A lethal poisoning in a healthy woman caused by deliberate ingestion of aluminium phosphide (AlP), a pesticide used to kill rodents and insects. Toxicity of AlP and review of cases reported to the National Poisons Information Service (London)...
Aluminium is abundant in nature. Even our drinking water is treated with the use of aluminium. Our salt is laced with aluminium (anti-caking agent 554), so is baking powder. Some Deodorant and lots of cosmetics contain aluminium. How many baking products do you consume ? How much salt you consume every single day? How many cosmetics do you use. Aluminium is widely used along with thousands of other chemicals, preservatives, colorants and chemical agents.
Aluminum build up in the body can be toxic in excessive amounts. Even in small amounts it can cause health problems, if it is deposited in the brain. Many of the symptoms of aluminum toxicity mimic those of Alzheimer’s disease and osteoporosis. Colic, rickets, gastrointestinal problems, interference with the metabolism of calcium, extreme nervousness, anemia, headaches, decreased liver and kidney function, memory loss, speech problems, softening of the bones, and aching muscles can all be caused by aluminum toxicity.
Aluminum is excreted by the kidneys. Toxic amounts can impair kidney function. Aluminum can also accumulate in the brain causing seizures and reduced mental alertness. The brain is normally protected by a blood-brain barrier, which filters the blood before it reaches it. Elemental aluminum does not pass easily through this barrier, but certain compounds contained within aluminum, such as aluminum fluoride do. Interestingly, many municipal water supplies are treated with both aluminum sulfate and aluminum fluoride.
These two chemicals can also combine easily in the blood. Aluminum fluoride is also poorly excreted in the urine.
It is estimated that the normal person takes in between 3 and 10
milligrams of aluminum per day. It can be absorbed into the
body through the digestive tract, the lungs and the skin, and is also
absorbed by and accumulates in the bodies tissues. Aluminum is found
naturally in our air, water and soil. It is also used in the process
of making cooking pots and pans, utensils and foil. Other items such
as over the counter pain killers, anti-inflammatory products, and
douche preparations can also contain aluminum. Aluminum is also an
additive in most baking powders, is used in food processing, and is
present in antiperspirants, toothpaste, dental amalgams, bleached
flour, grated cheese, table salt (anti-caking agent 554 ), and beer, (especially when the beer
is in aluminum cans).
How to prevent or reduce aluminum toxicity
from happening to ourselves and our families?
1. Beware of any product containing aluminum ( or aluminium (something) or aluminium dihydroxyaluminum))
2. Read ingredient label; do not use products with the word “aluminium” something. Lots of the time aluminium is used with another chemical
3. Eat a diet that is high in fiber and includes apple pectin
4. Use water filters
5. Use stainless steel, iron and glass cookware. Do not use aluminium pots. Dispose of pots and pans once the enamel coating has worn off and you can see the silver color surface of aluminium
6. Avoid food or drinking from aluminium cans
7. Use ganoderma to improve your health. The germanium in Ganoderma binds to toxic compounds in the body and is carried out of the body with urine and stool
There is huge amount of research about chemicals and aluminium which we use on everyday basis and the health conserns associated with these chemicals. Start reading ingredient labels and try to use natural products. There is lots of healthy alternatives.
Despite its natural abundance, aluminium has no known function in biology. It is remarkably nontoxic, aluminium sulfate having an LD50 of 6207 mg/kg (oral, mouse), which corresponds to 500 grams for a 80 kg person. Despite the extremely low acute toxicity, the health effects of aluminium are of interest in view of the widespread occurrence of the element in the environment and in commerce.
Some toxicity can be traced to deposition in bone and the central nervous system, which is particularly increased in patients with reduced renal function. Because aluminium competes with calcium for absorption, increased amounts of dietary aluminium may contribute to the reduced skeletal mineralization (osteopenia) observed in preterm infants and infants with growth retardation. In very high doses, aluminium can cause neurotoxicity, and is associated with altered function of the blood-brain barrier. A small percentage of people are allergic to aluminium and experience contact dermatitis, digestive disorders, vomiting or other symptoms upon contact or ingestion of products containing aluminium, such as deodorants or antacids. In those without allergies, aluminium is not as toxic as heavy metals, but there is evidence of some toxicity if it is consumed in excessive amounts. Although the use of aluminium cookware has not been shown to lead to aluminium toxicity in general, excessive consumption of antacids containing aluminium compounds and excessive use of aluminium-containing antiperspirants provide more significant exposure levels. Studies have shown that consumption of acidic foods or liquids with aluminium significantly increases aluminium absorption, and maltol has been shown to increase the accumulation of aluminium in nervous and osseus tissue. Furthermore, aluminium increases estrogen-related gene expression in human breast cancer cells cultured in the laboratory. The estrogen-like effects of these salts have led to their classification as a metalloestrogen.
The effects of aluminium in antiperspirants has been examined over the course of decades with little evidence of skin irritation. Nonetheless, its occurrence in antiperspirants, dyes(such as aluminium lake), and food additives is controversial in some quarters. Although there is little evidence that normal exposure to aluminium presents a risk to healthy adults,some studies point to risks associated with increased exposure to the metal. Aluminium in food may be absorbed more than aluminium from water. Some researchers have expressed concerns that the aluminium in antiperspirants may increase the risk of breast cancer, and aluminium has controversially been implicated as a factor in Alzheimer's disease. The Camelford water pollution incident involved a number of people consuming aluminium sulfate. Investigations of the long-term health effects are still ongoing, but elevated brain aluminium concentrations have been found in post-mortem examinations of victims, and further research to determine if there is a link with cerebral amyloid angiopathy has been commissioned.
According to The Alzheimer's Society, the overwhelming medical and scientific opinion is that studies have not convincingly demonstrated a causal relationship between aluminium and Alzheimer's disease. Nevertheless, some studies, such as those on the PAQUID cohort, cite aluminium exposure as a risk factor for Alzheimer's disease. Some brain plaqueshave been found to contain increased levels of the metal. Research in this area has been inconclusive; aluminium accumulation may be a consequence of the disease rather than a causal agent. In any event, if there is any toxicity of aluminium, it must be via a very specific mechanism, since total human exposure to the element in the form of naturally occurring clay in soil and dust is enormously large over a lifetime. Scientific consensus does not yet exist about whether aluminium exposure could directly increase the risk of Alzheimer's disease.
Aluminium is primary among the factors that reduce plant growth on acid soils. Although it is generally harmless to plant growth in pH-neutral soils, the concentration in acid soils of toxic Al3+ cations increases and disturbs root growth and function.
Most acid soils are saturated with aluminium rather than hydrogen ions. The acidity of the soil is therefore a result of hydrolysis of aluminium compounds. This concept of "corrected lime potential" to define the degree of base saturation in soils became the basis for procedures now used in soil testing laboratories to determine the "lime requirement" of soils.
Wheat's adaptation to allow aluminium tolerance is such that the aluminium induces a release of organic compounds that bind to the harmful aluminium cations. Sorghum is believed to have the same tolerance mechanism. The first gene for aluminium tolerance has been identified in wheat. It was shown that sorghum's aluminium tolerance is controlled by a single gene, as for wheat. This is not the case in all plants.