metalloestrogen.
The effects of aluminium in antiperspirants has been examined over the course of decades with little evidence of skin irritation.[5] Nonetheless, its occurrence in antiperspirantsdyes(such as aluminium lake), and food additives is controversial in some quarters. Although there is little evidence that normal exposure to aluminium presents a risk to healthy adults,[71]some studies point to risks associated with increased exposure to the metal.[72] Aluminium in food may be absorbed more than aluminium from water.[73] Some researchers have expressed concerns that the aluminium in antiperspirants may increase the risk of breast cancer,[74] and aluminium has controversially been implicated as a factor in Alzheimer's disease.[75] The Camelford water pollution incident involved a number of people consuming aluminium sulfate. Investigations of the long-term health effects are still ongoing, but elevated brain aluminium concentrations have been found in post-mortem examinations of victims, and further research to determine if there is a link with cerebral amyloid angiopathy has been commissioned.[76]
According to The Alzheimer's Society, the overwhelming medical and scientific opinion is that studies have not convincingly demonstrated a causal relationship between aluminium and Alzheimer's disease.[77] Nevertheless, some studies, such as those on the PAQUID cohort,[78] cite aluminium exposure as a risk factor for Alzheimer's disease. Some brain plaqueshave been found to contain increased levels of the metal.[79] Research in this area has been inconclusive; aluminium accumulation may be a consequence of the disease rather than a causal agent. In any event, if there is any toxicity of aluminium, it must be via a very specific mechanism, since total human exposure to the element in the form of naturally occurring clay in soil and dust is enormously large over a lifetime.[80][81] Scientific consensus does not yet exist about whether aluminium exposure could directly increase the risk of Alzheimer's disease.[77]

Effect on plants

Aluminium is primary among the factors that reduce plant growth on acid soils. Although it is generally harmless to plant growth in pH-neutral soils, the concentration in acid soils of toxic Al3+ cations increases and disturbs root growth and function.[82][83][84][85]
Most acid soils are saturated with aluminium rather than hydrogen ions. The acidity of the soil is therefore a result of hydrolysis of aluminium compounds.[86] This concept of "corrected lime potential"[87] to define the degree of base saturation in soils became the basis for procedures now used in soil testing laboratories to determine the "lime requirement"[88] of soils.[89]
Wheat's adaptation to allow aluminium tolerance is such that the aluminium induces a release of organic compounds that bind to the harmful aluminium cationsSorghum is believed to have the same tolerance mechanism. The first gene for aluminium tolerance has been identified in wheat. It was shown that sorghum's aluminium tolerance is controlled by a single gene, as for wheat.[90] This is not the case in all plants.

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Following extract from
Shuchang H, Qiao N, Piye N, et al. Protective effects of gastrodia elata on aluminium-chloride-induced learning impairments and alterations of amino acid neurotransmitter release in adult rats. Restor Neurol Neurosci. 2008;26(6):467-73

Aluminum (Al), the most abundant metal in the earth’s crust, is ubiquitous in the environment and its extensive industrial utilization has stimulated considerable interest in the possible environmental toxicity of this metal. However, little is known about possible effects of Al as a trace element in animals and humans in normal conditions. It has recently become clear that when Al is mobilized from soil by acid rain, it poses a hazard to all exposed organisms (Birchall et al., 1989). Al is also thought to be a causal agent in some cases of encephalopathy and osteomalacia observed in patients with chronic renal failure caused by long-term hemodialysis (Tahara, 2004). Al toxicity in humans has been implicated in many neurodegenerative diseases such as Alzheimer’s disease, amyotrophic lateral sclerosis, and parkinsonism-dementia (Roberts, 1986; Garruto et al., 1994; Solomon et al., 2001). The mechanism of Al-inducted neurotoxicity and identification of effective treatment for such impairments is, therefore, an important public and occupational health priority for industrial and developing nations.

The information on this Website is for informational purpose only and is not intended to replace the care or advice of a physician. If you suspect you have an aluminium toxicity, please consult your physician. 

A hair analysis test can be used to determine levels of aluminium in the body

There is lots of research into the toxic and harmful effects of aluminium on the human body and brain. 
Some of the references below show aluminium association with certain diseases:

* Gupta VB, Anitha S, Hegde ML, et al. Aluminium in Alzheimer's disease: are we still at a crossroad?. Cell Mol Life Sci. Jan 2005

* Campbell, Arezoo. The Potential role of aluminum in Alzheimer's disease.Nephrology Dialysis Transplantation. 2002

* Proudfoot AT. Aluminium and zinc phosphide poisoning. Clin Toxicol (Phila). Feb 2009

* Brown RO, Morgan LM, Bhattacharya SK, Johnson PL, Minard G, Dickerson RN. Potential aluminum exposure from parenteral nutrition in patients with acute kidney injury. Ann Pharmacother. Oct 2008

* Malluche HH, Smith AJ, Abreo K, Faugere MC. The use of deferoxamine in the management of aluminium accumulation in bone in patients with renal failure. N Engl J Med. Jul 19 1984

* Drago D, Cavaliere A, Mascetra N, et al. Aluminum modulates effects of beta amyloid(1-42) on neuronal calcium homeostasis and mitochondria functioning and is altered in a triple transgenic mouse model of Alzheimer's disease. Rejuvenation Res. Oct 2008

* Andrasi E, Pali N, Molnar Z, Kosel S. Brain aluminum, magnesium and phosphorus contents of control and Alzheimer-diseased patients. J Alzheimers Dis. Aug 2005

* Blaylock RL, Strunecka A. Immune-glutamatergic dysfunction as a central mechanism of the autism spectrum disorders. Curr Med Chem. 2009

* Verstraeten SV, Aimo L, Oteiza PI. Aluminium and lead: molecular mechanisms of brain toxicity. Arch Toxicol. Nov 2008

* Aspenstrom-Fagerlund B, Sundstrom B, Tallkvist J, Ilback NG, Glynn AW. Fatty acids increase paracellular absorption of aluminium across Caco-2 cell monolayers. Chem Biol Interact. Oct 7 2009

* Mailloux RJ, Puiseux-Dao S, Appanna VD. Alpha-ketoglutarate abrogates the nuclear localization of HIF-1alpha in aluminum-exposed hepatocytes. Biochimie. Mar 2009

* Gilbert-Barness E, Barness LA, Wolff J, Harding C. Aluminum toxicity. Arch Pediatr Adolesc Med. May 1998

* Balasubramanyam A, Sailaja N, Mahboob M, Rahman MF, Hussain SM, Grover P. In vivo genotoxicity assessment of aluminium oxide nanomaterials in rat peripheral blood cells using the comet assay and micronucleus test. Mutagenesis. May 2009

* Bolt HM, Hengstler JG. Aluminium and lead toxicity revisited: mechanisms explaining the particular sensitivity of the brain to oxidative damage. Arch Toxicol. Nov 2008

* Kan WC, Chien CC, Wu CC, Su SB, Hwang JC, Wang HY. Comparison of low-dose deferoxamine versus standard-dose deferoxamine for treatment of aluminium overload among haemodialysis patients. Nephrol Dial Transplant. May 2010

* Shuchang H, Qiao N, Piye N, et al. Protective effects of gastrodia elata on aluminium-chloride-induced learning impairments and alterations of amino acid neurotransmitter release in adult rats. Restor Neurol Neurosci. 2008

* Vasudevaraju P, Govindaraju M, Palanisamy AP, Sambamurti K, Rao KS. Molecular toxicity of aluminium in relation to neurodegeneration. Indian J Med Res. Oct 2008

* Bogris SL, Johal NS, Hussein I, Duffy PG, Mushtaq I. Is it safe to use aluminum in the treatment of pediatric hemorrhagic cystitis? A case discussion of aluminum intoxication and review of the literature. J Pediatr Hematol Oncol. Apr 2009

* Hernandez G, Bollini A, Huarte M, et al. In vitro effect of aluminium upon erythrocyte membrane properties.Clin Hemorheol Microcirc. 2008

* Lemire J, Mailloux R, Puiseux-Dao S, Appanna VD. Aluminum-induced defective mitochondrial metabolism perturbs cytoskeletal dynamics in human astrocytoma cells. J Neurosci Res. May 1 2009

* Bihaqi SW, Sharma M, Singh AP, Tiwari M. Neuroprotective role of Convolvulus pluricaulis on aluminium induced neurotoxicity in rat brain. J Ethnopharmacol. Jul 30 2009

* Becaria A, Campbell A, Bondy SC. Aluminum as a toxicant. Toxicol Ind Health. Aug 2002

* Bogle RG, Theron P, Brooks P, Dargan PI, Redhead J. Aluminium phosphide poisoning. Emerg Med J. Jan 2006

Candy JM, McArthur FK, Oakley AE, et al. Aluminium accumulation in relation to senile plaque and neurofibrillary tangle formation in the brains of patients with renal failure. J Neurol Sci. Feb 1992

* Cannata-Andia JB, Fernandez-Martin JL. The clinical impact of aluminium overload in renal failure. Nephrol Dial Transplant. 2002

* Exley C, Swarbrick L, Gherardi RK, Authier FJ. A role for the body burden of aluminium in vaccine-associated macrophagic myofasciitis and chronic fatigue syndrome. Med Hypotheses. Feb 2009

* Chang TM, Barre P. Effect of desferrioxamine on removal of aluminum and iron by coated charcoal haemoperfusion and haemodialysis. Lancet. Nov 5 1983

* Chappard D, Insalaco P, Audran M. Aluminum osteodystrophy and celiac disease. Calcif Tissue Int. Jan 2004

* Drüeke TB, Lacour B, Touam M, et al. Effect of aluminum on hematopoiesis. Kidney Int Suppl. Feb 1986

* Friga V, Linos A, Linos DA. Is aluminum toxicity responsible for uremic pruritus in chronic hemodialysis patients?. Nephron. 1997

* McCarthy JT, Milliner DS, Johnson WJ. Clinical experience with desferrioxamine in dialysis patients with aluminium toxicity. Q J Med. Mar 1990

* Graske A, Thuvander A, Johannisson A, et al. Influence of aluminium on the immune system--an experimental study on volunteers. Biometals. Jun 2000

.* Hem JD. Geochemistry and aqueous chemistry of aluminum. Kidney Int Suppl. Feb 1986;

* Kosier JH. Aluminum toxicity in the 1990s. ANNA J. Aug 1999;26(4):423-4.

* Priest ND. The biological behaviour and bioavailability of aluminium in man, with special reference to studies employing aluminium-26 as a tracer: review and study update. J Environ Monit. May 2004

* Ward MK, Feest TG, Ellis HA, Parkinson IS, Kerr DN. Osteomalacic dialysis osteodystrophy: Evidence for a water-borne aetiological agent, probably aluminium. Lancet. Apr 22 1978

* Yokel RA, McNamara PJ. Aluminium toxicokinetics: an updated minireview. Pharmacol Toxicol. Apr 2001

* Key L, Bell N. Osteomalacia and disorders of vitamin D metabolism. In: Internal Medicine. 4th ed. 1994

* Riihimaki V, Valkonen S, Engstrom B, Tossavainen A, Mutanen P, Aitio A. Behavior of aluminum in aluminum welders and manufacturers of aluminum sulfate--impact on biological monitoring. Scand J Work Environ Health. Dec 2008

* Trapp GA. Interactions of aluminum with cofactors, enzymes, and other proteins. Kidney Int Suppl. Feb 1986

* Domingo JL. Reproductive and developmental toxicity of aluminum: a review. Neurotoxicol Teratol. Jul-Aug 1995

GABA and glutamate are two key amino acid neurotransmitters in the CNS. Changes in levels of GABA in either direction may be expected to impair normal nervous system function. In this study, Al-exposed rats (Aluminium exposed rats) had a lower glutamate levels and higher GABA level. 

                                                        Neurotoxic effect of enteral aluminium

...Rats were treated for 90 days with water-soluble, insoluble or chelated aluminium compounds. The daily treatments given were as follows: controls, NaCl (100 mg/kg body weight) plus citric acid (30 mg/kg); AlCl3 (30 or 100 mg/kg); Al(OH)3 (100 mg/kg) plus citric acid (30 mg/kg); Al(OH)3 (300 mg/kg). Their learning ability was determined in the labyrinth test at day 90, and the choline-acetyltransferase, acetylcholinesterase activity and aluminium content of the brains were measured. Soluble and chelated aluminium compounds seriously worsened the learning ability, and the aluminium content of the brain was elevated. Acetylcholinesterase activity increased and choline-acetyltransferase activity decreased, resulting in a diminished cholinergic activity, which is a characteristic of Alzheimer's disease...

                                                                      HEALTH TOPICS


THE ROOT CAUSE of CANCER-Nobel Prize Winner Dr.Otto H Warburg 



Avoid these Food Additives